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273 studies tagged intestinal, dated 1995-01-01 to 2026-10-23. Bibliography: BibTeX (.bib) · RIS (.ris)
Newest in this topic
- Evidence that the perinatal maternal microbiome mediates metabolite signaling programs that drive sex-specific microglial metabolic alterations and Alzheimer’s disease susceptibility 2026-10-23
- Change in gut microbiome precedes symptomatic Alzheimer’s disease and is correlated with amyloid-beta accumulation in the brain 2026-10-19
- The Gut–Liver–Brain Axis in Neurodegeneration: Unraveling Metabolic–Immune Crosstalk from Peripheral Sensing to Central Vulnerability 2026-10-08
Studies
273 studies carry this tag.
Date range
1995 to 2026
Top journals
- Journal of Alzheimer's disease : JAD 13
- Scientific reports 8
- Nutrients 7
- Brain, behavior, and immunity 6
- Frontiers in cellular and infection microbiology 6
168 journals in all.
Newest first, then by address. Each study has a usefulness score out of 100 (rubric version 4.0); tap a score to see what makes it up. Tags were assigned automatically from each study's title and summary. The same studies as a filter in the library.
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Maternal microbiome-dependent programming of neuroimmune and metabolic pathways during early development may critically shape sex-specific susceptibility to Alzheimer’s disease (AD) later in life.
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Early Ap accumulation is associated with distinct gut microbial signatures and reduced neuroactive metabolic potential, identifying targets for potential preclinical AD intervention.
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However, increasing evidence indicates that systemic metabolic dysfunction, chronic inflammation, intestinal dysbiosis, hepatic abnormalities, and barrier impairment may substantially influence disease susceptibility and progression.
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Environmental exposures are major but understudied determinants of chronic disease.
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Alzheimer’s disease is a progressive, currently irreversible, neurodegenerative disorder associated with the accumulation of β-Amyloid (Aβ) plaques between cells and tau (τ) tangles within cells.
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Alzheimer’s disease (AD) is a neurodegenerative disorder characterized primarily by progressive cognitive impairment.
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These findings identify TTR as a contributor of adaptive neurovascular homeostasis during gut-derived neuroinflammation.
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16/100
Alzheimer’s disease (AD) is a multifactorial neurodegenerative disorder influenced by genetic, environmental, and lifestyle factors.
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Gut microbiome differences in Alzheimer’s disease (AD) are typically cataloged taxon by taxon, yet bacterial competition and cross-feeding make species’ roles dependent on the entire community.
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Gut microbial metabolites serve as functional mediators of gut–brain communication, linking microbial alterations to neurodegenerative pathology.
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Our findings provide evidence that age-related changes in the gut microbiota, particularly reductions in Bifidobacterium , partially mediate cognitive decline in older adults.
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Research on gut microbiota in Alzheimer’s disease (AD) has progressed from observational correlations to the validation of causal relationships.
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Alzheimer’s disease (AD) is a progressive neurodegenerative disorder characterized by amyloid-β plaques, neurofibrillary tau tangles, neuroinflammation, and progressive cognitive decline.
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DMII provides a metabolomics-based framework for assessing diet-related chronic inflammation using metabolomics data.
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Alzheimer's disease (AD) is incurable and increasingly attributed to gene-environment interactions.
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Alzheimer's disease (AD), the leading cause of dementia worldwide, imposes a growing clinical and societal burden, yet no therapies have been proven to alter its progression despite decades of intensive research.
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16/100
Structured Abstract INTRODUCTION Adults with Down syndrome (DS) have a higher risk of developing Alzheimer’s disease (AD).
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Alzheimer's disease (AD) is projected to become the highest-burden neurological disorder globally.
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Alzheimer's disease (AD) is a progressive neurodegenerative disorder with limited treatment options, underscoring the need for novel therapeutic targets.
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The gut-brain axis plays a critical role in maintaining neuronal integrity and influencing the progression of Alzheimer's disease (AD).
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43/100
Microbiota residing in the human gastrointestinal tract play a critical role in maintaining homeostasis through immune regulation, metabolic activity, and signaling to the central nervous system.
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Nutritional supplementation is emerging as a promising strategy to support clinical management of early Alzheimer's disease (AD), partly through modulation of the intestinal microbiome via the microbiota-gut-brain axis.
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Seventy-four participants completed the study.
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Previous studies in humans and humanized APOE mice have demonstrated APOE-genotype-specific differences in the GMB.
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pAD patients showed intestinal inflammation, an altered GM profile, blood changes in the tryptophan metabolism, and reduced glutamate levels compared with HC (p-value < 0.049).
