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glucose-metabolism
13 studies tagged glucose-metabolism, dated 2007-07-13 to 2024-09-11. Bibliography: BibTeX (.bib) · RIS (.ris)
Newest in this topic
- Acute exercise alters brain glucose metabolism in aging and Alzheimer's disease 2024-09-11
- Rasagiline effects on glucose metabolism, cognition, and tau in Alzheimer's dementia 2021-02-14
- Regional Disconnection in Alzheimer Dementia and Amyloid-Positive Mild Cognitive Impairment: Association Between EEG Functional Connectivity and Brain Glucose Metabolism 2020-11-23
Studies
13 studies carry this tag.
Date range
2007 to 2024
Top journals
- Neurobiology of aging 3
- Alzheimer's & dementia (New York, N. Y.) 1
- Brain connectivity 1
- Cerebral cortex (New York, N.Y. : 1991) 1
- Clinical and translational imaging 1
11 journals in all.
Newest first, then by address. Each study has a usefulness score out of 100 (rubric version 4.0); tap a score to see what makes it up. Tags were assigned automatically from each study's title and summary. The same studies as a filter in the library.
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There is evidence that aerobic exercise improves brain health.
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These outcomes illustrate the potential benefits of rasagiline on clinical and neuroimaging measures in patients with mild to moderate AD.
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Glucose hypometabolism in temporoparietal lobes was significantly associated with altered EEG functional connectivity of the same regions of interest in clinically diagnosed MCI and AD patients and in patients with biomarker-verified AD pathology.
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Molecular imaging of tauopathies is complicated by the differing specificities and off-target binding properties of available radioligands for positron emission tomography (PET). [ 18 F]-APN-1607 ([ 18 F]-PM-PBB3) is a newly developed PET tracer with promising properties for tau imaging.
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Tau is associated with hypometabolism in patients with Alzheimer's disease.
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An essential feature of Alzheimer's disease (AD) is implicated in brain energy metabolic impairment that is considered underlying pathogenesis of cognitive impairment.
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Alzheimer's disease (AD) is an age-dependent neurodegenerative disorder associated with progressive loss of cognitive function. 2-[ 18 F]fluoro-2-deoxy-D-glucose (FDG) positron emission tomography (PET) has long been used to measure resting-state cerebral metabolic rates of glucose, a proxy for neuronal activity.
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36/100
The ε4 allele of the polymorphic apolipoprotein E gene is associated with increased risk of Alzheimer's disease (AD), deposition of β-amyloid (Aβ), and reduction in cerebral glucose metabolism in asymptomatic people.
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β-Amyloid (Aβ), a feature of Alzheimer's disease (AD) pathology, may precede reduced glucose metabolism and gray matter (GM) volume and cognitive decline in patients with AD.
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Epidemiological studies suggest that elevated blood pressure (BP) in midlife is associated with increased risk of Alzheimer's disease (AD) in late life.
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Glucose dysmetabolism has been consistently associated with an increased risk of cognitive disorders, and brain insulin resistance could play a role in Alzheimer's disease (AD) pathogenesis.
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The authors investigated relationships between glucose metabolism, amyloid load, and measures of cognitive and functional impairment in Alzheimer's disease (AD).
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36/100
With age, the brain undergoes both structural and functional alterations, probably resulting in reported cognitive declines.