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Home › Research › Topics › transgenic

transgenic

1,157 studies tagged transgenic, dated 1986-01-01 to 2026-09-25. Bibliography: BibTeX (.bib) · RIS (.ris)

Newest in this topic

  1. What Do Sugar, Gila Monsters, and the Therapeutic Dance Have in Common With GLP-1RA, Stroke, and Dementia? 2026-09-25
  2. Myo-inositol as a spectroscopic marker of neuroinflammation in frontotemporal lobar degeneration 2026-09-20
  3. Laromestrocel preserves brain volume and cognitive function in Alzheimers disease by inhibiting neuroinflammation 2026-09-17

Studies

1,157 studies carry this tag.

Date range

1986 to 2026

Top journals

  • The Journal of biological chemistry 109
  • The Journal of neuroscience : the official journal of the Society for Neuroscience 42
  • Proceedings of the National Academy of Sciences of the United States of America 39
  • Neuron 28
  • Nature 23

363 journals in all.

Newest first, then by address. Each study has a usefulness score out of 100 (rubric version 4.0); tap a score to see what makes it up. Tags were assigned automatically from each study's title and summary. The same studies as a filter in the library.

  1. Ubiquitinated pathological lesions in frontotemporal lobar degeneration contain the TAR DNA-binding protein, TDP-43

    ✓ Peer-reviewed alzhub usefulness score 54 out of 100. Acta neuropathologica 2007-01-12 Brain tissue and pathology

    We have investigated the extent and pattern of immunostaining for the TAR DNA-binding protein, TDP-43, in 37 patients with frontotemporal lobar degeneration with ubiquitin (UBQ) pathology (FTLD-U).

  2. Nicotinic acetylcholine receptors and nicotinic cholinergic mechanisms of the central nervous system

    ✓ Peer-reviewed alzhub usefulness score 48 out of 100. Annual review of pharmacology and toxicology 2007-01-01 Disparities

    Subtypes of neuronal nicotinic acetylcholine receptors (nAChRs) are constructed from numerous subunit combinations that compose channel-receptor complexes with varied functional and pharmacological characteristics.

  3. Perinatal iron deficiency results in altered developmental expression of genes mediating energy metabolism and neuronal morphogenesis in hippocampus

    ✓ Peer-reviewed alzhub usefulness score 42 out of 100. Hippocampus 2007-01-01 Brain scans and imaging

    The human and rat hippocampus is highly susceptible to iron deficiency (ID) during the late fetal, early neonatal time period which is a peak time of regulated brain iron uptake and utilization.

  4. The peripheral benzodiazepine receptor (Translocator protein 18kDa) in microglia: from pathology to imaging

    ✓ Peer-reviewed alzhub usefulness score 45 out of 100. Progress in neurobiology 2006-12-06 Inflammation and the immune system

    Microglia constitute the primary resident immune surveillance cell in the brain and are thought to play a significant role in the pathogenesis of several neurodegenerative disorders, such as Alzheimer's disease, multiple sclerosis, Parkinson's disease and HIV-associated dementia.

  5. Isoflurane-induced apoptosis: a potential pathogenic link between delirium and dementia

    ✓ Peer-reviewed alzhub usefulness score 36 out of 100. The journals of gerontology. Series A, Biological sciences and medical sciences 2006-12-01 Clinical safety

    The demonstration that isoflurane contributes to well-described mechanisms of Alzheimer's neuropathogenesis provides a plausible link between the acute effects of anesthesia, a well-described risk factor for delirium, and the more long-term sequelae of dementia.

  6. Activation of beta2-adrenergic receptor stimulates gamma-secretase activity and accelerates amyloid plaque formation

    ✓ Peer-reviewed alzhub usefulness score 50 out of 100. Nature medicine 2006-11-19 Brain tissue and pathology

    Amyloid plaque is the hallmark and primary cause of Alzheimer disease.

