News
What's happening in Alzheimer's care, research funding, and treatment — summarized simply, each item linked to the original reporting.
Free to read, “May need a subscription” and “Archived copy”: what they mean
Whether an article is free to read is a guess, not a test of your access. Each item is one of three:
- No label: free to read (304 of 349). The publisher’s page marks it free, or the outlet publishes free to read: Medical Xpress, ScienceDaily, NIH (incl. NIA and PMC), Alzheimer's Association, Alzheimer's News Today, NPR, Alzheimer's Disease International, or a .gov site.
- May need a subscription (11 of 349). The publisher’s own page marks the article as not free to read, or — when the page says nothing — the outlet sells subscriptions: The New York Times, STAT, Scientific American, The Washington Post, The Wall Street Journal, Financial Times, The Economist, Bloomberg, The Atlantic, The New Yorker, Wired, Los Angeles Times, The Boston Globe, The Times, The Telegraph, New Scientist, Endpoints News, Science. Each badge says which.
- “Free to read? Not known” (34 of 349). The outlet is on neither list and its page said nothing. We do not call these paywalled.
Archived copy links go to a copy the Internet Archive (web.archive.org) saved, dated the day it was saved. We show one only where the Internet Archive reported a working saved copy when we checked; an archived copy of a subscription article sometimes shows only its opening. 265 of 349 items have one.
Scientists restore a brain protein and reverse signs of aging in mice
Scientists found that declining levels of the brain protein Menin may help drive aging, with lower levels in mice linked to inflammation, cognitive decline, weaker bones, and thinner skin. …
Read at ScienceDaily → Archived copy (1 Oct 2026), Internet ArchiveScientists discover a major brain shift between ages 50 and 75
Scientists have uncovered sweeping changes in how the human brain controls and organizes its genome beginning in midlife, offering new clues to why aging sharply increases the risk of Alzheimer’s and other neurodegenerative diseases. …
Read at ScienceDaily → Archived copy (21 Sep 2026), Internet ArchiveScientists find a new layer of Alzheimer’s hidden in the genome
Scientists found that the 3D organization of DNA is disrupted in several types of brain cells affected by Alzheimer’s disease, altering how important genes are switched on and off. …
Read at ScienceDaily → Archived copy (20 Sep 2026), Internet ArchiveReaching 90 without dementia doesn’t mean you’re in the clear
A study of people over 90 found that dementia risk remains strongly shaped by sex, race and genetics, with women and black participants facing notably higher risks. …
A major Alzheimer’s risk gene may shrink brain cells years before symptoms
Researchers have uncovered a possible reason why the Alzheimer’s risk gene APOE4 can disrupt the brain long before memory problems begin. In mice, APOE4 increased a protein called Nell2, shrinking neurons and making memory circuits unusually active. …
Read at ScienceDaily → Archived copy (1 Oct 2026), Internet ArchiveA little-known protein may be fueling Alzheimer’s — and scientists found a way to block it
An experimental Alzheimer’s compound prevented damaging protein clumps from forming in the brains of mice, helping nerve cells survive longer and reducing amyloid buildup. …
Read at ScienceDaily →Women taking estrogen had fewer signs of Alzheimer’s in their brains
Women who used estrogen-only hormone therapy later in life were less likely to develop dementia and showed fewer signs of Alzheimer’s disease in their brains, according to a large study of more than 21,000 participants. …
Read at ScienceDaily → Archived copy (3 Oct 2026), Internet ArchiveNearly half of dementia cases may be linked to risks you can change
A long-term brain study found that common risks such as smoking, high blood pressure, heart disease, and high blood lipids were strongly linked to damage associated with vascular dementia. …
Scientists discover a hidden brain shift that begins around age 50
Beginning around age 50, the brain’s memory center appears to lose many of its longtime immune cells and replace them with more inflammatory ones. This surprising shift may help explain how normal aging sets the stage for Alzheimer’s disease and dementia.
Read at ScienceDaily → Archived copy (27 Sep 2026), Internet ArchiveAI can tell if your brain is aging faster than you are
A person’s sleeping brain may reveal warning signs of dementia long before memory problems begin. Researchers used machine learning to analyze EEG recordings from about 7,000 adults and found that an older-than-expected “brain age” was tied to a sharply higher dementia risk. …
Scientists discover why poor sleep may harm some brains more
A gene involved in the brain’s overnight cleaning system may change how strongly poor sleep affects Alzheimer’s-related brain changes. The findings point toward more personalized prevention strategies based on both genetics and sleep habits.
Read at ScienceDaily → Archived copy (1 Aug 2026), Internet ArchiveScientists discover how obesity may fuel Alzheimer’s disease
Researchers have uncovered a possible biological pathway connecting obesity to Alzheimer’s disease. Obesity appears to raise levels of certain fat molecules that can travel to the brain, disrupt its immune defenses, and promote harmful amyloid buildup. …
Read at ScienceDaily →A hidden Alzheimer’s tipping point may decide who gets dementia
Scientists have identified a possible tipping point that helps determine whether Alzheimer’s-related brain changes lead to dementia. The key may lie in how the brain’s immune cells respond to plaques and tau…
Superagers keep youthful memories but their DNA does not explain why
Scientists expected SuperAgers might simply inherit fewer Alzheimer’s-related risks, but their genes looked much like those of typical older adults. …
Read at ScienceDaily → Archived copy (25 Sep 2026), Internet ArchiveAPOE2 may protect the brain from Alzheimer’s and aging
The longevity-linked APOE2 gene appears to protect brain cells by reducing DNA damage and helping neurons recover from stress. The finding could open a new path toward treatments that mimic APOE2’s defenses in people at higher genetic risk for Alzheimer’s.
