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microbiota-gut-brain
100 studies tagged microbiota-gut-brain, dated 1995-01-01 to 2026-10-08. Bibliography: BibTeX (.bib) · RIS (.ris)
Newest in this topic
- The Gut–Liver–Brain Axis in Neurodegeneration: Unraveling Metabolic–Immune Crosstalk from Peripheral Sensing to Central Vulnerability 2026-10-08
- The microbiota-gut-brain axis in mental and neurodegenerative disorders: opportunities for prevention and intervention 2025-10-01
- Microbiota-gut-brain axis in neurodegenerative diseases: molecular mechanisms and therapeutic targets 2025-09-15
Studies
100 studies carry this tag.
Date range
1995 to 2026
Top journals
- Journal of neuroinflammation 4
- Frontiers in cellular and infection microbiology 3
- Frontiers in immunology 3
- Nutrients 3
- AIMS microbiology 2
86 journals in all.
Newest first, then by address. Each study has a usefulness score out of 100 (rubric version 4.0); tap a score to see what makes it up. Tags were assigned automatically from each study's title and summary. The same studies as a filter in the library.
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However, increasing evidence indicates that systemic metabolic dysfunction, chronic inflammation, intestinal dysbiosis, hepatic abnormalities, and barrier impairment may substantially influence disease susceptibility and progression.
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The microbiota-gut-brain axis (MGBA) is increasingly recognized as a critical regulator of brain health, influencing both neurodevelopment and age-related neurological decline.
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The microbiota-gut-brain axis (MGBA) is an intricate bidirectional communication network that links intestinal microbiota with the central nervous system (CNS) through immune, neural, endocrine, and metabolic pathways.
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43/100
This review explores the molecular processes underlying MGBA interactions, including vagus nerve signaling, systemic immunological responses, and metabolites produced by microorganisms.
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Recent studies increasingly highlight the complex interaction between gut microbiota and mental health, drawing attention to the role of the microbiota-gut-brain axis (MGBA) in the pathophysiology of mental and neurodevelopmental disorders.
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The gut microbiome plays a significant role in regulating gastrointestinal (GI) function and modulating the gut-brain axis, which describes the bidirectional communication between the GI tract and the central nervous system (CNS).
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The complex interrelation of gut microbiota with human health underlines the profound influence this microbial ecosystem has on mechanisms of disease and wellness.
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The human oral microbiome can affect brain functions directly through the trigeminal nerve and olfactory system and indirectly via the oral-gut-brain axis.
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The gut microbiota, consisting of trillions of microorganisms, plays a critical role in regulating host physiology, including metabolism, immune responses, and brain function.
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Neurodegenerative diseases are severe, age-related conditions with complex etiologies that result in significant morbidity and mortality.
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Microbes have inhabited the earth for hundreds of millions of years longer than humans.
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Along with mounting evidence that gut microbiota and their metabolites migrate endogenously to distal organs, the 'gut-lung axis,' 'gut-brain axis,' 'gut-liver axis' and 'gut-renal axis' have been established.
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One of the most challenging and controversial issues in microbiome research is related to gut microbial metabolism and neuropsychological disorders.
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Trillions of microbes live symbiotically in the host, specifically in mucosal tissues such as the gut.
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Neurodegenerative diseases are caused by the loss of neurons and/or their myelin sheaths, which deteriorate over time and become dysfunctional.
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The gut microbiome plays a pivotal role in maintaining human health, with numerous studies demonstrating that alterations in microbial compositions can significantly affect the development and progression of various immune-mediated diseases affecting both the digestive tract and the central nervous system (CNS).
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Inflammatory Bowel Disease (IBD), a chronic nonspecific intestinal inflammatory disease, is comprised of Ulcerative Colitis (UC) and Crohn's Disease (CD).
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The microbiota-gut-brain axis refers to the intricate bidirectional communication between commensal microorganisms residing in the digestive tract and the central nervous system, along neuroendocrine, metabolic, immune, and inflammatory pathways.
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Increasing research links the gut microbiome to neurodegenerative disorders.
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The gut microbiota (GM) plays an important role in the physiology and pathology of the host.
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The multifaceted microbiota characterizing our gut plays a crucial role in maintaining immune, metabolic and tissue homeostasis of the intestine as well as of distal organs, including the central nervous system.
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Clinical and animal studies have shown that gut microbiome disturbances can affect neural function and behaviors via the microbiota-gut-brain axis, and may be implicated in the pathogenesis of several brain diseases.
