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Newest in this topic

  1. Intracranial metastasis as a mimic of amyloid-related imaging abnormalities 2026-10-15
  2. L-685,458, an aspartyl protease transition state mimic, is a potent inhibitor of amyloid beta-protein precursor gamma-secretase activity 2000-08-01

Studies

2 studies carry this tag.

Date range

2000 to 2026

Top journals

  • Biochemistry 1
  • Neurological sciences : official journal of the Italian Neurological Society and of the Italian Society of Clinical Neurophysiology 1

Newest first, then by address. Each study has a usefulness score out of 100 (rubric version 4.0); tap a score to see what makes it up. Tags were assigned automatically from each study's title and summary. The same studies as a filter in the library.

  1. Intracranial metastasis as a mimic of amyloid-related imaging abnormalities

    ✓ Peer-reviewed alzhub usefulness score 48 out of 100. Neurological sciences : official journal of the Italian Neurological Society and of the Italian Society of Clinical Neurophysiology 2026-10-15 Treatments

    This report describes a 79-year-old woman with biomarker-confirmed Alzheimer's disease who developed progressive vasogenic edema initially interpreted as amyloid-related imaging abnormalities (ARIA) with edema during lecanemab therapy, but was ultimately found to have multifocal intracranial metastases from recurrent papillary thyroid carcinoma.

  2. L-685,458, an aspartyl protease transition state mimic, is a potent inhibitor of amyloid beta-protein precursor gamma-secretase activity

    ✓ Peer-reviewed alzhub usefulness score 36 out of 100. Biochemistry 2000-08-01 Risk genes beyond APOE

    Progressive cerebral amyloid beta-protein (A beta) deposition is believed to play a central role in the pathogenesis of Alzheimer's disease (AD).

All topic tags with their study counts