Home › Research › Topics › protection
protection
6 studies tagged protection, dated 2003-02-03 to 2018-10-09. Bibliography: BibTeX (.bib) · RIS (.ris)
Newest in this topic
- The antimicrobial protection hypothesis of Alzheimer's disease 2018-10-09
- Cellular and molecular events mediated by docosahexaenoic acid-derived neuroprotectin D1 signaling in photoreceptor cell survival and brain protection 2009-06-11
- Role of human aldo-keto-reductase AKR1B10 in the protection against toxic aldehydes 2008-11-01
Studies
6 studies carry this tag.
Date range
2003 to 2018
Top journals
- Alzheimer's & dementia : the journal of the Alzheimer's Association 1
- Chemico-biological interactions 1
- International journal of hyperthermia : the official journal of European Society for Hyperthermic Oncology, North American Hyperthermia Group 1
- Proceedings of the National Academy of Sciences of the United States of America 1
- Prostaglandins, leukotrienes, and essential fatty acids 1
6 journals in all.
Newest first, then by address. Each study has a usefulness score out of 100 (rubric version 4.0); tap a score to see what makes it up. Tags were assigned automatically from each study's title and summary. The same studies as a filter in the library.
-
Mounting data link elevated brain microbe levels with AD.
-
Deficiency in docosahexaenoic acid (DHA) is associated with impaired visual and neurological postnatal development, cognitive decline, macular degeneration, and other neurodegenerative diseases.
-
Damage of cell membranes by reactive oxygen species results in the formation of toxic lipid peroxides which may ultimately lead to cell death.
-
36/100
Heat shock proteins (Hsps) are highly conserved and under physiological conditions act as molecular chaperones and/or have anti-apoptotic activities.
-
Intracellular amyloidbeta peptide (iAbeta1-42) accumulates in the Alzheimer's disease brain before plaque and tangle formation (Gouras et al., 2000) and is extremely toxic to human neurons (Zhang et al., 2002).
-
Transgenic PDAPP mice, which express a disease-linked isoform of the human amyloid precursor protein, exhibit CNS pathology that is similar to Alzheimer's disease.