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protein-transgenic
3 studies tagged protein-transgenic, dated 2005-05-01 to 2015-07-20. Bibliography: BibTeX (.bib) · RIS (.ris)
Newest in this topic
- Tau post-translational modifications in wild-type and human amyloid precursor protein transgenic mice 2015-07-20
- Complement C3 deficiency leads to accelerated amyloid beta plaque deposition and neurodegeneration and modulation of the microglia/macrophage phenotype in amyloid precursor protein transgenic mice 2008-06-01
- Overexpression of monocyte chemotactic protein-1/CCL2 in beta-amyloid precursor protein transgenic mice show accelerated diffuse beta-amyloid deposition 2005-05-01
Studies
3 studies carry this tag.
Date range
2005 to 2015
Top journals
- Nature neuroscience 1
- The American journal of pathology 1
- The Journal of neuroscience : the official journal of the Society for Neuroscience 1
Newest first, then by address. Each study has a usefulness score out of 100 (rubric version 4.0); tap a score to see what makes it up. Tags were assigned automatically from each study's title and summary. The same studies as a filter in the library.
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The microtubule-associated protein tau has been implicated in the pathogenesis of Alzheimer's disease (AD) and other neurodegenerative disorders.
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Complement factor C3 is the central component of the complement system and a key inflammatory protein activated in Alzheimer's disease (AD).
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Microglia accumulation at the site of amyloid plaques is a strong indication that microglia play a major role in Alzheimer's disease pathogenesis.