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Home › Research › Topics › generation

generation

4 studies tagged generation, dated 1998-04-01 to 2022-01-20. Bibliography: BibTeX (.bib) · RIS (.ris)

Newest in this topic

  1. Expression of the transcription factor PU.1 induces the generation of microglia-like cells in human cortical organoids 2022-01-20
  2. Obesity in aging exacerbates blood-brain barrier disruption, neuroinflammation, and oxidative stress in the mouse hippocampus: effects on expression of genes involved in beta-amyloid generation and Alzheimer's disease 2013-11-22
  3. Deletion of tumor necrosis factor death receptor inhibits amyloid beta generation and prevents learning and memory deficits in Alzheimer's mice 2007-08-01

Studies

4 studies carry this tag.

Date range

1998 to 2022

Top journals

  • Nature communications 1
  • Nature medicine 1
  • The Journal of cell biology 1
  • The journals of gerontology. Series A, Biological sciences and medical sciences 1

Newest first, then by address. Each study has a usefulness score out of 100 (rubric version 4.0); tap a score to see what makes it up. Tags were assigned automatically from each study's title and summary. The same studies as a filter in the library.

  1. Expression of the transcription factor PU.1 induces the generation of microglia-like cells in human cortical organoids

    ✓ Peer-reviewed alzhub usefulness score 47 out of 100. Nature communications 2022-01-20 Genetics

    Microglia play a role in the emergence and preservation of a healthy brain microenvironment.

  2. Obesity in aging exacerbates blood-brain barrier disruption, neuroinflammation, and oxidative stress in the mouse hippocampus: effects on expression of genes involved in beta-amyloid generation and Alzheimer's disease

    ✓ Peer-reviewed alzhub usefulness score 36 out of 100. The journals of gerontology. Series A, Biological sciences and medical sciences 2013-11-22 Risk genes beyond APOE

    There is growing evidence that obesity has deleterious effects on the brain and cognitive function in the elderly population.

  3. Deletion of tumor necrosis factor death receptor inhibits amyloid beta generation and prevents learning and memory deficits in Alzheimer's mice

    ✓ Peer-reviewed alzhub usefulness score 40 out of 100. The Journal of cell biology 2007-08-01 Risk genes beyond APOE

    The tumor necrosis factor type 1 death receptor (TNFR1) contributes to apoptosis.

  4. Estrogen reduces neuronal generation of Alzheimer beta-amyloid peptides

    ✓ Peer-reviewed alzhub usefulness score 41 out of 100. Nature medicine 1998-04-01 Risk genes beyond APOE

    Alzheimer's disease (AD) is characterized by the accumulation of cerebral plaques composed of 40- and 42-amino acid beta-amyloid (Abeta) peptides, and autosomal dominant forms of AD appear to cause disease by promoting brain Abeta accumulation.

All topic tags with their study counts