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Home › Research › Topics › amyloidosis

amyloidosis

7 studies tagged amyloidosis, dated 2015-06-18 to 2025-12-17. Bibliography: BibTeX (.bib) · RIS (.ris)

Newest in this topic

  1. Lysophosphatidylcholines are associated with amyloidosis in early stages of Alzheimer's disease 2025-12-17
  2. INPP5D modulates TREM2 loss-of-function phenotypes in a β-amyloidosis mouse model 2023-03-13
  3. The Combined Effects of Amyloidosis and Serotonin Deficiency by Tryptophan Hydroxylase-2 Knockout Impacts Viability of the APP/PS1 Mouse Model of Alzheimer's Disease 2022-01-01

Studies

7 studies carry this tag.

Date range

2015 to 2025

Top journals

  • Nature aging 2
  • Alzheimer's research & therapy 1
  • Current Alzheimer research 1
  • Journal of Alzheimer's disease : JAD 1
  • PloS one 1

6 journals in all.

Newest first, then by address. Each study has a usefulness score out of 100 (rubric version 4.0); tap a score to see what makes it up. Tags were assigned automatically from each study's title and summary. The same studies as a filter in the library.

  1. Lysophosphatidylcholines are associated with amyloidosis in early stages of Alzheimer's disease

    ✓ Peer-reviewed alzhub usefulness score 60 out of 100. Nature aging 2025-12-17 Biomarkers

    Circulating metabolites can identify biochemical risk factors related to Alzheimer's disease (AD).

  2. INPP5D modulates TREM2 loss-of-function phenotypes in a β-amyloidosis mouse model

    ✓ Peer-reviewed alzhub usefulness score 40 out of 100. iScience 2023-03-13 TREM2 and immune genetics

    The genetic associations of TREM2 loss-of-function variants with Alzheimer disease (AD) indicate the protective roles of microglia in AD pathogenesis.

  3. The Combined Effects of Amyloidosis and Serotonin Deficiency by Tryptophan Hydroxylase-2 Knockout Impacts Viability of the APP/PS1 Mouse Model of Alzheimer's Disease

    ✓ Peer-reviewed alzhub usefulness score 40 out of 100. Journal of Alzheimer's disease : JAD 2022-01-01 Biomarkers

    The results suggest that Tph2 inactivation influences AβPP processing, at least in the hippocampus, although levels of Aβ are unchanged.

  4. Alzheimer's disease modification mediated by bone marrow-derived macrophages via a TREM2-independent pathway in mouse model of amyloidosis

    ✓ Peer-reviewed alzhub usefulness score 43 out of 100. Nature aging 2021-12-20 TREM2 and immune genetics

    Microglia and monocyte-derived macrophages (MDM) are key players in dealing with Alzheimer's disease.

  5. Associations of Alzheimer's disease risk variants with gene expression, amyloidosis, tauopathy, and neurodegeneration

    ✓ Peer-reviewed alzhub usefulness score 59 out of 100. Alzheimer's research & therapy 2021-01-08 Genetics

    Genetic variants might play functional roles and suggest potential mechanisms in AD pathogenesis, which opens doors to uncover novel targets for AD treatment.

  6. Microdose Lithium NP03 Diminishes Pre-Plaque Oxidative Damage and Neuroinflammation in a Rat Model of Alzheimer's-like Amyloidosis

    ✓ Peer-reviewed alzhub usefulness score 38 out of 100. Current Alzheimer research 2018-01-01 TREM2 and immune genetics

    These results suggest that NP03 may function to slow the AD-like pathology in part by modifying oxidative/nitrosative damage and neuroinflammation, raising the possibility that low doses of microencapsulated lithium might be of therapeutic-preventive value during very early or preclinical AD.

  7. Cannabinoid CB2 Receptors in a Mouse Model of Aβ Amyloidosis: Immunohistochemical Analysis and Suitability as a PET Biomarker of Neuroinflammation

    ✓ Peer-reviewed alzhub usefulness score 40 out of 100. PloS one 2015-06-18 Biomarkers

    In Alzheimer's disease (AD), one of the early responses to Aβ amyloidosis is recruitment of microglia to areas of new plaque.

All topic tags with their study counts