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Newest in this topic

  1. APOE ɛ4 dose associates with increased brain iron and β-amyloid via blood-brain barrier dysfunction 2022-04-28
  2. Lithium suppression of tau induces brain iron accumulation and neurodegeneration 2016-07-12
  3. Alzheimer's disease therapeutics targeted to the control of amyloid precursor protein translation: maintenance of brain iron homeostasis 2014-02-07

Studies

3 studies carry this tag.

Date range

2014 to 2022

Top journals

  • Biochemical pharmacology 1
  • Journal of neurology, neurosurgery, and psychiatry 1
  • Molecular psychiatry 1

Newest first, then by address. Each study has a usefulness score out of 100 (rubric version 4.0); tap a score to see what makes it up. Tags were assigned automatically from each study's title and summary. The same studies as a filter in the library.

  1. APOE ɛ4 dose associates with increased brain iron and β-amyloid via blood-brain barrier dysfunction

    ✓ Peer-reviewed alzhub usefulness score 55 out of 100. Journal of neurology, neurosurgery, and psychiatry 2022-04-28 Brain scans and imaging

    Our results suggest that an increased APOE ɛ4 dose is associated with decreased effective brain-waste clearance, such as iron and β-amyloid, through the BBB.

  2. Lithium suppression of tau induces brain iron accumulation and neurodegeneration

    ✓ Peer-reviewed alzhub usefulness score 41 out of 100. Molecular psychiatry 2016-07-12 Drug development

    Lithium is a first-line therapy for bipolar affective disorder.

  3. Alzheimer's disease therapeutics targeted to the control of amyloid precursor protein translation: maintenance of brain iron homeostasis

    ✓ Peer-reviewed alzhub usefulness score 36 out of 100. Biochemical pharmacology 2014-02-07 Treatments

    The neurotoxicity of amyloid beta (Aβ), a major cleavage product of the amyloid precursor protein (APP), is enhanced by iron, as found in the amyloid plaques of Alzheimer's disease (AD) patients.

All topic tags with their study counts