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Home › Research › Topics › antisense

antisense

4 studies tagged antisense, dated 2000-01-01 to 2023-10-19. Bibliography: BibTeX (.bib) · RIS (.ris)

Newest in this topic

  1. Antisense oligonucleotide-based targeting of Tau-tubulin kinase 1 prevents hippocampal accumulation of phosphorylated tau in PS19 tauopathy mice 2023-10-19
  2. Structural Diversity of Sense and Antisense RNA Hexanucleotide Repeats Associated with ALS and FTLD 2020-01-25
  3. Antisense directed against PS-1 gene decreases brain oxidative markers in aged senescence accelerated mice (SAMP8) and reverses learning and memory impairment: a proteomics study 2013-06-15

Studies

4 studies carry this tag.

Date range

2000 to 2023

Top journals

  • Acta neuropathologica communications 1
  • Free radical biology & medicine 1
  • Journal of molecular medicine (Berlin, Germany) 1
  • Molecules (Basel, Switzerland) 1

Newest first, then by address. Each study has a usefulness score out of 100 (rubric version 4.0); tap a score to see what makes it up. Tags were assigned automatically from each study's title and summary. The same studies as a filter in the library.

  1. Antisense oligonucleotide-based targeting of Tau-tubulin kinase 1 prevents hippocampal accumulation of phosphorylated tau in PS19 tauopathy mice

    ✓ Peer-reviewed alzhub usefulness score 43 out of 100. Acta neuropathologica communications 2023-10-19 Drug development

    Tau tubulin kinase-1 (TTBK1), a neuron-specific tau kinase, is highly expressed in the entorhinal cortex and hippocampal regions, where early tau pathology evolves in Alzheimer's disease (AD).

  2. Structural Diversity of Sense and Antisense RNA Hexanucleotide Repeats Associated with ALS and FTLD

    ✓ Peer-reviewed alzhub usefulness score 41 out of 100. Molecules (Basel, Switzerland) 2020-01-25 Drug development

    The hexanucleotide expansion GGGGCC located in C9orf72 gene represents the most common genetic cause of amyotrophic lateral sclerosis (ALS) and frontotemporal lobar dementia (FTLD).

  3. Antisense directed against PS-1 gene decreases brain oxidative markers in aged senescence accelerated mice (SAMP8) and reverses learning and memory impairment: a proteomics study

    ✓ Peer-reviewed alzhub usefulness score 36 out of 100. Free radical biology & medicine 2013-06-15 Drug development

    Amyloid β-peptide (Aβ) plays a central role in the pathophysiology of Alzheimer's disease (AD) through the induction of oxidative stress.

  4. Antisense prevention of neuronal damages following head injury in mice

    ✓ Peer-reviewed alzhub usefulness score 36 out of 100. Journal of molecular medicine (Berlin, Germany) 2000-01-01 Drug development

    Closed head injury (CHI) is an important cause of death among young adults and a prominent risk factor for nonfamilial Alzheimer's disease.

All topic tags with their study counts