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amyloid-imaging
6 studies tagged amyloid-imaging, dated 2006-08-14 to 2015-05-12. Bibliography: BibTeX (.bib) · RIS (.ris)
Newest in this topic
- Development of a process to disclose amyloid imaging results to cognitively normal older adult research participants 2015-05-12
- Correspondence between in vivo (11)C-PiB-PET amyloid imaging and postmortem, region-matched assessment of plaques 2012-08-05
- Predicting sites of new hemorrhage with amyloid imaging in cerebral amyloid angiopathy 2012-07-11
Studies
6 studies carry this tag.
Date range
2006 to 2015
Top journals
- Acta neuropathologica 1
- Alzheimer's research & therapy 1
- Journal of neurochemistry 1
- NeuroImage 1
- Neurology 1
6 journals in all.
Newest first, then by address. Each study has a usefulness score out of 100 (rubric version 4.0); tap a score to see what makes it up. Tags were assigned automatically from each study's title and summary. The same studies as a filter in the library.
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We developed a document for use with potential study participants to describe the process of amyloid imaging and the implications of amyloid imaging results; and a disclosure process with attention to ongoing monitoring of both mood and safety to receive this information.
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The definitive Alzheimer's disease (AD) diagnosis requires postmortem confirmation of neuropathological hallmarks-amyloid-β (Aβ) plaques and neurofibrillary tangles (NFTs).
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Our results provide direct evidence that new CAA-related hemorrhages occur preferentially at sites of increased amyloid deposition and suggest that PiB-PET imaging may be a useful tool in prediction of incident hemorrhages in patients with CAA.
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36/100
The distinctive cortical uptake of the tracer (18)F-FDDNP (2-(1-{6-[(2-fluoroethyl(methyl)amino]-2-naphthyl}ethylidene)malononitrile) in Alzheimer's disease (AD) is believed to be because of its binding to both neurofibrillary tangles (NFTs) and highly fibrillar senile plaques.
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Imaging agents capable of assessing amyloid-beta (Abeta) content in vivo in the brains of Alzheimer's disease (AD) subjects likely will be important as diagnostic agents to detect Abeta plaques in the brain as well as to help test the amyloid cascade hypothesis of AD and as an aid to assess the efficacy of anti-amyloid therapeutics currently under development and in clinical trials.
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Deposition of amyloid plaques is believed to be a central event in the development of Alzheimer's disease (AD).