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exposure
237 studies tagged exposure, dated 1994-07-01 to 2026-10-11. Bibliography: BibTeX (.bib) · RIS (.ris)
Newest in this topic
- Carbonaceous Components Underlying Wildfire Smoke PM 2.5 -Associated Alzheimer’s Disease and Related Dementias in Older US Adults 2026-10-11
- Micro- and nanoplastics alter electrophysiological brain patterns and reshape human neurodevelopmental trajectories 2026-09-24
- Neuroexposomics to Prevent Neurodegenerative Disease: Challenges, Next Steps, and Recommendations 2026-08-31
Studies
237 studies carry this tag.
Date range
1994 to 2026
Top journals
- Journal of Alzheimer's disease : JAD 24
- Environmental research 21
- Environment international 8
- Environmental health perspectives 8
- medRxiv 7
130 journals in all.
Newest first, then by address. Each study has a usefulness score out of 100 (rubric version 4.0); tap a score to see what makes it up. Tags were assigned automatically from each study's title and summary. The same studies as a filter in the library.
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These effects on elevation of brain Aβ exceed those associated with trisomy 21, a known risk for early onset AD pathology, raising the possibility that clinical importance might be attached.
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Alzheimer's disease (AD) could result from a multifactorial process involving both genetic predisposition and exposure to environmental factors like pesticides.
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Several lines of evidence indicate that the etiology of late-onset Alzheimer's disease (LOAD) is complex, with significant contributions from both genes and environmental factors.
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Alzheimer's disease (AD) is the most common form of dementia affecting millions of people worldwide and will continue to affect millions more with population aging on the rise.
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Exposure to ambient air pollution is a serious and common public health concern associated with growing morbidity and mortality worldwide.
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Air pollution exposures have been linked to neuroinflammation and neuropathology.
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The data support that the midbrain may be more sensitive to the neuroinflammatory effects of subchronic air pollution exposure.
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Preliminary studies have shown associations between chronic pesticide exposure in occupational settings and neurological disorders.
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Human biomonitoring provides an efficient and cost-effective way to identify and quantify exposure to chemical substances, including those having deleterious eff ects on human organisms.
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614 subjects were available for investigation at follow-up.
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Our results indicate that different brain regions may be uniquely responsive to changes induced by exposure to DEE.
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Oxidative stress is involved in many neurodegenerative diseases.
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Air pollution has been implicated as a chronic source of neuroinflammation and reactive oxygen species (ROS) that produce neuropathology and central nervous system (CNS) disease.
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These data suggest that beta-HCH is associated with a diagnosis of PD.
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Mexico City (MC) residents are exposed to severe air pollution and exhibit olfactory bulb inflammation.
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36/100
In the present review we summarize recent advances in the understanding of the interaction between genetics and environmental factors involved in complex multi-factorial neurodegenerative disorders such as Alzheimer's disease (AD), Parkinson's disease (PD) and Amyotrophic Lateral Sclerosis (ALS).
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Humans are routinely exposed to a vast array of environmental neurotoxicants, including pesticides, endocrine disrupters, and heavy metals.
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Air pollution is a serious environmental problem.
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Significantly, technological advancement has also changed the character of particulate pollution, increasing the proportion of nanometer-sized particles--"nanoparticles"--and expanding the variety of chemical compositions.
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In January 2004, the normally picturesque Cache Valley in northern Utah made national headlines with the highest PM2.5 levels in the nation.
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The reviewed studies indicate that genetic predisposition for disease is best predicted in the context of environmental exposures.
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Parkinson's disease (PD) is the most common cause of the parkinsonian syndromes and the most frequent neurodegenerative disease after Alzheimer's disease.
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Alzheimer's disease (AD) is a progressive neurodegenerative disease that affects millions in the aging population worldwide and will affect millions more in the next 20 years.
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Epidemiological evidence of an association between Alzheimer's disease (AD) and the most frequently studied occupational exposures--pesticides, solvents, electromagnetic fields (EMF), lead and aluminium--is inconsistent.
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The association of pesticide exposure with Parkinson's disease suggests a causative role.
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Exposures to particulate matter and gaseous air pollutants have been associated with respiratory tract inflammation, disruption of the nasal respiratory and olfactory barriers, systemic inflammation, production of mediators of inflammation capable of reaching the brain and systemic circulation of particulate matter.
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Support was observed for hypothesized excess neurodegenerative disease associated with a variety of occupations, 60 Hz magnetic fields and welding.
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36/100
Air pollution is a complex mixture of gases (e.g., ozone), particulate matter, and organic compounds present in outdoor and indoor air.
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The built environment has direct and indirect effects on mental health.
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Acute, subchronic, or chronic exposures to particulate matter (PM) and pollutant gases affect people in urban areas and those exposed to fires, disasters, and wars.
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The authors investigated the hypothesis that exposure to pesticides could be related to central nervous system disorders in a prospective cohort study of 1,507 French elderly (1992-1998).
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Exposure to complex mixtures of air pollutants produces inflammation in the upper and lower respiratory tract.
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The objectives of the study were (1) to determine whether concentrations of particles in the ambient air of Montreal, Quebec, were associated with daily all-cause and cause-specific mortality in the period 1984 to 1993, and (2) to determine whether groups of the population had higher than average risks of death from exposure to particles.
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36/100
Neurodegenerative disease occurs more frequently in some occupations than in others, and this distribution, which may indicate occupational risk factors, should be further investigated.
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Hyper-aluminemia often develops in patients with renal failure being treated with intermittent hemodialysis on a chronic basis, and in severe cases results in an encephalopathy.
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We reviewed 46 published reports associating cigarette smoking and Parkinson's disease.
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Epidemiological studies have suggested an etiologic relationship between pesticide exposure and Parkinson's disease (PD).
From across this tag
7 of the 237 studies in this tag, picked by a fixed rule (a hash of each study's address), not by score or date; each page of this list shows a different 7. The same 7 appear here every time.
- Olfactory deposition of inhaled nanoparticles in humans
- Role of Environmental Toxicants on Neurodegenerative Disorders
- Air Pollution, Health, and Economics: Evaluating the Impact of WHO targets and Guideline Values on Mortality and Morbidity in Low- and Middle-Income Countries
- Mediation of the relationship between air pollution and dementia: A UK Biobank study
- PM2.5 and hospital admissions among Medicare enrollees with chronic debilitating brain disorders
- Impact of air pollution and climate change on mental health outcomes: an umbrella review of global evidence
- Emerging risk of environmental factors: insight mechanisms of Alzheimer's diseases