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acidic-protein
11 studies tagged acidic-protein, dated 1994-02-01 to 2025-07-21. Bibliography: BibTeX (.bib) · RIS (.ris)
Newest in this topic
- The Role of Glial Fibrillary Acidic Protein in the Neuropathology of Alzheimer's Disease and Its Potential as a Blood Biomarker for Early Diagnosis and Progression 2025-07-21
- Glial fibrillary acidic protein in Alzheimer's disease: a narrative review 2024-11-07
- Clinical and biological relevance of glial fibrillary acidic protein in Alzheimer's disease 2023-11-03
Studies
11 studies carry this tag.
Date range
1994 to 2025
Top journals
- Journal of Alzheimer's disease : JAD 3
- Alzheimer's research & therapy 1
- Brain communications 1
- European neurology 1
- Molecular neurobiology 1
9 journals in all.
Newest first, then by address. Each study has a usefulness score out of 100 (rubric version 4.0); tap a score to see what makes it up. Tags were assigned automatically from each study's title and summary. The same studies as a filter in the library.
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Alzheimer's disease (AD) is a neurodegenerative disease characterised by neuropathological hallmarks, including extracellular amyloid plaques and neurofibrillary tangles.
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Astrocytes are fundamental in neural functioning and homeostasis in the central nervous system.
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Our findings indicate that GFAP is of potential clinical utility in screening amyloid pathology and predicting future cognitive decline.
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There is an association between plasma GFAP and regional 18F-SMBT-1 PET, and this association appears to be dependent on brain Aβ load.
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Serum GFAP is increased in FTLD, correlates with cognition and GABAergic deficits, and thus shows promise as a biomarker of disease severity in FTLD.
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Glial fibrillary acidic protein (GFAP) and protein S-100B are established indicators of astrogliosis in neuropathology.
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Recent studies demonstrate roles for activation of caspases and cleavage of cellular proteins within neurons of the Alzheimer's disease (AD) brain.
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Chronic inflammation is known to play an important role in the heterogeneous pathogenesis of Alzheimer's disease (AD).
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We describe a new human isoform, GFAP epsilon, of the intermediary filament protein GFAP (glial fibrillary acidic protein).
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To gain insight of the underlying mechanisms of astroglial response to Alzheimer's disease (AD), the level of glial fibrillary acidic protein (GFAP) in cerebrospinal fluid (CSF) from controls and AD subjects were immunochemically determined, and the correlation between that level and dementia severity of AD patients was evaluated.
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Transforming growth factor-beta 1 was studied as a possible regulator of messenger RNAs in astrocytes and neurons that increase after hippocampal deafferentation by perforant path transection: tubulin alpha 1, clusterin and glial fibrillary acidic protein messenger RNA.