News
What's happening in Alzheimer's care, research funding, and treatment — summarized simply, each item linked to the original reporting.
Free to read, “May need a subscription” and “Archived copy”: what they mean
Whether an article is free to read is a guess, not a test of your access. Each item is one of three:
- No label: free to read (308 of 354). The publisher’s page marks it free, or the outlet publishes free to read: Medical Xpress, ScienceDaily, NIH (incl. NIA and PMC), Alzheimer's Association, Alzheimer's News Today, NPR, Alzheimer's Disease International, or a .gov site.
- May need a subscription (11 of 354). The publisher’s own page marks the article as not free to read, or — when the page says nothing — the outlet sells subscriptions: The New York Times, STAT, Scientific American, The Washington Post, The Wall Street Journal, Financial Times, The Economist, Bloomberg, The Atlantic, The New Yorker, Wired, Los Angeles Times, The Boston Globe, The Times, The Telegraph, New Scientist, Endpoints News, Science. Each badge says which.
- “Free to read? Not known” (35 of 354). The outlet is on neither list and its page said nothing. We do not call these paywalled.
Archived copy links go to a copy the Internet Archive (web.archive.org) saved, dated the day it was saved. We show one only where the Internet Archive reported a working saved copy when we checked; an archived copy of a subscription article sometimes shows only its opening. 266 of 354 items have one.
Scientists discover new Alzheimer’s drug that repairs DNA damage and reduces brain inflammation
Scientists have discovered that a drug originally developed for spinal cord injuries can tackle several key features of Alzheimer’s disease, including DNA damage and inflammation. …
Read at ScienceDaily →Scientists find a way to fight Alzheimer’s brain damage without removing toxic tau
Scientists have found a promising way to protect the brain from Alzheimer’s-related damage by blocking immune cells that contribute to the destruction of neurons. …
Read at ScienceDaily → Archived copy (20 Oct 2026), Internet Archive"Mind-blowing" Alzheimer’s breakthrough: Two hours of sleep restored without clearing brain plaques
Scientists have uncovered an unexpected culprit behind sleep loss in Alzheimer’s disease: overactive immune cells in the brain. By temporarily removing these cells, researchers restored more than two hours of sleep per day in mice, revealing a promising new target for treatment.
Read at ScienceDaily → Archived copy (19 Oct 2026), Internet ArchiveBrain cells meant to protect us may be fueling cognitive decline
Scientists have uncovered a surprising cause of age-related cognitive decline involving brain cells previously thought to be entirely beneficial. …
Read at ScienceDaily → Archived copy (17 Oct 2026), Internet ArchiveScientists find a youth protein that helps aging brain cells act young again
Scientists have discovered that a protein linked to youth helps protect the brain’s immune cells from age-related decline. Restoring the protein in older mice improved the cells’ ability to clear debris and reduced signs of inflammation…
Read at ScienceDaily → Archived copy (18 Oct 2026), Internet ArchiveRare study reveals a striking divide inside an Alzheimer’s patient’s brain
A rare examination of an Alzheimer’s patient’s brain revealed that regions cleared of amyloid plaques had remarkably little of the harmful tau buildup associated with memory loss, while neighboring areas showed extensive damage. …
Scientists find hidden driver of aging and a simple amino acid that fights memory loss in mice
Scientists discovered that declining levels of a protein called Menin may help drive aging, and restoring it in older mice reversed several age-related changes in just 30 days. …
Read at ScienceDaily → Archived copy (12 Oct 2026), Internet ArchiveScientists restore a brain protein and reverse signs of aging in mice
Scientists found that declining levels of the brain protein Menin may help drive aging, with lower levels in mice linked to inflammation, cognitive decline, weaker bones, and thinner skin. …
Read at ScienceDaily → Archived copy (1 Oct 2026), Internet ArchiveA major Alzheimer’s risk gene may shrink brain cells years before symptoms
Researchers have uncovered a possible reason why the Alzheimer’s risk gene APOE4 can disrupt the brain long before memory problems begin. In mice, APOE4 increased a protein called Nell2, shrinking neurons and making memory circuits unusually active. …
Read at ScienceDaily → Archived copy (1 Oct 2026), Internet ArchiveScientists discover a hidden brain shift that begins around age 50
Beginning around age 50, the brain’s memory center appears to lose many of its longtime immune cells and replace them with more inflammatory ones. This surprising shift may help explain how normal aging sets the stage for Alzheimer’s disease and dementia.
Read at ScienceDaily → Archived copy (27 Sep 2026), Internet ArchiveA hidden Alzheimer’s tipping point may decide who gets dementia
Scientists have identified a possible tipping point that helps determine whether Alzheimer’s-related brain changes lead to dementia. The key may lie in how the brain’s immune cells respond to plaques and tau…
APOE2 may protect the brain from Alzheimer’s and aging
The longevity-linked APOE2 gene appears to protect brain cells by reducing DNA damage and helping neurons recover from stress. The finding could open a new path toward treatments that mimic APOE2’s defenses in people at higher genetic risk for Alzheimer’s.
