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exposure
238 studies tagged exposure, dated 1994-07-01 to 2026-10-11. Bibliography: BibTeX (.bib) · RIS (.ris)
Newest in this topic
- Carbonaceous Components Underlying Wildfire Smoke PM 2.5 -Associated Alzheimer’s Disease and Related Dementias in Older US Adults 2026-10-11
- Micro- and nanoplastics alter electrophysiological brain patterns and reshape human neurodevelopmental trajectories 2026-09-24
- Neuroexposomics to Prevent Neurodegenerative Disease: Challenges, Next Steps, and Recommendations 2026-08-31
Studies
238 studies carry this tag.
Date range
1994 to 2026
Top journals
- Journal of Alzheimer's disease : JAD 24
- Environmental research 21
- Environment international 8
- Environmental health perspectives 8
- medRxiv 7
130 journals in all.
Newest first, then by address. Each study has a usefulness score out of 100 (rubric version 4.0); tap a score to see what makes it up. Tags were assigned automatically from each study's title and summary. The same studies as a filter in the library.
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Between Jan 1, 2000, and Dec 31, 2016, of 63 038 019 individuals who were aged 65 years or older during the study period, we identified 1·0 million cases of Parkinson's disease and 3·4 million cases of Alzheimer's disease and related dementias based on primary and secondary diagnosis billing codes.
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The present review aims to provide an update on health risks associated with the low-to-moderate levels of environmental cadmium (Cd) and lead (Pb) to which most populations are exposed.
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45/100
We observed significant increases in all-cause hospital admissions (2.53% in PD and 2.49% in AD/dementia) attributable to high PM2.5 exposure.
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Climate change is one of the biggest challenges humanity is facing in the 21st century.
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54/100
Increases in ambient PM2.5 concentrations were not consistently associated with increased hospital admissions rates.
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Air pollution is an important cause of non-communicable diseases globally with particulate matter (PM) as one of the main air pollutants.
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A health impact assessment (HIA) is an important tool for making informed decisions regarding the design and evaluation of environmental interventions.
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50/100
Among the compounds detected in greater abundance in the CSF of patients with DLB compared with patients with AD, only one had a xenobiotic profile potentially related to the pathophysiology of DLB.
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The vast majority of neurodegenerative disease cannot be attributed to genetic causes alone and as a result, there is significant interest in identifying environmental modifiers of disease risk.
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Among 218 participants with 469 AF episodes we observed higher odds of AF following higher 24-hour mean levels of PM10 and O3, reaching statistical significance for PM10 levels averaged over the previous 12-24-hours [OR 1.10 (95%CI 1.01-1.19) per IQR of PM10 (7.8 µg/m3)].
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Increased exposure to urban green space was associated with reduced incidence of dementia and stroke.
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Environmental toxicants have been implicated in neurodegenerative diseases, and pesticide exposure is a suspected environmental risk factor for Alzheimer's disease (AD).
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Neurodegenerative disorders are typically sporadic in nature in addition to usually influenced through an extensive range of environmental factors, lifestyle, and genetic elements.
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Recent extensive evidence indicates that air pollution, in addition to causing respiratory and cardiovascular diseases, may also negatively affect the brain and contribute to central nervous system diseases.
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In cognitively unimpaired adults with increased risk for AD, increased exposure to air pollution was suggested to be associated with greater global atrophy and reduced volume and thickness in specific brain areas known to be affected in AD, thus suggesting a potential link between environmental exposures and cerebral vulnerability to AD.
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Pesticides are widely-used chemicals commonly applied in agriculture for the protection of crops from pests.
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Exposure to air pollutants is associated with an increased risk of developing Alzheimer's disease (AD).
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Air pollution exposure may exacerbate AD development.
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Epidemiological studies are associating elevated exposure to air pollution with increased risk of Alzheimer's disease and other neurodegenerative disorders.
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Alzheimer's disease and related dementias lack effective treatment or cures and are major public health challenges.
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In this non-demented study population, of which 62% were cognitively normal and 38% were in mild cognitive impairment state, exposure to the highest tertile of PM10mean was associated with increased risk of amyloid-β (Aβ) positivity (odds ratio 2.19, 95% confidence interval 1.13 to 4.26) even after controlling all potential confounders.
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As one of the most harmful air pollutants, fine particulate matter (PM2.5) has been implicated as a risk factor for multiple diseases, which has generated widespread public concern.
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39/100
Alzheimer's disease (AD), an aging-related neurodegenerative disease, is a major cause of dementia in the elderly.
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Higher TRAP-NOx was associated with incident dementia (HR = 1.55 for the highest versus lower tertiles, 95% CI = 1.04, 2.55).
