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exposure
238 studies tagged exposure, dated 1994-07-01 to 2026-10-11. Bibliography: BibTeX (.bib) · RIS (.ris)
Newest in this topic
- Carbonaceous Components Underlying Wildfire Smoke PM 2.5 -Associated Alzheimer’s Disease and Related Dementias in Older US Adults 2026-10-11
- Micro- and nanoplastics alter electrophysiological brain patterns and reshape human neurodevelopmental trajectories 2026-09-24
- Neuroexposomics to Prevent Neurodegenerative Disease: Challenges, Next Steps, and Recommendations 2026-08-31
Studies
238 studies carry this tag.
Date range
1994 to 2026
Top journals
- Journal of Alzheimer's disease : JAD 24
- Environmental research 21
- Environment international 8
- Environmental health perspectives 8
- medRxiv 7
130 journals in all.
Newest first, then by address. Each study has a usefulness score out of 100 (rubric version 4.0); tap a score to see what makes it up. Tags were assigned automatically from each study's title and summary. The same studies as a filter in the library.
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We found no evidence that exposure to road traffic noise, either independently or in combination with traffic air pollution, was associated with risk of dementia in our study area.
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If the associations we observed are causal, then air pollution from residential wood burning, and air pollution from traffic, might be independent important risk factors for dementia.
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Air pollution is a multifaceted toxic chemical mixture capable of assaulting the central nervous system.
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As incidence of Alzheimer's disease (AD) and other neurodegenerative diseases rise, there is increasing interest in environmental factors which may contribute to disease onset and progression.
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For the first time, these results suggested that the effects of PM2.5 under AD context were possibly mediated by NLRP3 inflammasome activation, which was triggered by ROS.
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Current evidence for an association between ambient ozone exposure and mental health outcomes is inconclusive and further high quality studies are needed to assess any potential links given the strong biologic plausibility.
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Exposures to fine particulate matter (PM 2.5 ) and ozone (O 3 ) above USEPA standards are associated with Alzheimer's disease (AD) risk.
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Prolonged intake of excessive amounts of ethanol is known to have adverse effects on the central nervous system (CNS).
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Our findings suggest a potential protective role of green spaces on mental health (depression and anxiety) in adults, but further studies, especially longitudinal studies, are needed to provide further evidence of these benefits and of the mediation role of exposures like air pollution and noise.
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Increasing evidence points to an association of airborne pollutant exposure with respiratory, cardiovascular, and neurological pathology.
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A discussion with theoretical and possible future research directions is provided and it is clear that the need is not only of longitudinal population studies and of better knowledge of epigenetics markers but, especially, of environmental policy interventions based on the green economy.
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Inhalation exposure to DEE causes accelerated plaque formation and motor function impairment in 5XFAD transgenic mice.
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Our study shows that increasing long-term exposure to air pollution may increase the odds of depression and the use of antidepressants and benzodiazepines.
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The purpose of this review is to describe recent clinical and epidemiological studies examining the adverse effects of urban air pollution on the central nervous system (CNS).
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These results show TLR4 activation is integral in brain inflammatory responses to air pollution, and warrant further study of TLR4 in accelerated cognitive aging by air pollution.
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Traffic-related air pollution (TRAP) is associated with lower cognition and reduced white matter volume in older adults, specifically for particulate matter <2.5-μm diameter (PM 2.5 ).
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Millions of children and young adults are exposed to fine particulate matter (PM2.5) and ozone, associated with Alzheimer's disease (AD) risk.
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During the study period, there were 1175 DDE hospital admissions.
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Pesticides are a family of compounds which have brought many benefits to mankind in the agricultural, industrial, and health areas, but their toxicities in both humans and animals have always been a concern.
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Exposure to fine particulate matter (PM2.5) and ozone (O3) above US EPA standards is associated with Alzheimer's disease (AD) risk, while Mn toxicity induces parkinsonism.
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Air pollution is a major concern of new civilized world, which has a serious toxicological impact on human health and the environment.