  7. Upregulated expression of purinergic P2X(7) receptor in Alzheimer disease and amyloid-beta peptide-treated microglia and in peptide-injected rat hippocampus

    ✓ Peer-reviewed alzhub usefulness score 45 out of 100. Journal of neuropathology and experimental neurology 2006-11-01 Inflammation and the immune system

    The expression of the purinergic receptor subtype P2X(7)R, a nonselective cationic channel activated by high levels of adenosine triphosphate (ATP), has been studied in adult microglia obtained from Alzheimer disease (AD) and nondemented (ND) brains, in fetal human microglia exposed to Abeta(1-42) peptide and in vivo in Abeta(1-42)-injected rat hippocampus.

  8. TDP-43 is a component of ubiquitin-positive tau-negative inclusions in frontotemporal lobar degeneration and amyotrophic lateral sclerosis

    ✓ Peer-reviewed alzhub usefulness score 56 out of 100. Biochemical and biophysical research communications 2006-10-30 Lewy body dementia

    Ubiquitin-positive tau-negative neuronal cytoplasmic inclusions and dystrophic neurites are common pathological features in frontotemporal lobar degeneration (FTLD) with or without symptoms of motor neuron disease and in amyotrophic lateral sclerosis (ALS).

  9. Chronic lithium administration to FTDP-17 tau and GSK-3beta overexpressing mice prevents tau hyperphosphorylation and neurofibrillary tangle formation, but pre-formed neurofibrillary tangles do not revert

    ✓ Peer-reviewed alzhub usefulness score 36 out of 100. Journal of neurochemistry 2006-10-24 Disease mechanisms

    Glycogen synthase kinase-3 (GSK-3) has been proposed as the main kinase able to aberrantly phosphorylate tau in Alzheimer's disease (AD) and related tauopathies, raising the possibility of designing novel therapeutic interventions for AD based on GSK-3 inhibition.

  10. Reduction of soluble Abeta and tau, but not soluble Abeta alone, ameliorates cognitive decline in transgenic mice with plaques and tangles

    ✓ Peer-reviewed alzhub usefulness score 36 out of 100. The Journal of biological chemistry 2006-10-20 Disease mechanisms

    Increasing evidence points to soluble assemblies of aggregating proteins as a major mediator of neuronal and synaptic dysfunction.

  11. Cdk5 is involved in NFT-like tauopathy induced by transient cerebral ischemia in female rats

    ✓ Peer-reviewed alzhub usefulness score 36 out of 100. Biochimica et biophysica acta 2006-10-18 Disease mechanisms

    Although neurofibrillary tangle (NFT) formation is a central event in both familial and sporadic Alzheimer's disease (AD), neither cellular origin nor functional consequence of the NFTs are fully understood.

  12. The HIV Env variant N283 enhances macrophage tropism and is associated with brain infection and dementia

    ✓ Peer-reviewed alzhub usefulness score 45 out of 100. Proceedings of the National Academy of Sciences of the United States of America 2006-10-02 Inflammation and the immune system

    HIV infects tissue macrophages and brain microglia, which express lower levels of CD4 and CCR5 than CD4+ T cells in peripheral blood.

  13. Ubiquitinated TDP-43 in frontotemporal lobar degeneration and amyotrophic lateral sclerosis

    ✓ Peer-reviewed alzhub usefulness score 62 out of 100. Science (New York, N.Y.) 2006-10-01 Lewy body dementia

    Ubiquitin-positive, tau- and alpha-synuclein-negative inclusions are hallmarks of frontotemporal lobar degeneration with ubiquitin-positive inclusions and amyotrophic lateral sclerosis.

  14. Apolipoprotein E and low density lipoprotein receptor-related protein facilitate intraneuronal Abeta42 accumulation in amyloid model mice

    ✓ Peer-reviewed alzhub usefulness score 45 out of 100. The Journal of biological chemistry 2006-09-29 Genetics

    The low density lipoprotein receptor-related protein (LRP) is highly expressed in the brain and has been shown to alter the metabolism of amyloid precursor protein and amyloid-beta peptide (Abeta) in vitro.

  15. Expression profiles for macrophage alternative activation genes in AD and in mouse models of AD

    ✓ Peer-reviewed alzhub usefulness score 46 out of 100. Journal of neuroinflammation 2006-09-27 Risk genes beyond APOE

    Microglia are associated with neuritic plaques in Alzheimer disease (AD) and serve as a primary component of the innate immune response in the brain.