Alzheimer’s breakthrough: Scientists restore two hours of sleep without clearing brain plaques
Researchers have uncovered a surprising culprit behind sleep loss in Alzheimer’s disease: the brain’s own immune cells. In mice with amyloid plaques, overactive microglia triggered inflammation that kept the brain from getting enough deep, restorative sleep. …
Read at ScienceDaily → Archived copy (22 Sep 2026), Internet ArchiveScientists discover a protein that protects the brain from Alzheimer’s damage
A protein called SORLA may act as a powerful defense against the toxic tau tangles involved in Alzheimer’s disease. Mice with extra SORLA had less tau accumulation, less brain atrophy…
Read at ScienceDaily → Archived copy (2 Oct 2026), Internet ArchiveNew Alzheimer's drug repairs DNA damage and reduces brain inflammation
A drug originally developed for spinal cord injury may offer a fresh approach to treating Alzheimer’s disease. In mouse studies, KCL-286 repaired dangerous DNA damage, reduced inflammation…
Read at ScienceDaily → Archived copy (17 Jul 2026), Internet ArchiveThe hidden skeleton “gatekeeper” inside brain cells could help fight Alzheimer's
Researchers have discovered that a microscopic skeleton inside neurons does much more than hold cells together. It acts as a gatekeeper that controls what brain cells absorb and when they absorb it. …
Read at ScienceDaily → Archived copy (15 Jul 2026), Internet ArchiveAlzheimer's tau protein has a surprising secret role in memory
Researchers found that tau is essential for turning new experiences into lasting memories by helping organize the brain's memory-storing cells. …
Read at ScienceDaily →Scientists may have finally found how Alzheimer's kills brain cells
Researchers have identified a previously overlooked mechanism of brain cell death that appears to play a major role in Alzheimer's disease and frontotemporal dementia. …
Read at ScienceDaily → Archived copy (23 Sep 2026), Internet ArchiveScientists discover why some brains resist Alzheimer's
Some brains appear to fight back against Alzheimer's by helping immature brain cells survive damage instead of succumbing to it. Understanding this natural resilience could point researchers toward entirely new ways to protect memory and slow dementia.
Read at ScienceDaily →Scientists may have finally found how Alzheimer's spreads through the brain
A common brain protein may be giving Alzheimer’s disease an unexpected way to spread, carrying toxic Tau proteins from damaged neurons into healthy ones. …
Millions take omega-3 fish oil for brain health but a new study found no benefit
Fish oil supplements successfully delivered omega-3s to the brain, but a two-year study found no meaningful benefits for memory, cognition, or Alzheimer’s-related brain changes. …
Tubulin prevents toxic brain protein clumps linked to Alzheimer’s and Parkinson’s
Scientists at Baylor College of Medicine may have uncovered a promising new way to combat Alzheimer’s and Parkinson’s disease. Instead of trying to stop Tau and alpha-synuclein proteins from gathering into tiny droplets inside brain cells, the researchers found that tubulin…
Read at ScienceDaily → Archived copy (22 Jun 2026), Internet ArchiveScientists reprogram brain immune cells to fight Alzheimer’s
A newly identified molecule called OLE helped restore the brain’s immune cells to a more protective state in Alzheimer’s models. The treatment reduced toxic plaque buildup and improved memory, raising hopes for a new therapeutic approach.
Read at ScienceDaily →Scientists May Have Found What Really Triggers Alzheimer’s Disease
Scientists may have uncovered a hidden trigger behind Alzheimer’s disease. Instead of plaques being the root cause, amyloid beta appears to interfere with tau, a protein that helps keep neurons functioning properly. …
Read at ScienceDaily → Archived copy (19 Sep 2026), Internet ArchiveScientists discover a surprising cancer link to Alzheimer’s disease
Researchers discovered that mutations linked to blood cancers may help trigger Alzheimer’s disease by creating overly inflammatory immune cells in the brain. …
Read at ScienceDaily → Archived copy (22 Sep 2026), Internet ArchivePopular joint supplement glucosamine linked to faster Alzheimer’s progression
A major study suggests glucosamine, a popular supplement for joint pain, could be linked to faster progression from mild cognitive impairment to Alzheimer’s disease. …
Read at ScienceDaily → Archived copy (8 Oct 2026), Internet ArchiveScientists found a new Alzheimer’s trigger and a drug that stops it
Researchers have identified a new Alzheimer’s target and created an experimental compound that blocks a damaging process inside brain cells. In mice, the treatment slowed nerve cell loss, reduced Alzheimer’s-related changes, and even appeared to promote healthier aging.
Read at ScienceDaily → Archived copy (23 Sep 2026), Internet ArchiveFollow new items in a feed reader: Atom feed.