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The gut-brain axis explains that changes in the intestinal microbiota influence Alzheimer's disease (AD).
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The microbiota-gut-brain axis (MGBA) is increasingly recognized as a critical regulator of brain health, influencing both neurodevelopment and age-related neurological decline.
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The microbiota-gut-brain axis (MGBA) is an intricate bidirectional communication network that links intestinal microbiota with the central nervous system (CNS) through immune, neural, endocrine, and metabolic pathways.
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66/100
Emerging evidence suggests a potential role of gut dysbiosis in neurodegenerative disorders and, in particular, Alzheimer's disease (AD) pathology and cognitive decline.
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43/100
This review explores the molecular processes underlying MGBA interactions, including vagus nerve signaling, systemic immunological responses, and metabolites produced by microorganisms.
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Recent studies increasingly highlight the complex interaction between gut microbiota and mental health, drawing attention to the role of the microbiota-gut-brain axis (MGBA) in the pathophysiology of mental and neurodevelopmental disorders.
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Alzheimer's disease (AD) is increasingly recognized as a condition shaped not only by central nervous system pathology but also by complex, bidirectional interactions between the gut, brain, and immune system.
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The gut microbiome plays a significant role in regulating gastrointestinal (GI) function and modulating the gut-brain axis, which describes the bidirectional communication between the GI tract and the central nervous system (CNS).
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We identified 59 key microbial species linked to MCI and AD biomarkers.
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The complex interrelation of gut microbiota with human health underlines the profound influence this microbial ecosystem has on mechanisms of disease and wellness.
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The human oral microbiome can affect brain functions directly through the trigeminal nerve and olfactory system and indirectly via the oral-gut-brain axis.
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The hallmarks of Alzheimer's disease (AD) include brain dysfunction and the buildup of amyloid and tau proteins.
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The gut microbiota, consisting of trillions of microorganisms, plays a critical role in regulating host physiology, including metabolism, immune responses, and brain function.
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Alzheimer's disease (AD) is a progressive, degenerative disorder of the central nervous system.
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Alzheimer's disease (AD) is a pervasive neurodegenerative disorder, and new approaches for its prevention and therapy are critically needed.
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Neurodegenerative diseases are severe, age-related conditions with complex etiologies that result in significant morbidity and mortality.
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Despite recent advances, severe acute pancreatitis (SAP) remains a lethal inflammation with limited treatment options.
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Microbes have inhabited the earth for hundreds of millions of years longer than humans.
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The gut microbiota plays a crucial role in the pathogenesis of AD through continuous bidirectional communication via the microbiota-gut-brain axis.
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Psen1 expression was regulated in experimental gut inflammation and in patients with IBD.
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Arteriosclerotic cerebral small vessel disease (aCSVD) is a major cause of stroke and dementia.
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Along with mounting evidence that gut microbiota and their metabolites migrate endogenously to distal organs, the 'gut-lung axis,' 'gut-brain axis,' 'gut-liver axis' and 'gut-renal axis' have been established.
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43/100
The gastrointestinal (GI) tract, home to the largest microbial population in the human body, plays a crucial role in overall health through various mechanisms.
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One of the most challenging and controversial issues in microbiome research is related to gut microbial metabolism and neuropsychological disorders.
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Several bacterial species belonging to the Proteobacteria (Escherichia coli) and Verrucomicrobia (Akkermansia muciniphila) phyla were positively associated with metformin treatment, while bacterial species belonging to the Firmicutes phylum (Romboutsia timonensis, Romboutsia ilealis) were negatively associated.
From across this tag
9 of the 273 studies in this tag, picked by a fixed rule (a hash of each study's address), not by score or date; each page of this list shows a different 9. The same 9 appear here every time.
- The Potential Role of Gut Microbiota in Alzheimer's Disease: From Diagnosis to Treatment
- Gut Microbiota Modification via Glucagon-like Peptide-1 with Beneficial Neuroprotective Effects
- Microbiota-derived short chain fatty acids modulate microglia and promote Aβ plaque deposition
- Intestinal microbial dysbiosis aggravates the progression of Alzheimer's disease in Drosophila
- Metabolites of soil microorganisms modulate amyloid β production in Alzheimer's neurons
- Western diet as a trigger of Alzheimer's disease: From metabolic syndrome and systemic inflammation to neuroinflammation and neurodegeneration
- The Microbiota-Gut-Brain Axis
- Bacteroides fragilis Lipopolysaccharide and Inflammatory Signaling in Alzheimer's Disease
- Relationship Between Plasma Neurofilament Light Chain, Gut Microbiota, and Dementia: A Cross-Sectional Study