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Newest in this topic

  1. Objective subtle cognitive decline and plasma phosphorylated tau181: Early markers of Alzheimer's disease-related declines 2021-10-14
  2. Apathy and anxiety are early markers of Alzheimer's disease 2019-10-19
  3. Apolipoprotein C-III is an amyloid-β-binding protein and an early marker for Alzheimer's disease 2014-01-01

Studies

3 studies carry this tag.

Date range

2014 to 2021

Top journals

  • Alzheimer's & dementia (Amsterdam, Netherlands) 1
  • Journal of Alzheimer's disease : JAD 1
  • Neurobiology of aging 1

Newest first, then by address. Each study has a usefulness score out of 100 (rubric version 4.0); tap a score to see what makes it up. Tags were assigned automatically from each study's title and summary. The same studies as a filter in the library.

  1. Objective subtle cognitive decline and plasma phosphorylated tau181: Early markers of Alzheimer's disease-related declines

    ✓ Peer-reviewed alzhub usefulness score 71 out of 100. Alzheimer's & dementia (Amsterdam, Netherlands) 2021-10-14 Biomarkers

    Despite assumptions that cognitive changes invariably follow biomarker changes, early neuropsychological difficulties may emerge before/concurrently with plasma p-tau181 changes.

  2. Apathy and anxiety are early markers of Alzheimer's disease

    ✓ Peer-reviewed alzhub usefulness score 60 out of 100. Neurobiology of aging 2019-10-19 Brain scans and imaging

    In this study, we investigated associations between neuropsychiatric symptoms (i.e., apathy, anxiety, and depression) and cerebral atrophy, white matter lesions (WML), beta-amyloid (Aβ) deposition, and cognitive decline in a nondemented sample. 104 cognitively unimpaired and 53 subjects with mild cognitive impairment were followed for up to 4 years within the Swedish BioFINDER study.

  3. Apolipoprotein C-III is an amyloid-β-binding protein and an early marker for Alzheimer's disease

    ✓ Peer-reviewed alzhub usefulness score 41 out of 100. Journal of Alzheimer's disease : JAD 2014-01-01 Treatments

    It has been demonstrated that peripheral injection of anti-amyloid-β (Aβ) antibodies to patients with Alzheimer's disease (AD) and AD transgenic mice facilitate Aβ clearance.

All topic tags with their study counts