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Home › Research › Topics › amyloid-deposition

amyloid-deposition

8 studies tagged amyloid-deposition, dated 2001-08-01 to 2025-09-05. Bibliography: BibTeX (.bib) · RIS (.ris)

Newest in this topic

  1. Hemispheric asymmetry of tau pathology is related to asymmetric amyloid deposition in Alzheimer's Disease 2025-09-05
  2. Metabotropic glutamate receptor 5 (mGluR5) is associated with neurodegeneration and amyloid deposition in Alzheimer's disease: A [18F]PSS232 PET/MRI study 2024-01-12
  3. Blood-brain barrier breakdown is linked to tau pathology and neuronal injury in a differential manner according to amyloid deposition 2023-06-07

Studies

8 studies carry this tag.

Date range

2001 to 2025

Top journals

  • ACS chemical neuroscience 1
  • Alzheimer's research & therapy 1
  • FASEB journal : official publication of the Federation of American Societies for Experimental Biology 1
  • Hippocampus 1
  • Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism 1

8 journals in all.

Newest first, then by address. Each study has a usefulness score out of 100 (rubric version 4.0); tap a score to see what makes it up. Tags were assigned automatically from each study's title and summary. The same studies as a filter in the library.

  1. Hemispheric asymmetry of tau pathology is related to asymmetric amyloid deposition in Alzheimer's Disease

    ✓ Peer-reviewed alzhub usefulness score 72 out of 100. Nature communications 2025-09-05 Biomarkers

    The distribution of tau pathology in Alzheimer's disease (AD) shows remarkable inter-individual heterogeneity, including hemispheric asymmetry.

  2. Metabotropic glutamate receptor 5 (mGluR5) is associated with neurodegeneration and amyloid deposition in Alzheimer's disease: A [18F]PSS232 PET/MRI study

    ✓ Peer-reviewed alzhub usefulness score 52 out of 100. Alzheimer's research & therapy 2024-01-12 Biomarkers

    [ 18 F]PSS232 PET can quantify the changes of mGluR5 availability in the progression of AD. mGluR5 availability correlated not only with neuropathological biomarkers of AD but also with neurodegenerative biomarkers and cognitive performance. mGluR5 may be a novel neurodegenerative biomarker, and whether mGluR5 could be a potential therapeutic target for AD needs to be further studied.

  3. Blood-brain barrier breakdown is linked to tau pathology and neuronal injury in a differential manner according to amyloid deposition

    ✓ Peer-reviewed alzhub usefulness score 47 out of 100. Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism 2023-06-07 Biomarkers

    The blood-brain barrier (BBB) breakdown has been suggested as an early marker for Alzheimer's disease (AD); yet the relationship between BBB breakdown and AD-specific biomarkers based on the amyloid/tau/neurodegeneration framework is not clear.

  4. Plasma Core Alzheimer's Disease Biomarkers Predict Amyloid Deposition Burden by Positron Emission Tomography in Chinese Individuals with Cognitive Decline

    ✓ Peer-reviewed alzhub usefulness score 48 out of 100. ACS chemical neuroscience 2022-12-22 Biomarkers

    Blood-based biomarkers have been considered as a promising method for the diagnosis of Alzheimer's disease (AD).

  5. Blood acetylcholinesterase level is a potential biomarker for the early detection of cerebral amyloid deposition in cognitively normal individuals

    ✓ Peer-reviewed alzhub usefulness score 47 out of 100. Neurobiology of aging 2018-09-08 Biomarkers

    Cerebral β-amyloid (cAβ) deposition and cholinergic dysfunction have been considered as major pathological and functional hallmarks of Alzheimer's disease (AD).

  6. Neuronal and glial alterations, increased anxiety, and cognitive impairment before hippocampal amyloid deposition in PDAPP mice, model of Alzheimer's disease

    ✓ Peer-reviewed alzhub usefulness score 36 out of 100. Hippocampus 2013-11-12 Biomarkers

    In the context of Alzheimer's disease (AD), hippocampal alterations have been well described in advanced stages of the pathology, when amyloid deposition, inflammation and glial activation occur, but less attention has been directed to studying early brain and behavioral changes.

  7. The Psen1-L166P-knock-in mutation leads to amyloid deposition in human wild-type amyloid precursor protein YAC transgenic mice

    ✓ Peer-reviewed alzhub usefulness score 36 out of 100. FASEB journal : official publication of the Federation of American Societies for Experimental Biology 2012-03-29 Biomarkers

    Genetically engineered mice have been generated to model cerebral β-amyloidosis, one of the hallmarks of Alzheimer disease (AD) pathology, based on the overexpression of a mutated cDNA of the amyloid-β precursor protein (AβPP) or by knock-in of the murine Aβpp gene alone or with presenilin1 mutations.

  8. Neuroinflammation-induced acceleration of amyloid deposition in the APPV717F transgenic mouse

    ✓ Peer-reviewed alzhub usefulness score 36 out of 100. The European journal of neuroscience 2001-08-01 Biomarkers

    It has been postulated that neuroinflammation plays a critical role in the pathogenesis of Alzheimer's disease (AD).

All topic tags with their study counts