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9 studies tagged sub, dated 2018-01-09 to 2025-02-01. Bibliography: BibTeX (.bib) · RIS (.ris)
Newest in this topic
- Influence of cognitive impairment and race on plasma p-Tau217 in two diverse cohorts 2025-02-01
- Plasma p-tau217 and neurofilament/p-tau217 ratio in differentiating Alzheimer's disease from syndromes associated with frontotemporal lobar degeneration 2025-01-08
- Plasma p-tau181 shows stronger network association to Alzheimer's disease dementia than neurofilament light and total tau 2021-12-02
Studies
9 studies carry this tag.
Date range
2018 to 2025
Top journals
- Alzheimer's & dementia : the journal of the Alzheimer's Association 4
- ACS chemical neuroscience 1
- Brain and behavior 1
- CNS neuroscience & therapeutics 1
- International journal of molecular sciences 1
6 journals in all.
Newest first, then by address. Each study has a usefulness score out of 100 (rubric version 4.0); tap a score to see what makes it up. Tags were assigned automatically from each study's title and summary. The same studies as a filter in the library.
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Severity of cognitive impairment and race both influence plasma p-Tau 217 levels beyond race-associated differences in CSF p-Tau 181 .
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Plasma p-tau 217 and the NfL/p-tau 217 ratio are promising non-invasive biomarkers for differentiating AD from FTLD, suggesting their use as a potential alternative to traditional diagnostic methods.
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These results support plasma p-tau 181 for the detection of AD dementia and the use of blood-based biomarkers for optimal disease detection.
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The current study was undertaken to unveil the protective effects of Luteolin, a natural flavonoid, against amyloid-beta (Aβ 1 - 42 )-induced neuroinflammation, amyloidogenesis, and synaptic dysfunction in mice.
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Beta-amyloid (Aβ 1-42 ) triggers the phosphorylation of tau protein in Alzheimer's disease (AD), but the relationship between phosphorylated tau (p-tau) and Aβ 1-42 in the blood is not elucidated.
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62/100
The assessment of serum NfL and p-Tau 181 may provide a comprehensive view of FTLD, aiding in the differential diagnosis, in staging disease severity and in defining survival probability.
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These results indicate that FS (p-tau231 and MMSE) has a strong potential to provide an early distinction between AD and VaD.
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Overall, these results support the vervet as a non-human primate model of amyloid-related neurodegeneration, such as Alzheimer's disease and cerebral amyloid angiopathy, and highlight Aβ 1-40 and p-tau 181 as potentially valuable biomarkers of these processes.
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Novel and established cerebrospinal fluid markers perform with at least fair accuracy in the discrimination between ADD and FTD.