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vascular
908 studies tagged vascular, dated 1975-09-01 to 2026-09-14. Bibliography: BibTeX (.bib) · RIS (.ris)
Newest in this topic
- Pathway-specific polygenic risk scores reveal underlying mechanisms of cerebral small vessel disease 2026-09-14
- Cerebral Small Vessel Disease and Risk of Cardiovascular Events and All-Cause Mortality: The Framingham Heart Study 2026-09-07
- Regional Distribution of Mineralized Blood Vessels in Alzheimer Disease and Clinicopathological Associations: A Multicenter Autopsy Study 2026-09-01
Studies
908 studies carry this tag.
Date range
1975 to 2026
Top journals
- Neurology 93
- Stroke 61
- Alzheimer s & Dementia 31
- Brain : a journal of neurology 20
- Annals of neurology 19
343 journals in all.
Newest first, then by address. Each study has a usefulness score out of 100 (rubric version 4.0); tap a score to see what makes it up. Tags were assigned automatically from each study's title and summary. The same studies as a filter in the library.
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39/100
Cerebral small vessel disease (cSVD) is a major contributor to stroke, dementia, and cognitive decline.
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Cerebral small vessel disease (cSVD) is associated with vascular cognitive impairment, dementia, and stroke.
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Frontotemporal lobar degeneration (FTLD) is the second most common early-onset dementia.
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In 3413 participants (mean age: 59 ± 14 years, 53.4% women), 11% had prevalent CVD or AF, 8% had prevalent CVD, and 4% had prevalent AF.
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= 0.009). Significant differences in annualized changes in these FA and MD values were found depending on the presence or absence of type 2 diabetes and smoking.
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Residing in communities characterized by socioeconomic disadvantage may confer risk for neurodegenerative brain changes and future neuropathology.
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Vascular dementia (VaD) ranks as the second most prevalent subtype of dementia, surpassed only by Alzheimer's disease (AD).
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), whereas the effect of region was only borderline-significant when comparing NAWM, WML peripheries and WML (p = 0.03) and non-significant for the other comparisons (p > 0.29).
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45/100
White matter hyperintensities (WMH) are associated with cortical thinning in distant brain regions.
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We included 71 DLB (76.8 ± 7.4 years old, 25 females) and 71 age- and sex-matched AD patients (and 75.2 ± 5.3 years old, 27 females).
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Cerebral small vessel disease (cSVD), a major contributor to stroke, cognitive decline, and vascular dementia, accounts for around 25% of ischemic strokes and significantly impacts age-related neurological disability.
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White matter hyperintensities (WMHs) are a common finding on magnetic resonance (MR) images in older individuals, appearing as high-signal intensity regions on fluid-attenuated inversion recovery (FLAIR) imaging.
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Previous studies have shown that preeclampsia increases the risk of cerebrovascular and cardiovascular diseases, as well as cognitive impairments, up to evolving dementia in the future.
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Compared with stage 0 CKM syndrome, stages 3 and 4 were significantly associated with an increased risk of dementia (hazard ratio [HR], 1.35; 95% confidence interval [CI], 1.14-1.59; HR, 1.80; 95% CI 1.57-2.07).
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Vascular dementia (VaD), the second-leading cause of dementia, is primarily a white matter ischemic disease with no direct therapies.
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The plasma PlGF level was significantly elevated in patients with SIVD compared with CUCs, and showed a high accuracy with an area under the curve of 0.893.
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Higher ARTS score was associated (all ps < 0.01) with incident MCI (hazard ratio [HR] = 1.29), dementia (HR = 1.33), and stroke (HR = 1.52) and with diastolic/systolic blood pressure, self-reported hypertension, claudication, congestive heart failure, smoking, and glomerular filtration rate in the combined sample.
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51/100
Our findings suggest that possible sarcopenia is a significant and independent predictor of delirium in older patients with acute stroke.
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The mean age of the participants was 68 ± 9 years and 39% were female.
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In Africa in 2021, 17·3 (95% UI 15·5-19·2) million DALYs were lost due to strokes, 17·6 (15·5-19·6) million were lost due to ischaemic heart diseases, and 1·8 (0·8-4·0) million were lost due to dementia.
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White matter hyperintensities (WMHs) and cerebral β-amyloid (Aβ) have been characterized as clinically significant biomarkers associated with greater cognitive decline and incidence of Alzheimer's Disease (AD) dementia.
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In total, 156 individuals were assessed for eligibility, and 79 (51%) were recruited (mean age 61.6 ± 6.9 (range 60-75) years; 68% male).
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Superficial white matter (SWM), containing short-range association connections beneath the cortex, has been affected in mild cognitive impairment and AD, with gaps in understanding the disease's early stages.
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We recruited 210 participants; 205 (66.8% male; aged 66.4±11.1 years) had useable data.
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It is often associated with recurrent ICH, seizures, and cognitive impairment.
