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Home › Research › Topics › neuronal-cells

neuronal-cells

3 studies tagged neuronal-cells, dated 2004-06-01 to 2024-08-05. Bibliography: BibTeX (.bib) · RIS (.ris)

Newest in this topic

  1. Single-cell spatial transcriptomics reveals distinct patterns of dysregulation in non-neuronal and neuronal cells induced by the Trem2R47H Alzheimer's risk gene mutation 2024-08-05
  2. Isoform-Specific Effects of Apolipoprotein E on Markers of Inflammation and Toxicity in Brain Glia and Neuronal Cells In Vitro 2021-05-27
  3. Acetylcholinesterase-Abeta complexes are more toxic than Abeta fibrils in rat hippocampus: effect on rat beta-amyloid aggregation, laminin expression, reactive astrocytosis, and neuronal cell loss 2004-06-01

Studies

3 studies carry this tag.

Date range

2004 to 2024

Top journals

  • Current issues in molecular biology 1
  • Molecular psychiatry 1
  • The American journal of pathology 1

Newest first, then by address. Each study has a usefulness score out of 100 (rubric version 4.0); tap a score to see what makes it up. Tags were assigned automatically from each study's title and summary. The same studies as a filter in the library.

  1. Single-cell spatial transcriptomics reveals distinct patterns of dysregulation in non-neuronal and neuronal cells induced by the Trem2R47H Alzheimer's risk gene mutation

    ✓ Peer-reviewed alzhub usefulness score 48 out of 100. Molecular psychiatry 2024-08-05 TREM2 and immune genetics

    The R47H missense mutation of the TREM2 gene is a known risk factor for development of Alzheimer's Disease.

  2. Isoform-Specific Effects of Apolipoprotein E on Markers of Inflammation and Toxicity in Brain Glia and Neuronal Cells In Vitro

    ✓ Peer-reviewed alzhub usefulness score 43 out of 100. Current issues in molecular biology 2021-05-27 TREM2 and immune genetics

    Mutations to the cholesterol transport protein apolipoprotein E (ApoE) have been identified as a major risk factor for the development of sporadic or late-onset Alzheimer's disease (AD), with the e4 allele representing an increased risk and the rare e2 allele having a reduced risk compared to the primary e3 form.

  3. Acetylcholinesterase-Abeta complexes are more toxic than Abeta fibrils in rat hippocampus: effect on rat beta-amyloid aggregation, laminin expression, reactive astrocytosis, and neuronal cell loss

    ✓ Peer-reviewed alzhub usefulness score 36 out of 100. The American journal of pathology 2004-06-01 Biomarkers

    Neuropathological changes generated by human amyloid-beta peptide (Abeta) fibrils and Abeta-acetylcholinesterase (Abeta-AChE) complexes were compared in rat hippocampus in vivo.

All topic tags with their study counts