  16. Dyrk1A phosphorylates alpha-synuclein and enhances intracellular inclusion formation

    ✓ Peer-reviewed alzhub usefulness score 44 out of 100. The Journal of biological chemistry 2006-09-07 Down syndrome and Alzheimer's

    Lewy bodies (LBs) are pathological hallmarks of Parkinson disease (PD) but also occur in Alzheimer disease (AD) and dementia of LBs.

  17. Mutations in progranulin are a major cause of ubiquitin-positive frontotemporal lobar degeneration

    ✓ Peer-reviewed alzhub usefulness score 59 out of 100. Human molecular genetics 2006-09-01 Lewy body dementia

    Null mutations in the progranulin gene (PGRN) were recently reported to cause tau-negative frontotemporal dementia linked to chromosome 17.

  18. Differential effects of mitochondrial heat shock protein 60 and related molecular chaperones to prevent intracellular beta-amyloid-induced inhibition of complex IV and limit apoptosis

    ✓ Peer-reviewed alzhub usefulness score 43 out of 100. The Journal of biological chemistry 2006-08-03 Risk genes beyond APOE

    Defects in mitochondrial oxidative metabolism, in particular decreased activity of cytochrome c oxidase, have been reported in Alzheimer disease tissue and in cultured cells that overexpress amyloid precursor protein.

  19. beta-Amyloid-induced dynamin 1 degradation is mediated by N-methyl-D-aspartate receptors in hippocampal neurons

    ✓ Peer-reviewed alzhub usefulness score 36 out of 100. The Journal of biological chemistry 2006-07-24 Disease mechanisms

    Alzheimer disease (AD) is a progressive, neurodegenerative disorder that leads to debilitating cognitive deficits.

  20. Phosphorylation of Ser-129 is the dominant pathological modification of alpha-synuclein in familial and sporadic Lewy body disease

    ✓ Peer-reviewed alzhub usefulness score 55 out of 100. The Journal of biological chemistry 2006-07-17 Lewy body dementia

    A comprehensive, unbiased inventory of synuclein forms present in Lewy bodies from patients with dementia with Lewy bodies was carried out using two-dimensional immunoblot analysis, novel sandwich enzyme-linked immunosorbent assays with modification-specific synuclein antibodies, and mass spectroscopy.

  21. Null mutations in progranulin cause ubiquitin-positive frontotemporal dementia linked to chromosome 17q21

    ✓ Peer-reviewed alzhub usefulness score 64 out of 100. Nature 2006-07-16 Lewy body dementia

    Frontotemporal dementia (FTD) with ubiquitin-immunoreactive neuronal inclusions (both cytoplasmic and nuclear) of unknown nature has been linked to a chromosome 17q21 region (FTDU-17) containing MAPT (microtubule-associated protein tau).

  22. Contribution of presenilin transmembrane domains 6 and 7 to a water-containing cavity in the gamma-secretase complex

    ✓ Peer-reviewed alzhub usefulness score 43 out of 100. The Journal of biological chemistry 2006-07-14 Risk genes beyond APOE

    Gamma-secretase is a multiprotein complex responsible for the intramembranous cleavage of the amyloid precursor protein and other type I transmembrane proteins.

  23. In vitro analysis of mouse neural stem cells genetically modified to stably express human NGF by a novel multigenic viral expression system

    ✓ Peer-reviewed alzhub usefulness score 32 out of 100. Neurological research 2006-07-01 Biomarkers

    Features of multipotentiality of engineered NSC-derived cell lines versus naive cells (control-NSC) were assessed by immunocytochemical analysis in differentiation conditions.

  24. MAPK-activated protein kinase 2 deficiency in microglia inhibits pro-inflammatory mediator release and resultant neurotoxicity. Relevance to neuroinflammation in a transgenic mouse model of Alzheimer disease

    ✓ Peer-reviewed alzhub usefulness score 44 out of 100. The Journal of biological chemistry 2006-06-14 Risk genes beyond APOE

    MAPK-activated protein kinase 2 (MAPKAP K2 or MK2) is one of several kinases directly regulated by p38 MAPK.

