Each study has a usefulness score out of 100 (rubric version 4.0). Tap a score to see what makes it up.
Highest usefulness score first. Studies without a score come last.
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MetS and cognitive decline share common cardiometabolic pathways as MetS components can trigger cognitive impairment.
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Alzheimer's disease (AD), a type of dementia, is characterized by progressive memory decline and cognition impairment.
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In the brains of Apoeshl mice, the levels of apoM were lower, while those of ceramides were higher.
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The oral cavity is the initial chamber of digestive tract; the saliva swallowed daily contains an estimated 1.5 × 1012 oral bacteria.
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Evidence that the gut microbiota plays a key role in the pathogenesis of Alzheimer's disease is already unravelling.
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Given the complex bidirectional communication system that exists between the gut microbiome and the brain, there is growing interest in the gut microbiome as a novel and potentially modifiable risk factor for Alzheimer's disease (AD).
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Alzheimer's disease (AD), the most common neurodegenerative disorder, is accompanied by cognitive impairment and shows representative pathological features, including senile plaques and neurofibrillary tangles in the brain.
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Alzheimer's disease (AD) is a progressive neurodegenerative disease with high morbidity, disability, and fatality rate, significantly increasing the global burden of public health.
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Neuroinflammation is an early detectable marker of mild cognitive impairment, the transition state between normal cognition and dementia.
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Stimulation of glucagon-like peptide-1 (GLP-1) receptors increases the insulin release in the pancreas during high glucose levels, and also stimulates a feeling of satiety.
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The clearance of damaged or unwanted mitochondria by autophagy (also known as mitophagy) is a mitochondrial quality control mechanism postulated to play an essential role in cellular homeostasis, metabolism, and development and confers protection against a wide range of diseases.
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Extra-virgin olive oil (EVOO), a popular functional food and major source of fat in the Mediterranean diet, possesses a variety of healthful components, including monounsaturated fatty acids and bioactive phenolic compounds that, individually and collectively, exert beneficial effects on cardiometabolic markers of health and act as neuroprotective agents through their anti-inflammatory and antioxidant activities.
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Increasing evidence shows a close relationship between Alzheimer's disease (AD) and type 2 diabetes mellitus (T2DM).
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The intricate connection between central and enteric nervous systems is well established with emerging evidence linking gut microbiota function as a significant new contributor to gut-brain axis signaling.
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Frailty is a critical intermediate status of the aging process with a multidimensional and multisystem nature and at higher risk for adverse health-related outcomes, including falls, disability, hospitalizations, institutionalization, mortality, dementia, and Alzheimer's disease.
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Since decades, there is a change in concept of the gut-brain axis.
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This special Issue presents comprehensive and state-of-the-art advances in supporting the crucial role of the bidirectional interactions between the Brain-Gut Axis in health and diseases with an emphasis on the microbiome-gut-brain axis and its implications in variety of neurological disorders.
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The gut-brain axis refers to the bidirectional communication between the gut and brain, and regulates intestinal homeostasis and the central nervous system via neural networks and neuroendocrine, immune, and inflammatory pathways.
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Memory deterioration in Alzheimer's disease (AD) is thought to be underpinned by aberrant amyloid β (Aβ) accumulation, which contributes to synaptic plasticity impairment.
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Alzheimer's disease (AD) is often comorbid with other pathologies.
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Evidence linking the gut-brain axis to Alzheimer's disease (AD) is accumulating, but the characteristics of causally important microbes are poorly understood.
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Sleep plays a critical role in homeostasis of the body and mind.
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GLP-1 is derived from intestinal L cells, which takes effect through binding to GLP-1R and is inactivated by the enzyme dipeptidyl peptidase-4 (DPP-4).
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Prevention of cardiovascular events and regression of atherosclerotic changes are the primary aims of preventive cardiovascular medicine.
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Nutrition is known to play an important role in the pathogenesis of Alzheimer's disease.
From across this topic
10 of the 1,048 studies in this topic, picked by a fixed rule (a hash of each study's address), not by score or date; each page of this list shows a different 10. The same 10 appear here every time.
- Kynurenine pathway metabolites in humans: disease and healthy States
- Computerized cognitive training enhances cognitive function in Alzheimer's disease by downregulating Ruminococcus-TMAO pathway
- Targeting synaptic dysfunction in Alzheimer's disease by administering a specific nutrient combination
- Tetrahydrobiopterin biosynthesis, regeneration and functions
- Microbiota Signaling Pathways that Influence Neurologic Disease
- Oleuropein-Rich Olive Leaf Extract Attenuates Neuroinflammation in the Alzheimer's Disease Mouse Model
- Human gut microbiota: the links with dementia development
- Oxidative stress and mitochondrial dysfunction-linked neurodegenerative disorders
- ApoE isoform- and microbiota-dependent progression of neurodegeneration in a mouse model of tauopathy
- Neuroprotective Actions of Glucagon-Like Peptide-1 (GLP-1) Analogues in Alzheimer's and Parkinson's Diseases