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Home › Research › Topics › mice-overexpressing

mice-overexpressing

2 studies tagged mice-overexpressing, dated 1999-02-01 to 2008-01-17. Bibliography: BibTeX (.bib) · RIS (.ris)

Newest in this topic

  1. Nox2-derived radicals contribute to neurovascular and behavioral dysfunction in mice overexpressing the amyloid precursor protein 2008-01-17
  2. SOD1 rescues cerebral endothelial dysfunction in mice overexpressing amyloid precursor protein 1999-02-01

Studies

2 studies carry this tag.

Date range

1999 to 2008

Top journals

  • Nature neuroscience 1
  • Proceedings of the National Academy of Sciences of the United States of America 1

Newest first, then by address. Each study has a usefulness score out of 100 (rubric version 4.0); tap a score to see what makes it up. Tags were assigned automatically from each study's title and summary. The same studies as a filter in the library.

  1. Nox2-derived radicals contribute to neurovascular and behavioral dysfunction in mice overexpressing the amyloid precursor protein

    ✓ Peer-reviewed alzhub usefulness score 36 out of 100. Proceedings of the National Academy of Sciences of the United States of America 2008-01-17 Risk genes beyond APOE

    Alterations in cerebrovascular regulation related to vascular oxidative stress have been implicated in the mechanisms of Alzheimer's disease (AD), but their role in the amyloid deposition and cognitive impairment associated with AD remains unclear.

  2. SOD1 rescues cerebral endothelial dysfunction in mice overexpressing amyloid precursor protein

    ✓ Peer-reviewed alzhub usefulness score 40 out of 100. Nature neuroscience 1999-02-01 Risk genes beyond APOE

    Peptides derived from proteolytic processing of the beta-amyloid precursor protein (APP), including the amyloid-beta peptide, are important for the pathogenesis of Alzheimer's dementia.

All topic tags with their study counts