  25. Tumor necrosis factor-alpha induces neurotoxicity via glutamate release from hemichannels of activated microglia in an autocrine manner

    ✓ Peer-reviewed alzhub usefulness score 49 out of 100. The Journal of biological chemistry 2006-05-23 Inflammation and the immune system

    Glutamate released by activated microglia induces excitoneurotoxicity and may contribute to neuronal damage in neurodegenerative diseases, including Alzheimer disease, Parkinson disease, amyotrophic lateral sclerosis, and multiple sclerosis.

  26. ERK1/2 activation mediates Abeta oligomer-induced neurotoxicity via caspase-3 activation and tau cleavage in rat organotypic hippocampal slice cultures

    ✓ Peer-reviewed alzhub usefulness score 36 out of 100. The Journal of biological chemistry 2006-05-18 Disease mechanisms

    In this study, we investigated the molecular basis for the altered signal transduction associated with soluble amyloid beta-protein (Abeta) oligomer-mediated neurotoxicity in the hippocampus, which is primarily linked to cognitive dysfunction in Alzheimer disease (AD).

  27. Regulation of inducible nitric oxide synthase gene in glial cells

    ✓ Peer-reviewed alzhub usefulness score 43 out of 100. Antioxidants & redox signaling 2006-05-01 Inflammation and the immune system

    Elevated levels of NO produced within the central nervous system (CNS) are associated with the pathogenesis of neuroinflammatory and neurodegenerative human diseases such as multiple sclerosis, HIV dementia, brain ischemia, trauma, Parkinson's disease, and Alzheimer's disease.

  28. The common inhalation anesthetic isoflurane induces apoptosis and increases amyloid beta protein levels

    ✓ Peer-reviewed alzhub usefulness score 48 out of 100. Anesthesiology 2006-05-01 Risk genes beyond APOE

    The common inhalation anesthetic isoflurane has previously been reported to enhance the aggregation and cytotoxicity of the Alzheimer disease-associated amyloid beta protein (Abeta), the principal peptide component of cerebral beta-amyloid deposits.

  29. Transcriptional regulation of APH-1A and increased gamma-secretase cleavage of APP and Notch by HIF-1 and hypoxia

    ✓ Peer-reviewed alzhub usefulness score 41 out of 100. FASEB journal : official publication of the Federation of American Societies for Experimental Biology 2006-04-27 Risk genes beyond APOE

    The proteolytic cleavage of Alzheimer beta-amyloid precursor protein (APP) and signaling receptor Notch is mediated by the PS/gamma-secretase complex, which consists of presenilins, nicastrin, APH-1, and PEN-2.

  30. Neprilysin-sensitive synapse-associated amyloid-beta peptide oligomers impair neuronal plasticity and cognitive function

    ✓ Peer-reviewed alzhub usefulness score 46 out of 100. The Journal of biological chemistry 2006-04-24 Risk genes beyond APOE

    A subtle but chronic alteration in metabolic balance between amyloid-beta peptide (Abeta) anabolic and catabolic activities is thought to cause Abeta accumulation, leading to a decade-long pathological cascade of Alzheimer disease.

  31. ADAM10 activation is required for green tea (-)-epigallocatechin-3-gallate-induced alpha-secretase cleavage of amyloid precursor protein

    ✓ Peer-reviewed alzhub usefulness score 47 out of 100. The Journal of biological chemistry 2006-04-19 Risk genes beyond APOE

    Recently, we have shown that green tea polyphenol (-)-epigallocatechin-3-gallate (EGCG) exerts a beneficial role on reducing brain Abeta levels, resulting in mitigation of cerebral amyloidosis in a mouse model of Alzheimer disease.

  32. Intrathecal chemokine synthesis in mild cognitive impairment and Alzheimer disease

    ✓ Peer-reviewed alzhub usefulness score 60 out of 100. Archives of neurology 2006-04-01 Mild cognitive impairment

    Interferon-gamma-inducible protein 10 is specifically increased in MCI and seems to decrease with the progression of AD, whereas MCP-1 and IL-8 are up-regulated also in late stages of the disease, suggesting a role in phases in which neurodegeneration is prevalent.

