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amyloid-burden
8 studies tagged amyloid-burden, dated 2016-01-26 to 2025-01-14. Bibliography: BibTeX (.bib) · RIS (.ris)
Newest in this topic
- Clinical utility of plasma p-tau217 in identifying abnormal brain amyloid burden in an Asian cohort with high prevalence of concomitant cerebrovascular disease 2025-01-14
- The association of subjective sleep characteristics and plasma biomarkers of Alzheimer's disease pathology in older cognitively unimpaired adults with higher amyloid-β burden 2023-02-20
- Associations of plasma phosphorylated tau181 and neurofilament light chain with brain amyloid burden and cognition in objectively defined subtle cognitive decline patients 2022-09-08
Studies
8 studies carry this tag.
Date range
2016 to 2025
Top journals
- Journal of Alzheimer's disease : JAD 2
- Alzheimer's & dementia : the journal of the Alzheimer's Association 1
- American journal of respiratory and critical care medicine 1
- CNS neuroscience & therapeutics 1
- Journal of neurology 1
7 journals in all.
Newest first, then by address. Each study has a usefulness score out of 100 (rubric version 4.0); tap a score to see what makes it up. Tags were assigned automatically from each study's title and summary. The same studies as a filter in the library.
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Risk stratification for PET Aβ+ based on a plasma p-tau217 assay demonstrated potential diagnostic and prognostic utility in an Asian cohort with concomitant CeVD.
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We aimed to investigate the association of subjective sleep characteristics and plasma Alzheimer's disease (AD) biomarkers in older cognitively unimpaired adults with higher amyloid-β (Aβ) burden.
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Our findings highlight that plasma p-tau181, alone or combined with NfL, contributes to identifying high-risk AD populations.
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CD22 has been suggested to contribute to Alzheimer's disease (AD) pathogenesis by inhibiting microglial amyloid β (Aβ) phagocytosis.
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In cognitively unimpaired adults, elevated plasma p-tau181 is associated with future increased Aβ deposition across multiple brain regions.
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47/100
The developed model based on serum biomarkers and other co-variates could reflect the AD core pathology.
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In a sample of cognitively normal elderly, OSA was associated with markers of increased amyloid burden over the 2-year follow-up.
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We believe this is the first study to investigate associations between blood metabolites and neocortical amyloid burden (NAB) in the search for a blood-based biomarker for Alzheimer's disease (AD).