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Home › Research › Topics › normal

normal

6 studies tagged normal, dated 1993-11-01 to 2023-01-01. Bibliography: BibTeX (.bib) · RIS (.ris)

Newest in this topic

  1. Intracellular Amyloid-β in the Normal Rat Brain and Human Subjects and Its relevance for Alzheimer's Disease 2023-01-01
  2. Characterizing Amyloid-Positive Individuals With Normal Tau PET Levels After 5 Years: An ADNI Study 2022-03-21
  3. The levels of soluble versus insoluble brain Abeta distinguish Alzheimer's disease from normal and pathologic aging 1999-08-01

Studies

6 studies carry this tag.

Date range

1993 to 2023

Top journals

  • Brain research 1
  • Cerebral cortex (New York, N.Y. : 1991) 1
  • Experimental neurology 1
  • Journal of Alzheimer's disease : JAD 1
  • Journal of neurochemistry 1

6 journals in all.

Newest first, then by address. Each study has a usefulness score out of 100 (rubric version 4.0); tap a score to see what makes it up. Tags were assigned automatically from each study's title and summary. The same studies as a filter in the library.

  1. Intracellular Amyloid-β in the Normal Rat Brain and Human Subjects and Its relevance for Alzheimer's Disease

    ✓ Peer-reviewed alzhub usefulness score 43 out of 100. Journal of Alzheimer's disease : JAD 2023-01-01 Biomarkers

    Expression of iAβ42 in healthy Wistar rats predominates in the same structures where iAβ accumulates and Aβ plaques initially form in the much used, Wistar based McGill-R-Thy1-APP rat model for AD.

  2. Characterizing Amyloid-Positive Individuals With Normal Tau PET Levels After 5 Years: An ADNI Study

    ✓ Peer-reviewed alzhub usefulness score 64 out of 100. Neurology 2022-03-21 Brain scans and imaging

    Aβ-positive individuals can remain tau-negative for at least 5 years.

  3. The levels of soluble versus insoluble brain Abeta distinguish Alzheimer's disease from normal and pathologic aging

    ✓ Peer-reviewed alzhub usefulness score 40 out of 100. Experimental neurology 1999-08-01 Risk genes beyond APOE

    The abundance and solubility of Abeta peptides are critical determinants of amyloidosis in Alzheimer's disease (AD).

  4. Cytoarchitectonic definition of prefrontal areas in the normal human cortex: I. Remapping of areas 9 and 46 using quantitative criteria

    ✓ Peer-reviewed alzhub usefulness score 40 out of 100. Cerebral cortex (New York, N.Y. : 1991) 1995-07-01 Brain scans and imaging

    Layers III and V exhibit clearer sublamination in area 9, while layer IV is also somewhat wider in area 46 than in area 9 (9.3% vs 6.4% of cortical thickness); the overall thickness of the cortex is the same in both areas.

  5. Morphological diversities of CD44 positive astrocytes in the cerebral cortex of normal subjects and patients with Alzheimer's disease

    ✓ Peer-reviewed alzhub usefulness score 36 out of 100. Brain research 1993-12-01 Biomarkers

    The localization of CD44 was investigated immunohistochemically in postmortem human brain tissue of control subjects and patients with Alzheimer's disease.

  6. Detection of tau proteins in normal and Alzheimer's disease cerebrospinal fluid with a sensitive sandwich enzyme-linked immunosorbent assay

    ✓ Peer-reviewed alzhub usefulness score 36 out of 100. Journal of neurochemistry 1993-11-01 Tau biomarkers

    Alzheimer's disease is a progressive degenerative dementia characterized by the abundant presence of neurofibrillary tangles in neurons.

All topic tags with their study counts