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Newest in this topic

  1. Neural stem cell transplantation decreases neuroinflammation in a transgenic mouse model of Alzheimer's disease 2015-12-28
  2. Increased hippocampal neurogenesis in the progressive stage of Alzheimer's disease phenotype in an APP/PS1 double transgenic mouse model 2009-12-01
  3. Endogenous brain cytokine mRNA and inflammatory responses to lipopolysaccharide are elevated in the Tg2576 transgenic mouse model of Alzheimer's disease 2001-12-01

Studies

5 studies carry this tag.

Date range

2001 to 2015

Top journals

  • Brain research bulletin 1
  • Hippocampus 1
  • Journal of neurochemistry 1
  • The European journal of neuroscience 1
  • The Journal of neuroscience : the official journal of the Society for Neuroscience 1

Newest first, then by address. Each study has a usefulness score out of 100 (rubric version 4.0); tap a score to see what makes it up. Tags were assigned automatically from each study's title and summary. The same studies as a filter in the library.

  1. Neural stem cell transplantation decreases neuroinflammation in a transgenic mouse model of Alzheimer's disease

    ✓ Peer-reviewed alzhub usefulness score 36 out of 100. Journal of neurochemistry 2015-12-28 Biomarkers

    Inflammatory processes are considered to play an important role in the progression of neurodegenerative changes in Alzheimer's disease (AD).

  2. Increased hippocampal neurogenesis in the progressive stage of Alzheimer's disease phenotype in an APP/PS1 double transgenic mouse model

    ✓ Peer-reviewed alzhub usefulness score 36 out of 100. Hippocampus 2009-12-01 Biomarkers

    Alzheimer's disease (AD) is a progressive neurodegenerative disease associated with senile beta-amyloid (Abeta) plaques and cognitive decline.

  3. Endogenous brain cytokine mRNA and inflammatory responses to lipopolysaccharide are elevated in the Tg2576 transgenic mouse model of Alzheimer's disease

    ✓ Peer-reviewed alzhub usefulness score 36 out of 100. Brain research bulletin 2001-12-01 Biomarkers

    Beta-amyloid (Abeta) plaques have been shown to induce inflammatory changes in Alzheimer's disease brains.

  4. Neuroinflammation-induced acceleration of amyloid deposition in the APPV717F transgenic mouse

    ✓ Peer-reviewed alzhub usefulness score 36 out of 100. The European journal of neuroscience 2001-08-01 Biomarkers

    It has been postulated that neuroinflammation plays a critical role in the pathogenesis of Alzheimer's disease (AD).

  5. Alpha-1-antichymotrypsin promotes beta-sheet amyloid plaque deposition in a transgenic mouse model of Alzheimer's disease

    ✓ Peer-reviewed alzhub usefulness score 36 out of 100. The Journal of neuroscience : the official journal of the Society for Neuroscience 2001-03-01 Biomarkers

    Alpha(1)-antichymotrypsin (ACT), an acute-phase inflammatory protein, is an integral component of the amyloid deposits in Alzheimer's disease (AD) and has been shown to catalyze amyloid beta-peptide polymerization in vitro.

All topic tags with their study counts