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associations
7 studies tagged associations, dated 2015-01-01 to 2024-05-15. Bibliography: BibTeX (.bib) · RIS (.ris)
Newest in this topic
- Associations among plasma, MRI, and amyloid PET biomarkers of Alzheimer's disease and related dementias and the impact of health-related comorbidities in a community-dwelling cohort 2024-05-15
- The Associations of Phosphorylated Tau 181 and Tau 231 Levels in Plasma and Cerebrospinal Fluid with Cognitive Function in Alzheimer's Disease: A Systematic Review and Meta-Analysis 2024-01-01
- Associations of the A/T/N profiles in PET, CSF, and plasma biomarkers with Alzheimer's disease neuropathology at autopsy 2023-07-28
Studies
7 studies carry this tag.
Date range
2015 to 2024
Top journals
- Alzheimer's & dementia : the journal of the Alzheimer's Association 2
- Journal of Alzheimer's disease : JAD 1
- Journal of Parkinson's disease 1
- Journal of clinical medicine 1
- Journal of neurology, neurosurgery, and psychiatry 1
6 journals in all.
Newest first, then by address. Each study has a usefulness score out of 100 (rubric version 4.0); tap a score to see what makes it up. Tags were assigned automatically from each study's title and summary. The same studies as a filter in the library.
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Plasma biomarkers differed across diagnostic groups (DEM > MCI > CU), were altered in Aβ-PET-positive individuals, and were associated with poorer brain health and kidney function.
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CSF p-tau181 and p-tau231 biomarkers distinguished AD, MCI, and healthy populations.
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This study supports that PET- and CSF-related A/T/N profiles can be used to predict accurately the stages of AD neuropathology.
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CSF markers of iron and neuroinflammation have distinct associations with disease stages, while iron may be more intimately associated with apolipoprotein E and tau pathology.
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Alzheimer disease (AD) is a chronic neurodegenerative disease with a multitude of contributing genetic factors, many of which are related to inflammation.
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Brain degeneration in patients with Alzheimer's disease (AD) results from the accumulation of pathological amyloid- (Aβ) plaques and tau protein tangles, leading to altered plasma levels of biomarkers.
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The association between reduced CSF α-synuclein concentrations and cognition suggests that α-synuclein pathology contributes to early cognitive impairment in PD, in particular to executive-attentional dysfunction.