  33. Down regulation of trk but not p75NTR gene expression in single cholinergic basal forebrain neurons mark the progression of Alzheimer's disease

    ✓ Peer-reviewed alzhub usefulness score 47 out of 100. Journal of neurochemistry 2006-03-15 Mild cognitive impairment

    Dysfunction of cholinergic basal forebrain (CBF) neurons of the nucleus basalis (NB) is a cardinal feature of Alzheimer's disease (AD) and correlates with cognitive decline.

  34. 24(S)-hydroxycholesterol participates in a liver X receptor-controlled pathway in astrocytes that regulates apolipoprotein E-mediated cholesterol efflux

    ✓ Peer-reviewed alzhub usefulness score 46 out of 100. The Journal of biological chemistry 2006-03-08 Genetics

    Both apolipoprotein E (apoE) and 24(S)-hydroxycholesterol are involved in the pathogenesis of Alzheimer disease (AD).

  35. Elevated protein-bound levels of the lipid peroxidation product, 4-hydroxy-2-nonenal, in brain from persons with mild cognitive impairment

    ✓ Peer-reviewed alzhub usefulness score 45 out of 100. Neuroscience letters 2006-01-04 Mild cognitive impairment

    Oxidative damage is a feature of many age-related neurodegenerative diseases, including Alzheimer's disease (AD). 4-Hydroxy-2-nonenal (HNE) is a highly reactive product of the free radical-mediated lipid peroxidation of unsaturated lipids, particularly arachidonic acid, in cellular membranes.

  36. Aggregate-prone proteins are cleared from the cytosol by autophagy: therapeutic implications

    ✓ Peer-reviewed alzhub usefulness score 44 out of 100. Current topics in developmental biology 2006-01-01 Brain tissue and pathology

    Intracellular protein misfolding/aggregation are features of many late-onset neurodegenerative diseases, called proteinopathies.

  37. CCL2/monocyte chemoattractant protein-1 mediates enhanced transmigration of human immunodeficiency virus (HIV)-infected leukocytes across the blood-brain barrier: a potential mechanism of HIV-CNS invasion and NeuroAIDS

    ✓ Peer-reviewed alzhub usefulness score 47 out of 100. The Journal of neuroscience : the official journal of the Society for Neuroscience 2006-01-01 Inflammation and the immune system

    Encephalitis and dementia associated with acquired immunodeficiency syndrome (AIDS) are characterized by leukocyte infiltration into the CNS, microglia activation, aberrant chemokine expression, blood-brain barrier (BBB) disruption, and eventual loss of neurons.

  38. Rapamycin alleviates toxicity of different aggregate-prone proteins

    ✓ Peer-reviewed alzhub usefulness score 36 out of 100. Human molecular genetics 2005-12-20 Disease mechanisms

    Many neurodegenerative diseases are caused by intracellular, aggregate-prone proteins, including polyglutamine-expanded huntingtin in Huntington's disease (HD) and mutant tau in fronto-temporal dementia/tauopathy.

  39. Synaptic Activity Regulates Interstitial Fluid Amyloid-β Levels In Vivo

    ✓ Peer-reviewed alzhub usefulness score 56 out of 100. Neuron 2005-12-01 Disease mechanisms

  40. Selective butyrylcholinesterase inhibition elevates brain acetylcholine, augments learning and lowers Alzheimer beta-amyloid peptide in rodent

    ✓ Peer-reviewed alzhub usefulness score 51 out of 100. Proceedings of the National Academy of Sciences of the United States of America 2005-11-07 Risk genes beyond APOE

    Like acetylcholinesterase, butyrylcholinesterase (BChE) inactivates the neurotransmitter acetylcholine (ACh) and is hence a viable therapeutic target in Alzheimer's disease, which is characterized by a cholinergic deficit.

  41. Metallothionein-I-II and GFAP positivity in the brains from frontotemporal dementia patients

    ✓ Peer-reviewed alzhub usefulness score 31 out of 100. Journal of Alzheimer's disease : JAD 2005-11-01 Biomarkers

    Frontotemporal dementia regards a group of presenile progressive neurodegenerative form of dementias which includes Pick's disease, corticobasal degeneration, frontotemporal dementia with motor neuron disease, frontal lobe degeneration, dementia-parkinsonism-amyotrophy complex, familial non-specific dementia mapping to chromosome 3, non-Alzheimer degenerative dementia lacking distinctive histological features as well as a number other infrequent syndromes with dementia and focal neurological signs.