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Mitochondria are highly dynamic organelles that serve as the primary cellular energy-generating system.
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Microbial-derived metabolites are the intermediate or end products of bacterial digestion.
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Lewy body diseases, such as Parkinson's disease and dementia with Lewy bodies, vary in their clinical phenotype but exhibit the same defining pathological feature, α-synuclein aggregation.
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The intestinal barrier, which primarily consists of a mucus layer, an epithelial barrier, and a gut vascular barrier, has a crucial role in health and disease by facilitating nutrient absorption and preventing the entry of pathogens.
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Initial studies suggested that the fluctuations in the quantity, variety, and composition of the gut microbiota can significantly affect disease processes.
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The gut-brain axis describes a bidirectional interplay within the enteric environment between the intestinal epithelium, the mucosal immune system, and the microbiota with the enteric nervous system.
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Huntington's disease is a neurodegenerative disorder involving psychiatric, cognitive and motor symptoms.
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Hundreds of billions of commensal microorganisms live in and on our bodies, most of which colonize the gut shortly after birth and stay there for the rest of our lives.
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70/100
The results showed that intestinal expression of GLP-1 1 could reduce the intestinal pathogenic genus such as Enterobacteriaceae and was obviously associated with a greater number of beneficial genera such as Lactobacillus and Akkermansia.
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Emerging evidence suggests that both disruption of circadian rhythms and gut dysbiosis are closely related to aging-associated neurodegenerative diseases.
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It has been noticed in recent years that the unfavorable effects of the gut microbiota could exhaust host vigor and life, yet knowledge and theory are just beginning to be established.
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Over the last 10 years, there has been a growing interest in the relationship between gut microbiota, the brain, and neurologic-associated affections.
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Parkinson's disease (PD) is the second most common degenerative disease of the central nervous system (CNS) after Alzheimer's disease.
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The human body is full of an extensive number of commensal microbes, consisting of bacteria, viruses, and fungi, collectively termed the human microbiome.
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The intricate connection between central and enteric nervous systems is well established with emerging evidence linking gut microbiota function as a significant new contributor to gut-brain axis signaling.
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Since decades, there is a change in concept of the gut-brain axis.
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This special Issue presents comprehensive and state-of-the-art advances in supporting the crucial role of the bidirectional interactions between the Brain-Gut Axis in health and diseases with an emphasis on the microbiome-gut-brain axis and its implications in variety of neurological disorders.
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The gut-brain axis refers to the bidirectional communication between the gut and brain, and regulates intestinal homeostasis and the central nervous system via neural networks and neuroendocrine, immune, and inflammatory pathways.
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Recent research on the implications of gut microbiota on brain functions has helped to gather important information on the relationship between them.
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The human microbiota comprises trillions of symbiotic microorganisms and is involved in regulating gastrointestinal (GI), immune, nervous system and metabolic homeostasis.
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Clinicians are encouraged to consider patients' gut health when evaluating and treating psychiatric conditions, such as anxiety and depression.
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Administration of DSS induced colitis, impaired spatial and recognition memory, activated microglia, and increased A1-like astrocyte numbers.
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Many recent studies have shown that the gut microbiome plays important roles in human physiology and pathology.
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Emerging evidence indicates that gut microbiota is important in the regulation of brain activity and cognitive functions.
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There has been a steep increase in allergic and autoimmune diseases, reaching epidemic proportions and now affecting more than one billion people worldwide.
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The enteric nervous system (ENS) is the largest division of the peripheral nervous system and closely resembles components and functions of the central nervous system.
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The human digestive system is embedded with trillions of microbes of various species and genera.
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The microbiota-gut-brain axis is a bidirectional signaling mechanism between the gastrointestinal tract and the central nervous system.
From across this tag
7 of the 100 studies in this tag, picked by a fixed rule (a hash of each study's address), not by score or date; each page of this list shows a different 7. The same 7 appear here every time.
- The endotoxin hypothesis of neurodegeneration
- The gut microbiome in human neurological disease: A review
- Unhealthy gut, unhealthy brain: The role of the intestinal microbiota in neurodegenerative diseases
- The Gut Microbial Metabolite Trimethylamine-N-Oxide Is Present in Human Cerebrospinal Fluid
- Sex Differences in the Gut-Brain Axis: Implications for Mental Health
- Gut-brain Axis: Role of Lipids in the Regulation of Inflammation, Pain and CNS Diseases
- Gut microbiome in health and disease: Linking the microbiome-gut-brain axis and environmental factors in the pathogenesis of systemic and neurodegenerative diseases