News
What's happening in Alzheimer's care, research funding, and treatment — summarized simply, each item linked to the original reporting.
Free to read, “May need a subscription” and “Archived copy”: what they mean
Whether an article is free to read is a guess, not a test of your access. Each item is one of three:
- No label: free to read (304 of 349). The publisher’s page marks it free, or the outlet publishes free to read: Medical Xpress, ScienceDaily, NIH (incl. NIA and PMC), Alzheimer's Association, Alzheimer's News Today, NPR, Alzheimer's Disease International, or a .gov site.
- May need a subscription (11 of 349). The publisher’s own page marks the article as not free to read, or — when the page says nothing — the outlet sells subscriptions: The New York Times, STAT, Scientific American, The Washington Post, The Wall Street Journal, Financial Times, The Economist, Bloomberg, The Atlantic, The New Yorker, Wired, Los Angeles Times, The Boston Globe, The Times, The Telegraph, New Scientist, Endpoints News, Science. Each badge says which.
- “Free to read? Not known” (34 of 349). The outlet is on neither list and its page said nothing. We do not call these paywalled.
Archived copy links go to a copy the Internet Archive (web.archive.org) saved, dated the day it was saved. We show one only where the Internet Archive reported a working saved copy when we checked; an archived copy of a subscription article sometimes shows only its opening. 265 of 349 items have one.
Hidden ancestral links found in motor neuron disease and dementia could speed up gene discovery
Researchers at the Macquarie University Brain Institute and Clinic have helped accelerate the search for disease-causing genes by building new genetic 'families.' …
Read at Medical Xpress →Scientists discover new Alzheimer’s drug that repairs DNA damage and reduces brain inflammation
Scientists have discovered that a drug originally developed for spinal cord injuries can tackle several key features of Alzheimer’s disease, including DNA damage and inflammation. …
Read at ScienceDaily →New human data hints at psychedelics' potential to combat inflammation
Lab studies have shown that psychedelics like psilocybin can lower inflammation and may be protective for a range of illnesses. Now, researchers are publishing some of the first data in humans that signals this holds potential for patients with neurodegenerative diseases like…
Read at NPR → Archived copy (23 Oct 2026), Internet ArchiveMouse study reveals molecular link between high-fat diets and impaired brain function
The molecular mechanisms linking a high-calorie diet to cognitive impairment remain unclear. Researchers at the Institute of Science Tokyo, Japan, investigated this association in mice…
Read at Medical Xpress →New microscopy technology may help detect neurological diseases before neurons die
Scientists studying amyotrophic lateral sclerosis (ALS), Parkinson's, Alzheimer's and other neurological diseases are getting unprecedented views inside brain cells, using powerful new microscopes to uncover changes that may begin long before neurons die. …
Read at Medical Xpress →Scientists find a way to fight Alzheimer’s brain damage without removing toxic tau
Scientists have found a promising way to protect the brain from Alzheimer’s-related damage by blocking immune cells that contribute to the destruction of neurons. …
Read at ScienceDaily → Archived copy (20 Oct 2026), Internet Archive"Mind-blowing" Alzheimer’s breakthrough: Two hours of sleep restored without clearing brain plaques
Scientists have uncovered an unexpected culprit behind sleep loss in Alzheimer’s disease: overactive immune cells in the brain. By temporarily removing these cells, researchers restored more than two hours of sleep per day in mice, revealing a promising new target for treatment.
