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Home › Research › Topics › absence

absence

4 studies tagged absence, dated 2001-08-01 to 2026-07-01. Bibliography: BibTeX (.bib) · RIS (.ris)

Newest in this topic

  1. Donanemab Infusion and Recurrent Seizures in a Patient With Alzheimer's Disease and Epilepsy in the Absence of Amyloid-Related Imaging Abnormalities (ARIA) 2026-07-01
  2. In vivo hyperphosphorylation of tau is associated with synaptic loss and behavioral abnormalities in the absence of tau seeds 2024-12-24
  3. Absence of C1q leads to less neuropathology in transgenic mouse models of Alzheimer's disease 2004-07-01

Studies

4 studies carry this tag.

Date range

2001 to 2026

Top journals

  • Cureus 1
  • Experimental neurology 1
  • Nature neuroscience 1
  • The Journal of neuroscience : the official journal of the Society for Neuroscience 1

Newest first, then by address. Each study has a usefulness score out of 100 (rubric version 4.0); tap a score to see what makes it up. Tags were assigned automatically from each study's title and summary. The same studies as a filter in the library.

  1. Donanemab Infusion and Recurrent Seizures in a Patient With Alzheimer's Disease and Epilepsy in the Absence of Amyloid-Related Imaging Abnormalities (ARIA)

    ✓ Peer-reviewed alzhub usefulness score 52 out of 100. Cureus 2026-07-01 Treatments

    Inter-ictal magnetic resonance imaging (MRI), including fluid-attenuated inversion recovery (FLAIR) and susceptibility-weighted imaging (SWI) sequences, demonstrated no evidence of ARIA-E or ARIA-H. Routine electroencephalography (EEG) revealed no interictal epileptiform discharges.

  2. In vivo hyperphosphorylation of tau is associated with synaptic loss and behavioral abnormalities in the absence of tau seeds

    ✓ Peer-reviewed alzhub usefulness score 48 out of 100. Nature neuroscience 2024-12-24 Disease mechanisms

    Tau pathology is a hallmark of several neurodegenerative diseases, including frontotemporal dementia and Alzheimer's disease.

  3. Absence of C1q leads to less neuropathology in transgenic mouse models of Alzheimer's disease

    ✓ Peer-reviewed alzhub usefulness score 36 out of 100. The Journal of neuroscience : the official journal of the Society for Neuroscience 2004-07-01 Risk genes beyond APOE

    C1q, the recognition component of the classical complement activation pathway, is a multifunctional protein known to be expressed in brain of Alzheimer's disease (AD) patients.

  4. Behavioral phenotyping of GFAP-apoE3 and -apoE4 transgenic mice: apoE4 mice show profound working memory impairments in the absence of Alzheimer's-like neuropathology

    ✓ Peer-reviewed alzhub usefulness score 52 out of 100. Experimental neurology 2001-08-01 Biomarkers

    For the purpose of studying the potential neurobehavioral effects of different human apolipoprotein E (apoE) isoforms produced within the brain, transgenic (TG) mice were generated in which human apoE3 or apoE4 isoforms were under control of an astrocyte-specific, glial fibrillary acidic protein promoter and these TG mice were bred back to apoE knockout (KO) mice.

All topic tags with their study counts