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Home › Research › Topics › appswe

appswe

3 studies tagged appswe, dated 2003-10-01 to 2007-02-01. Bibliography: BibTeX (.bib) · RIS (.ris)

Newest in this topic

  1. Impaired spatial learning in the APPSwe + PSEN1DeltaE9 bigenic mouse model of Alzheimer's disease 2007-02-01
  2. Gender differences in the amount and deposition of amyloidbeta in APPswe and PS1 double transgenic mice 2003-12-01
  3. Lipopolysaccharide-induced-neuroinflammation increases intracellular accumulation of amyloid precursor protein and amyloid beta peptide in APPswe transgenic mice 2003-10-01

Studies

3 studies carry this tag.

Date range

2003 to 2007

Top journals

  • Neurobiology of disease 2
  • Genes, brain, and behavior 1

Newest first, then by address. Each study has a usefulness score out of 100 (rubric version 4.0); tap a score to see what makes it up. Tags were assigned automatically from each study's title and summary. The same studies as a filter in the library.

  1. Impaired spatial learning in the APPSwe + PSEN1DeltaE9 bigenic mouse model of Alzheimer's disease

    ✓ Peer-reviewed alzhub usefulness score 36 out of 100. Genes, brain, and behavior 2007-02-01 Risk genes beyond APOE

    Mice co-expressing the Swedish amyloid precursor protein mutation (APP(Swe)) and exon 9 deletion (DeltaE9) of the PSEN1 gene begin to develop amyloid plaques at 6-7 months of age.

  2. Gender differences in the amount and deposition of amyloidbeta in APPswe and PS1 double transgenic mice

    ✓ Peer-reviewed alzhub usefulness score 36 out of 100. Neurobiology of disease 2003-12-01 Risk genes beyond APOE

    Transgenic mice carrying both the human amyloid precursor protein (APP) with the Swedish mutation and the presenilin-1 A246E mutation (APP/PS1 mice) develop Alzheimer's disease-like amyloidbeta protein (Abeta) deposits around 9 months of age.

  3. Lipopolysaccharide-induced-neuroinflammation increases intracellular accumulation of amyloid precursor protein and amyloid beta peptide in APPswe transgenic mice

    ✓ Peer-reviewed alzhub usefulness score 36 out of 100. Neurobiology of disease 2003-10-01 Risk genes beyond APOE

    The present study was designed to examine whether brain inflammation caused by systemic administration of lipopolysaccharides (LPS) alters the expression/processing of amyloid precursor protein (APP) and increases the generation of amyloid beta peptide (Abeta).

All topic tags with their study counts