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Thirty-two patients met the inclusion criteria (n=11 moderate-to-severe SVD, mean age 72 ± 10 years, 6 women; n = 21 none-to-minimal SVD, mean age 70 ± 10 years, 12 women).
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A total of 197 participants were included in the study.
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During a median follow-up period of 4.6 years, stroke, MACEs, and AD dementia occurred in 24, 36, and 21 patients, respectively.
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Sensitivity analyses were performed to validate the robustness of the linear regression results.
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The observation group had significantly higher blood uric acid levels than the control group (P<0.05).
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The sample included 740 donors (417 female [56.4%]; mean [SD] age at death, 81.5 [10.3] years; mean [SD] VBI score, 0.7 [1.0]; and median life course ADI, 7.08 [IQR, 4.33-9.83]), with residential years spanning 1905 to 2018.
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A total of 43,160 participants were included (mean [standard deviation] age, 64.2 [7.7] years, 53.1 % female).
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OCTA is a handy, noninvasive tool that enables the visualization of diminished retinal microvascular density in cases of early CSVD.
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Pulse pressure, but not mean arterial pressure, significantly mediated the relationship between age and white matter hyperintensity load.
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Pathophysiological differences separating small vessel disease (SVD) from Alzheimer's disease (AD) may alter treatment approach.
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Vascular dementia (VaD) is a leading cause of cognitive decline, closely associated with cerebrovascular endothelial cell (CEC) dysfunction, blood-brain barrier (BBB) disruption, and neuroinflammation.
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38/100
The incidence of dementia, and specifically, Alzheimer's disease, is higher in women than men, even in middle age, making it possible to rule out lifespan differences between men and women as a contributing factor.
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Among 8.5 million hospitalized patients aged 50 years or older, 2,335 had CAA and 600,646 had other CVDs.
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Cerebral small vessel disease is a condition caused by chronic cerebral hypoperfusion due to microvascular damage and is a major contributor to stroke and dementia.
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The glymphatic system, a vital brain perivascular network for waste clearance, hinges on the functionality of the aquaporin 4 (AQP4) water channel.
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Higher structural stiffness of the carotid artery is associated with cognitive decline, whereas both structural and load‐dependent stiffness are associated with brain structural abnormalities common in Alzheimer disease‐related dementias.
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The NLRP3 inflammasome pathway destroys neuronal cells primarily through the production of IL-18 and IL-1β.
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Our research, conducted both in vivo and ex vivo, demonstrated a 50% decrease in pressure-induced constriction in the CADASIL ACT zone.
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Vascular dementia is a highly heterogeneous neurodegenerative disorder induced by a variety of factors.
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Vascular dementia (VD), characterized by cognitive decline and behavioral disorders, has seen a rapid increase in prevalence in recent years.
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The global aging population has led to a significant rise in dementia and cognitive decline, with Alzheimer's disease as the primary cause.
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Serum IGF-1 concentration was significantly reduced in patients and rats with arteriosclerotic cSVD.
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49/100
= 0.143). LVI was significantly associated with incident ischemic stroke in the MI cohort (adjusted hazard ratio [HR] 1.82, 95% CI 1.08-3.09), but not in the CCA cohort.
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The coronavirus disease 2019 (COVID-19) pandemic, caused by severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2), highlighted the virus's impact on the central nervous system and its potential to exacerbate neurodegenerative diseases, similar to that in Alzheimer disease (AD).
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44/100
Vascular dementia (VaD) is a heterogeneous group of brain disorders caused by cerebrovascular pathologies and the second most common cause of dementia, accounting for over 20% of cases and posing an important global health concern.
From across this tag
10 of the 907 studies in this tag, picked by a fixed rule (a hash of each study's address), not by score or date; each page of this list shows a different 10. The same 10 appear here every time.
- Arterial Stiffness and Long-Term Risk of Health Outcomes: The Framingham Heart Study
- Metabolic and vascular contributions to dementia: Soluble epoxide hydrolase‐derived linoleic acid oxylipins and glycemic status are related to cerebral small vessel disease markers, atrophy, and cognitive performance
- Hypoxic-ischemic brain lesions found in asphyxiating neonates
- In vivo characterization of chronic traumatic encephalopathy using [F-18]FDDNP PET brain imaging
- Vascular Dementia: From Pathophysiology to Therapeutic Frontiers
- Heightened emotional contagion in mild cognitive impairment and Alzheimer's disease is associated with temporal lobe degeneration
- Association of Vascular Risk Factors and Genetic Factors With Penetrance of Variants Causing Monogenic Stroke
- Multimarker Cerebral Small Vessel Disease Score and Risk of Incident Dementia in the Framingham Heart Study
- White matter hyperintensity spatial patterns: Risk factors and clinical correlates
- String Vessel Formation is Increased in the Brain of Parkinson Disease