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Newest in this topic

  1. Web-Based Assessment of Cognition and Dementia Risk Factors in Over 3000 Norwegian Adults Aged 50 Years and Older: Cross-Sectional PROTECT Norge Study 2025-08-25
  2. Does Soluble TREM2 Protect Against Alzheimer's Disease? 2022-01-28
  3. Anti-inflammatory treatment in AD mice protects against neuronal pathology 2009-08-10

Studies

4 studies carry this tag.

Date range

2003 to 2025

Top journals

  • Experimental neurology 1
  • Frontiers in aging neuroscience 1
  • JMIR aging 1
  • Journal of neurochemistry 1

Newest first, then by address. Each study has a usefulness score out of 100 (rubric version 4.0); tap a score to see what makes it up. Tags were assigned automatically from each study's title and summary. The same studies as a filter in the library.

  1. Web-Based Assessment of Cognition and Dementia Risk Factors in Over 3000 Norwegian Adults Aged 50 Years and Older: Cross-Sectional PROTECT Norge Study

    ✓ Peer-reviewed alzhub usefulness score 62 out of 100. JMIR aging 2025-08-25 Cognitive tests and assessment

    These data show the associations between dementia risk factors and cognition and attest to the excellent feasibility of the PROTECT Norge cohort, with over 3000 participants included over a period of 2 years and accessibility for people with early cognitive impairment.

  2. Does Soluble TREM2 Protect Against Alzheimer's Disease?

    ✓ Peer-reviewed alzhub usefulness score 43 out of 100. Frontiers in aging neuroscience 2022-01-28 TREM2 and immune genetics

    Triggering Receptor Expressed in Myeloid Cells 2 (TREM2) is a pattern recognition receptor on myeloid cells, and is upregulated on microglia surrounding amyloid plaques in Alzheimer's disease (AD).

  3. Anti-inflammatory treatment in AD mice protects against neuronal pathology

    ✓ Peer-reviewed alzhub usefulness score 36 out of 100. Experimental neurology 2009-08-10 Biomarkers

    Prior studies suggest that non-steroidal anti-inflammatory drugs (NSAIDs) may lower the incidence of Alzheimer's disease (AD) and delay onset or slow progression of symptoms in mouse models of AD.

  4. Amyloid beta-protein (Abeta)1-40 protects neurons from damage induced by Abeta1-42 in culture and in rat brain

    ✓ Peer-reviewed alzhub usefulness score 36 out of 100. Journal of neurochemistry 2003-11-01 Biomarkers

    Previously, we found that amyloid beta-protein (Abeta)1-42 exhibits neurotoxicity, while Abeta1-40 serves as an antioxidant molecule by quenching metal ions and inhibiting metal-mediated oxygen radical generation.

All topic tags with their study counts