Each study has a usefulness score out of 100 (rubric version 4.0). Tap a score to see what makes it up.
Highest usefulness score first. Studies without a score come last.
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Our MR analysis identified a notable association between genetically predicted PM2.5 (PM with a diameter of 2.5 μm or less) exposure and an elevated risk of AD (odds ratio, 2.160; 95% confidence interval, 1.481 to 3.149; p < 0.001).
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43/100
Environmental pollution exposures, including air, soil, water, light, and noise pollution, are critical issues that may implicate adverse mental health outcomes.
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This paper delves into the increasingly recognized yet complex relationship between air pollution and Neurological Diseases.
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Alzheimer's disease (AD) prevalence has increased in the last century which can be attributed to increased lifespan, but environment is also important.
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Here we show that two different PMs derived from exhausts of cars running on EN590 diesel or compressed natural gas (CNG) alter the function of human microglia-like cells in vitro.
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Respiratory viruses have a significant impact on health, as highlighted by the COVID-19 pandemic.
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43/100
The aging population worldwide is facing a significant increase in age-related non-communicable diseases, including cardiovascular and brain pathologies.
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43/100
Increased levels of ROS/RNS, generated from both endogenous (mitochondria, NADPH oxidases) and/or exogenous sources (radiation, certain drugs, foods, cigarette smoking, pollution) result in a harmful condition termed oxidative stress ("bad stress").
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Recent extensive evidence suggests that ambient fine particulate matter (PM2.5, with an aerodynamic diameter ≤2.5 μm) may be neurotoxic to the brain and cause central nervous system damage, contributing to neurodevelopmental disorders, such as autism spectrum disorders, neurodegenerative diseases, such as Alzheimer's disease and Parkinson's disease, and mental disorders, such as schizophrenia, depression, and bipolar disorder.
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Respirable fine particulate matter (PM 2.5 ) has been one of the most widely publicized indicators of pollution in recent years.
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Neurodegeneration leads to the loss of structural and functioning components of neurons over time.
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The increasing amount of particulate matter (PM) in the ambient air is a pressing public health issue globally.
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43/100
Fine particulate matter (PM2.5), a major component among air pollutants, highlights as a global health concern.
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Environmental exposure to neurotoxic metals and metalloids such as arsenic, cadmium, lead, mercury, or manganese is a global health concern affecting millions of people worldwide.
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Air pollution is regarded as an important risk factor for many diseases that affect a large proportion of the human population.
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These data demonstrate that chronic exposure to ambient TRAP promoted AD phenotypes in wildtype and genetically susceptible rats.
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43/100
Exposures to fine particulate matter PM2.5 and ozone O3 are associated with Alzheimer's disease (AD) risk.
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Environmental factors, including pesticides, have been linked to autism and neurodegeneration risk using retrospective epidemiological studies.
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Alzheimer's disease (AD) could result from a multifactorial process involving both genetic predisposition and exposure to environmental factors like pesticides.
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Epidemiological evidence demonstrates a link between air pollution exposure and the onset and progression of cognitive impairment and Alzheimer's disease (AD).
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Of the 7,511 non-demented participants at baseline, 545 developed dementia during a median follow-up of 7 years.
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The loss of olfactory stimulation correlates well with at least 68 widely differing neurological disorders, including depression, and we raise the possibility that this relationship may be causal.
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Traffic-related air pollution (TRAP) has been a common public health problem, which is associated with central nervous system dysfunction according to large-scale epidemiological studies.
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The adjusted model showed a significant inverse association of lithium levels with female, but not with male, or total prevalence of AD.
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The environmental role in disease progression has been appreciated for decades; however, understanding how airborne toxicant exposure can affect organs beyond the lungs is an underappreciated area of scientific inquiry.