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Home › Research › Topics › channel

channel

4 studies tagged channel, dated 2008-06-01 to 2022-05-11. Bibliography: BibTeX (.bib) · RIS (.ris)

Newest in this topic

  1. Cannabidiol Enhances Microglial Beta-Amyloid Peptide Phagocytosis and Clearance via Vanilloid Family Type 2 Channel Activation 2022-05-11
  2. TRPA1 channels promote astrocytic Ca2+ hyperactivity and synaptic dysfunction mediated by oligomeric forms of amyloid-β peptide 2017-07-06
  3. Enhanced expression of the voltage-dependent anion channel 1 (VDAC1) in Alzheimer's disease transgenic mice: an insight into the pathogenic effects of amyloid-β 2011-01-01

Studies

4 studies carry this tag.

Date range

2008 to 2022

Top journals

  • International journal of molecular sciences 1
  • Journal of Alzheimer's disease : JAD 1
  • Molecular neurodegeneration 1
  • Neuron 1

Newest first, then by address. Each study has a usefulness score out of 100 (rubric version 4.0); tap a score to see what makes it up. Tags were assigned automatically from each study's title and summary. The same studies as a filter in the library.

  1. Cannabidiol Enhances Microglial Beta-Amyloid Peptide Phagocytosis and Clearance via Vanilloid Family Type 2 Channel Activation

    ✓ Peer-reviewed alzhub usefulness score 43 out of 100. International journal of molecular sciences 2022-05-11 TREM2 and immune genetics

    Alzheimer's disease (AD) is associated with the accumulation and aggregation of amyloid in the brain.

  2. TRPA1 channels promote astrocytic Ca2+ hyperactivity and synaptic dysfunction mediated by oligomeric forms of amyloid-β peptide

    ✓ Peer-reviewed alzhub usefulness score 41 out of 100. Molecular neurodegeneration 2017-07-06 Genetics

    Our experiments demonstrate that astrocytes contribute to early Aβo toxicity exhibiting a global and local Ca 2+ hyperactivity that involves TRPA1 channels and is related to neuronal hyperactivity.

  3. Enhanced expression of the voltage-dependent anion channel 1 (VDAC1) in Alzheimer's disease transgenic mice: an insight into the pathogenic effects of amyloid-β

    ✓ Peer-reviewed alzhub usefulness score 36 out of 100. Journal of Alzheimer's disease : JAD 2011-01-01 Genetics

    The mitochondrial voltage-dependent anion channel 1 (VDAC1) is involved in the release of apoptotic proteins with possible relevance in Alzheimer's disease (AD) neuropathology.

  4. Mechanism of Ca2+ disruption in Alzheimer's disease by presenilin regulation of InsP3 receptor channel gating

    ✓ Peer-reviewed alzhub usefulness score 36 out of 100. Neuron 2008-06-01 Risk genes beyond APOE

    Mutations in presenilins (PS) are the major cause of familial Alzheimer's disease (FAD) and have been associated with calcium (Ca2+) signaling abnormalities.

All topic tags with their study counts