Read at ScienceDaily → Archived copy (19 Oct 2026), Internet ArchiveLab-designed molecule reduces amyloid beta, the protein linked to Alzheimer's disease, in mice
A research team led by the Molecular and Cellular Neurobiotechnology group at the Institute for Bioengineering of Catalonia (IBEC), in collaboration with Míriam Royo's laboratory at the Institute for Advanced Chemistry of Catalonia (IQAC-CSIC)…
Read at Medical Xpress →Brain cells meant to protect us may be fueling cognitive decline
Scientists have uncovered a surprising cause of age-related cognitive decline involving brain cells previously thought to be entirely beneficial. …
Read at ScienceDaily → Archived copy (17 Oct 2026), Internet ArchiveScientists find a youth protein that helps aging brain cells act young again
Scientists have discovered that a protein linked to youth helps protect the brain’s immune cells from age-related decline. Restoring the protein in older mice improved the cells’ ability to clear debris and reduced signs of inflammation…
Read at ScienceDaily → Archived copy (18 Oct 2026), Internet ArchiveResearchers restore neuron function in mice with Alzheimer's disease
Researchers were able to restore the function of neurons linked to cognitive function and information processing in mice that mimic key features of Alzheimer's disease, showing early promise for the development of a similar targeted therapy in humans. …
Read at Medical Xpress →Who gets Alzheimer's drugs? Race, vulnerability and health status shape access
The first disease-modifying drugs for Alzheimer's disease offer patients the ability to slow cognitive decline by targeting amyloid plaques in the brain. But a new study suggests that not all patients have the same opportunity to receive these treatments.
How the biggest Alzheimer's risk gene disrupts the process of insulating neurons
One of the main ways that the APOE4 gene variant, the biggest genetic risk factor for late-onset Alzheimer's disease, undermines brain circuit function is by disrupting the process of insulating neurons with a fatty sheath that optimizes their electrical transmission.
Read at Medical Xpress → Archived copy (16 Oct 2026), Internet ArchiveBrain-protective molecule may help preserve memory during aging
As humans age, both protective and harmful biological processes shape brain health. When the balance shifts in favor of harmful processes, age-related changes in the brain can accelerate, including synapse loss, disruptions in lipid metabolism and chronic inflammation.
Read at Medical Xpress → Archived copy (18 Oct 2026), Internet ArchiveRare study reveals a striking divide inside an Alzheimer’s patient’s brain
A rare examination of an Alzheimer’s patient’s brain revealed that regions cleared of amyloid plaques had remarkably little of the harmful tau buildup associated with memory loss, while neighboring areas showed extensive damage. …
Molecular glue degraders show early promise in reducing Alzheimer's-linked brain plaques
A research team led by Indiana University School of Medicine scientists has discovered a new method for reducing the formation of amyloid plaques…
Read at Medical Xpress → Archived copy (15 Oct 2026), Internet ArchiveDementia Cases Rising; Tylenol's Effect on Brain Organoids; Early-Onset Alzheimer's
(MedPage Today) -- An estimated 11.4 million new dementia cases occurred worldwide in 2023, and the number is projected to jump to 24.7 million new cases a year by 2050, a meta-analysis of 22 cohort studies showed. (JAMA Neurology) Investigational...
Free to read? Not known for this outlet.