Alzheimer’s breakthrough: Scientists restore two hours of sleep without clearing brain plaques
Researchers have uncovered a surprising culprit behind sleep loss in Alzheimer’s disease: the brain’s own immune cells. In mice with amyloid plaques, overactive microglia triggered inflammation that kept the brain from getting enough deep, restorative sleep. …
Read at ScienceDaily → Archived copy (22 Sep 2026), Internet ArchiveScientists discover a protein that protects the brain from Alzheimer’s damage
A protein called SORLA may act as a powerful defense against the toxic tau tangles involved in Alzheimer’s disease. Mice with extra SORLA had less tau accumulation, less brain atrophy…
Read at ScienceDaily → Archived copy (2 Oct 2026), Internet ArchiveNew Alzheimer's drug repairs DNA damage and reduces brain inflammation
A drug originally developed for spinal cord injury may offer a fresh approach to treating Alzheimer’s disease. In mouse studies, KCL-286 repaired dangerous DNA damage, reduced inflammation…
Read at ScienceDaily → Archived copy (17 Jul 2026), Internet ArchiveThe hidden skeleton “gatekeeper” inside brain cells could help fight Alzheimer's
Researchers have discovered that a microscopic skeleton inside neurons does much more than hold cells together. It acts as a gatekeeper that controls what brain cells absorb and when they absorb it. …
Read at ScienceDaily → Archived copy (15 Jul 2026), Internet ArchiveAlzheimer's tau protein has a surprising secret role in memory
Researchers found that tau is essential for turning new experiences into lasting memories by helping organize the brain's memory-storing cells. …
Read at ScienceDaily →Scientists may have finally found how Alzheimer's kills brain cells
Researchers have identified a previously overlooked mechanism of brain cell death that appears to play a major role in Alzheimer's disease and frontotemporal dementia. …
Read at ScienceDaily → Archived copy (23 Sep 2026), Internet ArchiveScientists may have finally found how Alzheimer's spreads through the brain
A common brain protein may be giving Alzheimer’s disease an unexpected way to spread, carrying toxic Tau proteins from damaged neurons into healthy ones. …
Scientists reprogram brain immune cells to fight Alzheimer’s
A newly identified molecule called OLE helped restore the brain’s immune cells to a more protective state in Alzheimer’s models. The treatment reduced toxic plaque buildup and improved memory, raising hopes for a new therapeutic approach.
Read at ScienceDaily →Scientists May Have Found What Really Triggers Alzheimer’s Disease
Scientists may have uncovered a hidden trigger behind Alzheimer’s disease. Instead of plaques being the root cause, amyloid beta appears to interfere with tau, a protein that helps keep neurons functioning properly. …
Read at ScienceDaily → Archived copy (19 Sep 2026), Internet ArchiveScientists discover a surprising cancer link to Alzheimer’s disease
Researchers discovered that mutations linked to blood cancers may help trigger Alzheimer’s disease by creating overly inflammatory immune cells in the brain. …
Read at ScienceDaily → Archived copy (22 Sep 2026), Internet ArchiveScientists found a new Alzheimer’s trigger and a drug that stops it
Researchers have identified a new Alzheimer’s target and created an experimental compound that blocks a damaging process inside brain cells. In mice, the treatment slowed nerve cell loss, reduced Alzheimer’s-related changes, and even appeared to promote healthier aging.
Read at ScienceDaily → Archived copy (23 Sep 2026), Internet ArchiveScientists found the hidden switch fueling alzheimer’s brain inflammation
Scientists at Scripps Research have uncovered a molecular “switch” that appears to fuel the damaging brain inflammation seen in Alzheimer’s disease. …
Read at ScienceDaily → Archived copy (27 Sep 2026), Internet ArchiveScientists found a hidden Alzheimer’s trigger and shut it down
A newly identified enzyme called IDOL could become a major new target in the fight against Alzheimer’s disease. Researchers found that removing it from neurons sharply reduced amyloid plaques and improved key brain processes linked to resilience and communication between cells. …
Read at ScienceDaily → Archived copy (23 Sep 2026), Internet ArchiveScientists reverse Alzheimer’s in mice with breakthrough nanotechnology
A new nanotechnology treatment reversed Alzheimer’s symptoms in mice by restoring the brain’s natural cleanup system. The specially engineered nanoparticles helped clear toxic amyloid proteins from the brain and repair the blood-brain barrier…
Boosting one protein helps the brain fight Alzheimer’s
Scientists have discovered a way to help the brain clean itself of harmful Alzheimer’s plaques by activating its own support cells. By increasing a protein called Sox9, researchers were able to boost the activity of astrocytes, star shaped cells that help maintain brain health. …
Read at ScienceDaily → Archived copy (2 May 2026), Internet ArchiveScientists restore memory by blocking a single Alzheimer’s protein
Researchers have identified a new potential weapon against Alzheimer’s: blocking a protein called PTP1B. In mice, this approach boosted memory and helped brain immune cells clear harmful plaque buildup. …
Read at ScienceDaily → Archived copy (23 Sep 2026), Internet ArchiveScientists discover Alzheimer’s hidden “death switch” in the brain
Scientists have uncovered a hidden “death switch” in the brain that may be driving Alzheimer’s disease—and even found a way to turn it off in mice. The culprit is a toxic pairing of two proteins that, when combined, triggers the destruction of brain cells and fuels memory loss. …
Read at ScienceDaily → Archived copy (26 Sep 2026), Internet ArchiveScientists turn brain cells into Alzheimer’s plaque cleaners
Scientists have developed a promising new approach to treating Alzheimer’s disease by turning ordinary brain cells into powerful plaque-clearing machines. …
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