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Newest in this topic

  1. Ageing and amyloidosis underlie the molecular and pathological alterations of tau in a mouse model of familial Alzheimer's disease 2019-10-31
  2. Heterochromatin anomalies and double-stranded RNA accumulation underlie C9orf72 poly(PR) toxicity 2019-02-01

Studies

2 studies carry this tag.

Date range

2019

Top journals

  • Science (New York, N.Y.) 1
  • Scientific reports 1

Newest first, then by address. Each study has a usefulness score out of 100 (rubric version 4.0); tap a score to see what makes it up. Tags were assigned automatically from each study's title and summary. The same studies as a filter in the library.

  1. Ageing and amyloidosis underlie the molecular and pathological alterations of tau in a mouse model of familial Alzheimer's disease

    ✓ Peer-reviewed alzhub usefulness score 41 out of 100. Scientific reports 2019-10-31 Brain scans and imaging

    Despite compelling evidence that the accumulation of amyloid-beta (Aβ) promotes neocortical MAPT (tau) aggregation in familial and idiopathic Alzheimer's disease (AD), murine models of cerebral amyloidosis are not considered to develop tau-associated pathology.

  2. Heterochromatin anomalies and double-stranded RNA accumulation underlie C9orf72 poly(PR) toxicity

    ✓ Peer-reviewed alzhub usefulness score 43 out of 100. Science (New York, N.Y.) 2019-02-01 Brain scans and imaging

    How hexanucleotide GGGGCC (G 4 C 2 ) repeat expansions in C9orf72 cause frontotemporal dementia (FTD) and amyotrophic lateral sclerosis (ALS) is not understood.

All topic tags with their study counts