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Evidence suggests that lifelong cumulative exposure to pesticides may generate lasting toxic effects on the central nervous system and contribute to the development of Alzheimer's disease (AD).
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We also identified several common challenges.
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We observed a significant association between blood cadmium levels and AD mortality among older adults in the US.
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Among nondiabetics, short- and medium-term PM2.5 were associated with higher FBG.
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Environmental factors, including pesticides, have been linked to autism and neurodegeneration risk using retrospective epidemiological studies.
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Long-term exposures to air pollution and traffic noise were positively associated with MCI, mainly with the amnestic subtype.
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Millions of urban children are chronically exposed to high concentrations of air pollutants, i.e., fine particulate matter (PM2.5) and ozone, associated with increased risk for Alzheimer's disease.
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Air pollution (indoors and outdoors) is a major issue in public health as epidemiological studies have highlighted its numerous detrimental health consequences (notably, respiratory and cardiovascular pathological conditions).
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The central nervous system is emerging as an important target for adverse health effects of air pollution, where it may contribute to neurodevelopmental and neurodegenerative disorders.
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Endocrine disrupting chemicals (EDC) are released into environment from different sources.
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Organophosphate pesticides (OPs) are used extensively throughout the world.
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PM2.5 exposure may contribute to WM loss in older women.
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In humans, olfactory dose of inhaled nanoparticles is highest for 1-2 nm particles with ∼1% of inhaled particles depositing in the olfactory region.
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Our findings raise concerns about potential effects of air pollution from traffic and other sources on the risk of PD, particularly in populations with high or increasing exposures.
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40/100
We found strong evidence of association for all three outcomes.
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Long-term exposure to the highest tertile of PM10 or ozone was significantly associated with an increased risk of AD and VaD.
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Neurodegenerative diseases including Alzheimer (AD) and Parkinson (PD) have attracted attention in last decades due to their high incidence worldwide.
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Millions of children are exposed to concentrations of air pollutants, including fine particulate matter (PM2.5), above safety standards.
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Children's urban air pollution exposures result in systemic and brain inflammation and the early hallmarks of Alzheimer's disease (AD).
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Several studies with animal research associate air pollution in Alzheimer's disease (AD) neuropathology, but the actual impact of air pollution on the risk of AD is unknown.
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Alzheimer's dis ease (AD) is a leading cause of mortality in the developed world with 70% risk attributable to genetics.
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The chronic health effects associated with sustained exposures to high concentrations of air pollutants are an important issue for millions of megacity residents and millions more living in smaller urban and rural areas.
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The results of this large retrospective, population-based study indicate that exposure to NO2 and CO is associated with an increased risk of dementia in the Taiwanese population.
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Between 1961 and 1971, military herbicides were used by the United States and allied forces for military purposes.
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Decreased olfactory function is very common in the older population, being present in over half of those between the ages of 65 and 80 years and in over three quarters of those over the age of 80 years.
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The etiology of most neurodegenerative disorders is multifactorial and consists of an interaction between environmental factors and genetic predisposition.
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Glyphosate, the active ingredient in Roundup®, is the most popular herbicide used worldwide.
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There has been a steep increase in the prevalence of dementia in recent decades, which has roughly followed an increase in pesticide use some decades earlier, a time when it is probable that current dementia patients could have been exposed to pesticides.
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Along with the wide use of pesticides in the world, the concerns over their health impacts are rapidly growing.
From across this tag
7 of the 238 studies in this tag, picked by a fixed rule (a hash of each study's address), not by score or date; each page of this list shows a different 7. The same 7 appear here every time.
- Urban air pollution: influences on olfactory function and pathology in exposed children and young adults
- Air pollution and brain damage
- Neurotoxicity of Diesel Exhaust Particles
- Association between air pollution and CSF sTREM2 in cognitively normal older adults: The CABLE study
- Fine Particulate Matter and Dementia Incidence in the Adult Changes in Thought Study
- Human Health and Ocean Pollution
- Health Impacts from Ambient Particle Exposure in Southern Sweden