  42. Lack of LDL receptor aggravates learning deficits and amyloid deposits in Alzheimer transgenic mice

    ✓ Peer-reviewed alzhub usefulness score 43 out of 100. Neurobiology of aging 2005-10-19 Genetics

    Emerging evidence indicates that cholesterol metabolism affects the pathogenesis of Alzheimer's disease (AD).

  43. A role for docosahexaenoic acid-derived neuroprotectin D1 in neural cell survival and Alzheimer disease

    ✓ Peer-reviewed alzhub usefulness score 49 out of 100. The Journal of clinical investigation 2005-10-01 Risk genes beyond APOE

    Deficiency in docosahexaenoic acid (DHA), a brain-essential omega-3 fatty acid, is associated with cognitive decline.

  44. Enhanced neurofibrillary tangle formation, cerebral atrophy, and cognitive deficits induced by repetitive mild brain injury in a transgenic tauopathy mouse model

    ✓ Peer-reviewed alzhub usefulness score 38 out of 100. Journal of neurotrauma 2005-10-01 Disease mechanisms

    Traumatic brain injury (TBI) is a risk factors for Alzheimer's disease (AD), and repetitive TBI (rTBI) may culminate in dementia pugilistica (DP), a syndrome characterized by progressive dementia, parkinsonism, and the hallmark brain lesions of AD, including neurofibrillary tangles (NFTs), formed by abnormal tau filaments and senile plaques (SPs) composed of Abeta fibrils.

  45. Hyperinsulinemia provokes synchronous increases in central inflammation and beta-amyloid in normal adults

    ✓ Peer-reviewed alzhub usefulness score 51 out of 100. Archives of neurology 2005-10-01 Treatments

    Inflammation has been implicated as a pathogenetic factor in Alzheimer disease, possibly via effects on beta-amyloid (Abeta).

  46. SIRT1 protects against microglia-dependent amyloid-beta toxicity through inhibiting NF-kappaB signaling

    ✓ Peer-reviewed alzhub usefulness score 51 out of 100. The Journal of biological chemistry 2005-09-23 Inflammation and the immune system

    Accumulating evidence suggests that neurodegeneration induced by pathogenic proteins depends on contributions from surrounding glia.

  47. RAC1 inhibition targets amyloid precursor protein processing by gamma-secretase and decreases Abeta production in vitro and in vivo

    ✓ Peer-reviewed alzhub usefulness score 41 out of 100. The Journal of biological chemistry 2005-09-08 Risk genes beyond APOE

    beta-Amyloid peptides (Abeta) that form the senile plaques of Alzheimer disease consist mainly of 40- and 42-amino acid (Abeta 40 and Abeta 42) peptides generated from the cleavage of the amyloid precursor protein (APP).

  48. Green tea epigallocatechin-3-gallate (EGCG) modulates amyloid precursor protein cleavage and reduces cerebral amyloidosis in Alzheimer transgenic mice

    ✓ Peer-reviewed alzhub usefulness score 52 out of 100. The Journal of neuroscience : the official journal of the Society for Neuroscience 2005-09-01 Risk genes beyond APOE

    Alzheimer's disease (AD) is a progressive neurodegenerative disorder pathologically characterized by deposition of beta-amyloid (Abeta) peptides as senile plaques in the brain.

  49. Mechanisms of statin-mediated inhibition of small G-protein function

    ✓ Peer-reviewed alzhub usefulness score 45 out of 100. The Journal of biological chemistry 2005-08-04 Inflammation and the immune system

    3-Hydroxy-3-methylglutaryl coenzyme A reductase inhibitors (statins) have been reported to reduce the risk of Alzheimer disease.

  50. Amino-terminally truncated Abeta peptide species are the main component of cotton wool plaques

    ✓ Peer-reviewed alzhub usefulness score 42 out of 100. Biochemistry 2005-08-01 Risk genes beyond APOE

    Cotton wool plaques (CWPs) are round lesions that lack a central amyloid core.

From across this tag

10 of the 1,153 studies in this tag, picked by a fixed rule (a hash of each study's address), not by score or date; each page of this list shows a different 10. The same 10 appear here every time.

All topic tags with their study counts