Read at MedPage Today → Archived copy (15 Oct 2026), Internet ArchiveThe shape of water at a key tau hotspot helps drive dementia-linked clumping
Diseases such as Alzheimer's and frontotemporal dementia are considered neurodegenerative tauopathies, in which tau proteins in the brain misfold and lead to cognitive decline. …
Read at Medical Xpress → Archived copy (13 Oct 2026), Internet ArchiveStudy offers new clues about how genetic risk for Alzheimer's affects brain immune cells
Researchers at the University of Colorado Anschutz and Pacific Northwest National Laboratory have found a new clue that may help explain why some people are more likely to develop Alzheimer's disease. …
Read at Medical Xpress → Archived copy (13 Oct 2026), Internet ArchiveDrug combination that reduces oxygen levels in tissues improves Parkinson's symptoms in mice
In a new study, Broad Institute researchers report that two small-molecule chemicals that reduce oxygen delivery to cells improved symptoms of neurodegeneration in a mouse model of Parkinson's disease. …
Read at Medical Xpress →Scientists find hidden driver of aging and a simple amino acid that fights memory loss in mice
Scientists discovered that declining levels of a protein called Menin may help drive aging, and restoring it in older mice reversed several age-related changes in just 30 days. …
Read at ScienceDaily → Archived copy (12 Oct 2026), Internet ArchiveIncreasing protective protein suppresses Parkinson's-linked buildup, more than doubling lifespan in mouse model
Parkinson's disease is the second most common neurodegenerative disease. Over the last 30 years, studies have shown that a protein called alpha-synuclein abnormally aggregates in people with Parkinson's disease…
Read at Medical Xpress → Archived copy (13 Oct 2026), Internet ArchiveManaging misfolded proteins to prevent Parkinson's disease
The protein alpha-synuclein is abundant in healthy brains and plays an important role in enabling communication between nerve cells. However, under certain conditions, misfolded alpha-synuclein proteins can accumulate into toxic clumps inside neurons. …
Read at Medical Xpress →Brain tissue removed during surgery reveals potential way to curb inflammation linked to Alzheimer's
Inflammatory conditions in the brain, including traumatic brain injury and degenerative diseases like Alzheimer's, could be targeted with an existing drug thanks to new research that shows it can work in brain cells.
Why it matters: New research in brain cells reveals an existing drug could potentially curb inflammatory conditions linked to Alzheimer's and traumatic brain injury.
Read at Medical Xpress → Archived copy (8 Oct 2026), Internet ArchiveHuman tau seeds trigger disease-specific shapes in mouse brains, new study finds
Tau is a protein that occurs naturally in the brain. It is essential for proper neuron development and for how neurons communicate with each other. The same protein, however, when misshapen or misfolded, can act as a driver of neurodegenerative diseases. …
Why it matters: Misshapen tau protein can act as a driver of neurodegenerative diseases despite its natural role in neuron development and communication.
Read at Medical Xpress → Archived copy (5 Oct 2026), Internet ArchiveResearchers slow neurodegeneration in mice by barring T cells from the brain
A new study in mice led by Washington University in St. Louis researchers suggests that stopping certain immune cells from entering the brain could limit neurodegeneration.
Why it matters: Stopping certain immune cells from entering the brain could limit neurodegeneration, according to a study in mice led by Washington University in St. Louis researchers.
May need a subscription Based on the outlet (STAT), not a check of this article.
Read at STAT News → Archived copy (30 Sep 2026), Internet ArchiveAlcohol and the Brain; The Next Alzheimer's Frontier; Mice With Human Brain Cells
(MedPage Today) -- The New York Times looked at what alcohol does to the brain and what amount, if any, may be safe. Ongoing phase I/II studies of ifezuntirgene inilparvovec (AMT-130) in Huntington's disease showed continued slowing of disease...
Free to read? Not known for this outlet.
Read at MedPage Today → Archived copy (1 Oct 2026), Internet ArchiveScientists restore a brain protein and reverse signs of aging in mice
Scientists found that declining levels of the brain protein Menin may help drive aging, with lower levels in mice linked to inflammation, cognitive decline, weaker bones, and thinner skin. …
Read at ScienceDaily → Archived copy (1 Oct 2026), Internet ArchiveAlzheimer’s may leave a mark on the brain long before plaques appear
Brain changes associated with Alzheimer’s may begin at least seven years before amyloid plaques become detectable with current PET scans. Researchers uncovered the signal by tracking healthy older adults with repeated brain imaging over nearly 20 years. …
Read at ScienceDaily → Archived copy (16 Sep 2026), Internet ArchiveA major Alzheimer’s risk gene may shrink brain cells years before symptoms
Researchers have uncovered a possible reason why the Alzheimer’s risk gene APOE4 can disrupt the brain long before memory problems begin. In mice, APOE4 increased a protein called Nell2, shrinking neurons and making memory circuits unusually active. …
Read at ScienceDaily → Archived copy (1 Oct 2026), Internet ArchiveFollow new items in a feed reader: Atom feed.