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pet-imaging
4 studies tagged pet-imaging, dated 2010-12-13 to 2024-01-01. Bibliography: BibTeX (.bib) · RIS (.ris)
Newest in this topic
- Longitudinal Phospho-tau217 Predicts Amyloid Positron Emission Tomography in Asymptomatic Alzheimer's Disease 2024-01-01
- Head-to-head comparison of plasma and PET imaging ATN markers in subjects with cognitive complaints 2023-06-29
- Population pharmacokinetic-pharmacodynamic analyses of amyloid positron emission tomography and plasma biomarkers for lecanemab in subjects with early Alzheimer's disease 2022-09-27
Studies
4 studies carry this tag.
Date range
2010 to 2024
Top journals
- Brain : a journal of neurology 1
- CPT: pharmacometrics & systems pharmacology 1
- The journal of prevention of Alzheimer's disease 1
- Translational neurodegeneration 1
Newest first, then by address. Each study has a usefulness score out of 100 (rubric version 4.0); tap a score to see what makes it up. Tags were assigned automatically from each study's title and summary. The same studies as a filter in the library.
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An ECL immunoassay testing plasma P-tau217 accurately predicts amyloid PET positivity in cognitively unimpaired individuals.
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55/100
Plasma p-tau181, as well as 18 F-florbetapir and 18 F-Florzolotau PET imaging can be considered as interchangeable biomarkers in the assessment of Aβ status in symptomatic stages of AD. 18 F-Florzolotau and 18 F-FDG PET imaging could serve as biomarkers for the severity of cognitive impairment.
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Lecanemab is a humanized immunoglobulin G1 monoclonal antibody that selectively binds to soluble Aβ aggregate species, while demonstrating low affinity for Aβ monomer.
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In summary, high (11)C-Pittsburgh Compound B positron emission tomography retention significantly correlates with both fibrillar β-amyloid and losses of neuronal nicotinic acetylcholine receptor subtypes at autopsy, suggesting a closer involvement of β-amyloid pathology with neuronal nicotinic acetylcholine receptor subtypes than with inflammatory processes.