Get help now — free helplines

You can call any of these for free. You do not need a diagnosis, an appointment, or a crisis to call — questions and plain exhaustion are reasons enough.

  • Alzheimer's Association 24/7 Helpline 800-272-3900 24 hours a day, 365 days a year Free, confidential support and information for people with dementia, caregivers, families and the public. Master's-level clinicians. Also Spanish.
  • 988 Suicide & Crisis Lifeline 988 24/7 Call or text 988, or chat at 988lifeline.org. For anyone in emotional distress or a mental-health crisis, including caregivers.
  • Alzheimer's Foundation of America (AFA) Helpline 866-232-8484 9:00 am - 9:00 pm ET, seven days a week Licensed social workers providing support, info and referrals. Also text message and web chat. Text: 646-586-5283

In a medical emergency, call 911. More numbers, and what each line does, on the help page.

Newest in this topic

  1. Synaptic basis of Alzheimer's disease: Focus on synaptic amyloid beta, P-tau and mitochondria 2020-11-04
  2. Lipidomic and Transcriptomic Basis of Lysosomal Dysfunction in Progranulin Deficiency 2017-09-01

Studies

2 studies carry this tag.

Date range

2017 to 2020

Top journals

  • Ageing research reviews 1
  • Cell reports 1

Newest first, then by address. Each study has a usefulness score out of 100 (rubric version 4.0); tap a score to see what makes it up. Tags were assigned automatically from each study's title and summary. The same studies as a filter in the library.

  1. Synaptic basis of Alzheimer's disease: Focus on synaptic amyloid beta, P-tau and mitochondria

    ✓ Peer-reviewed alzhub usefulness score 40 out of 100. Ageing research reviews 2020-11-04 Genetics

    Alzheimer's disease (AD) is a progressive and synaptic failure disease.

  2. Lipidomic and Transcriptomic Basis of Lysosomal Dysfunction in Progranulin Deficiency

    ✓ Peer-reviewed alzhub usefulness score 41 out of 100. Cell reports 2017-09-01 Genetics

    Defective lysosomal function defines many neurodegenerative diseases, such as neuronal ceroid lipofuscinoses (NCL) and Niemann-Pick type C (NPC), and is implicated in Alzheimer's disease (AD) and frontotemporal lobar degeneration (FTLD-TDP) with progranulin (PGRN) deficiency.

All topic tags with their study counts