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Home › Research › Topics › hippocampal-neurons

hippocampal-neurons

3 studies tagged hippocampal-neurons, dated 1994-07-01 to 2002-03-01. Bibliography: BibTeX (.bib) · RIS (.ris)

Newest in this topic

  1. Folic acid deficiency and homocysteine impair DNA repair in hippocampal neurons and sensitize them to amyloid toxicity in experimental models of Alzheimer's disease 2002-03-01
  2. Increased vulnerability of hippocampal neurons to excitotoxic necrosis in presenilin-1 mutant knock-in mice 1999-01-01
  3. Secreted forms of beta-amyloid precursor protein protect hippocampal neurons against amyloid beta-peptide-induced oxidative injury 1994-07-01

Studies

3 studies carry this tag.

Date range

1994 to 2002

Top journals

  • Experimental neurology 1
  • Nature medicine 1
  • The Journal of neuroscience : the official journal of the Society for Neuroscience 1

Newest first, then by address. Each study has a usefulness score out of 100 (rubric version 4.0); tap a score to see what makes it up. Tags were assigned automatically from each study's title and summary. The same studies as a filter in the library.

  1. Folic acid deficiency and homocysteine impair DNA repair in hippocampal neurons and sensitize them to amyloid toxicity in experimental models of Alzheimer's disease

    ✓ Peer-reviewed alzhub usefulness score 36 out of 100. The Journal of neuroscience : the official journal of the Society for Neuroscience 2002-03-01 Risk genes beyond APOE

    Recent epidemiological and clinical data suggest that persons with low folic acid levels and elevated homocysteine levels are at increased risk of Alzheimer's disease (AD), but the underlying mechanism is unknown.

  2. Increased vulnerability of hippocampal neurons to excitotoxic necrosis in presenilin-1 mutant knock-in mice

    ✓ Peer-reviewed alzhub usefulness score 41 out of 100. Nature medicine 1999-01-01 Risk genes beyond APOE

    Excitotoxicity, a form of neuronal injury in which excessive activation of glutamate receptors results in cellular calcium overload, has been implicated in the pathogenesis of Alzheimer disease (AD), although direct evidence is lacking.

  3. Secreted forms of beta-amyloid precursor protein protect hippocampal neurons against amyloid beta-peptide-induced oxidative injury

    ✓ Peer-reviewed alzhub usefulness score 36 out of 100. Experimental neurology 1994-07-01 Risk genes beyond APOE

    Alternative processing of the beta-amyloid precursor protein (beta APP) can result in liberation of either secreted forms of beta APP (APPSs), which may play roles in neuronal plasticity and survival, or amyloid beta-peptide (A beta), which can be neurotoxic.

All topic tags with their study counts