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Traffic-related air pollution particulate matter (TRAP-PM) is associated with increased risk of Alzheimer Disease (AD).
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63/100
Evidence regarding the association of specific air pollutants with vascular dementia (VaD) risk is limited.
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Airborne particulate matter poses a serious threat to human health.
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62/100
Our results showed a positive association between exposure to NOx and O 3 and hospitalization for dementia and a negative association between NO 2 exposure and hospitalization for dementia.
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Air pollution is a modifiable and preventable factor, and it is a possible risk factor for dementia.
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The third part of the DGP statement introduces the current body of knowledge on less studied health outcomes associated with exposure to ambient air pollution: the negative impact on metabolism leading to impaired glucose tolerance and diabetes as well as contribution to the development of neurodegenerative disorders and delayed cognitive function in children.
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Air pollution is a risk factor for cardiovascular and Alzheimer's disease (AD).
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39/100
In these models, PM2.5 concentration was associated with smaller left hippocampal volume.
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Exposure to ambient air pollution and noise is ubiquitous globally.
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PM2.5 particles in air pollution have been widely considered associated with respiratory and cardiovascular diseases.
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Based on 19 studies the weighted RR for occupational exposure to EMFs was 1.26 (95% confidence interval (CI) 1.07⁻1.50) for ALS, 1.33 (95% CI 1.07⁻1.64) for Alzheimer's disease and 1.02 (95% CI 0.83⁻1.26) for Parkinson's disease.
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As the median age of the population increases, the number of individuals with Alzheimer's disease (AD) and the associated socio-economic burden are predicted to worsen.
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Evidence is emerging that greater exposure to airborne pollutants is associated with increased risk of dementia.
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Fewer than 5% of Alzheimer's disease (AD) cases are demonstrably directly inherited, indicating that environmental factors may be important in initiating and/or promoting the disease.
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Dementia has become a major public health concern around the world.
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Among cognitively normal individuals, a 5 μg/m3 increase in 7-day and 1-year average PM2.5 was associated with elevated VCAM-1 (7-day: 35.4 (9.7, 61.1) ng/ml; 1-year: 51.8 (6.5, 97.1) ng/ml).
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43/100
Exposures to fine particulate matter PM2.5 and ozone O3 are associated with Alzheimer's disease (AD) risk.
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The associations of particulate matter (PM) pollution with the morbidity of overall and subtypes of mental disorders (MDs), as well as the corresponding morbidity burden, remain understudied, especially in developing countries.
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Air pollution is one of the greatest public health threats worldwide.
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In this article the neurotoxic disorders appearing in patients exposed to organophosphorus pesticides and known mechanisms involved are reviewed.
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71/100
We have found evidence of a positive association between residential levels of air pollution across London and being diagnosed with dementia, which is unexplained by known confounding factors.
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38/100
Poisoning from pesticides is a global public health problem and accounts for nearly 300,000 deaths worldwide every year.
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An emerging body of evidence has raised concern regarding the potentially harmful effects of inhaled pollutants on the central nervous system during the last decade.
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APOE ε4 allele carriers had significantly higher behavior problem scores than noncarriers, and adverse associations with PAHs and NO2 were stronger or limited to ε4 carriers for behavior problem scores (P-interaction 0.03 and 0.04), caudate volume (P-interaction 0.04 and 0.03), and inattentiveness trajectories (P-interaction 0.15 and 0.08, respectively).
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Organophosphorus (OP) compounds have been known as the most widely used pesticides during the past half century and there have been a huge body of literature regarding their association with human chronic diseases.
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There is growing evidence that air pollution is a risk factor for a number of neurodegenerative diseases, most notably Alzheimer's (AD) and Parkinson's (PD).
From across this tag
8 of the 238 studies in this tag, picked by a fixed rule (a hash of each study's address), not by score or date; each page of this list shows a different 8. The same 8 appear here every time.
- Rapid doubling of Alzheimer's amyloid-β40 and 42 levels in brains of mice exposed to a nickel nanoparticle model of air pollution
- Long-term exposure to residential green and blue spaces and anxiety and depression in adults: A cross-sectional study
- PM2.5 exposure exacerbates Alzheimer's disease pathology through lysosomal dysfunction in APP/PS1 mice
- Cerebrospinal Fluid Biomarkers in Highly Exposed PM2.5 Urbanites: The Risk of Alzheimer's and Parkinson's Diseases in Young Mexico City Residents
- Carbonaceous Components Underlying Wildfire Smoke PM 2.5 -Associated Alzheimer’s Disease and Related Dementias in Older US Adults
- Environmental pollution and extreme weather conditions: insights into the effect on mental health
- Impact of Air Pollution on Neurological and Psychiatric Health
- Air Pollution and the Risk of Parkinson's Disease: A Review