alzhub.org
Get help now — free helplines

You can call any of these for free. You do not need a diagnosis, an appointment, or a crisis to call — questions and plain exhaustion are reasons enough.

  • Alzheimer's Association 24/7 Helpline 800-272-3900 24 hours a day, 365 days a year Free, confidential support and information for people with dementia, caregivers, families and the public. Master's-level clinicians. Also Spanish.
  • 988 Suicide & Crisis Lifeline 988 24/7 Call or text 988, or chat at 988lifeline.org. For anyone in emotional distress or a mental-health crisis, including caregivers.
  • Alzheimer's Foundation of America (AFA) Helpline 866-232-8484 9:00 am - 9:00 pm ET, seven days a week Licensed social workers providing support, info and referrals. Also text message and web chat. Text: 646-586-5283

In a medical emergency, call 911. More numbers, and what each line does, on the help page.

Home › Research › Topics › pathogenesis

pathogenesis

7 studies tagged pathogenesis, dated 2012-03-30 to 2024-12-21. Bibliography: BibTeX (.bib) · RIS (.ris)

Newest in this topic

  1. Brain derived β-interferon is a potential player in Alzheimer's disease pathogenesis and cognitive impairment 2024-12-21
  2. Morphological and Molecular Profiling of Amyloid-β Species in Alzheimer's Pathogenesis 2024-10-24
  3. Potential Implications of miRNAs in the Pathogenesis, Diagnosis, and Therapeutics of Alzheimer's Disease 2023-11-13

Studies

7 studies carry this tag.

Date range

2012 to 2024

Top journals

  • Alzheimer's & dementia : the journal of the Alzheimer's Association 1
  • Alzheimer's research & therapy 1
  • Frontiers in aging neuroscience 1
  • International journal of molecular sciences 1
  • Molecular neurobiology 1

7 journals in all.

Newest first, then by address. Each study has a usefulness score out of 100 (rubric version 4.0); tap a score to see what makes it up. Tags were assigned automatically from each study's title and summary. The same studies as a filter in the library.

  1. Brain derived β-interferon is a potential player in Alzheimer's disease pathogenesis and cognitive impairment

    ✓ Peer-reviewed alzhub usefulness score 60 out of 100. Alzheimer's research & therapy 2024-12-21 Biomarkers

    Our findings demonstrate that distinct inflammatory molecules are associated with different cognitive disorders.

  2. Morphological and Molecular Profiling of Amyloid-β Species in Alzheimer's Pathogenesis

    ✓ Peer-reviewed alzhub usefulness score 45 out of 100. Molecular neurobiology 2024-10-24 Biomarkers

    Alzheimer's disease (AD) is the most common form of dementia around the world (~ 65%).

  3. Potential Implications of miRNAs in the Pathogenesis, Diagnosis, and Therapeutics of Alzheimer's Disease

    ✓ Peer-reviewed alzhub usefulness score 43 out of 100. International journal of molecular sciences 2023-11-13 Biomarkers

    Alzheimer's disease (AD) is a complex multifactorial disorder that poses a substantial burden on patients, caregivers, and society.

  4. Presenilin Is Essential for ApoE Secretion, a Novel Role of Presenilin Involved in Alzheimer's Disease Pathogenesis

    ✓ Peer-reviewed alzhub usefulness score 40 out of 100. The Journal of neuroscience : the official journal of the Society for Neuroscience 2022-01-05 Biomarkers

    Alzheimer's disease (AD) is a debilitating dementia characterized by progressive memory loss and aggregation of amyloid-β (Aβ) protein into amyloid plaques in patient brains.

  5. Effect of Presenilin Mutations on APP Cleavage; Insights into the Pathogenesis of FAD

    ✓ Peer-reviewed alzhub usefulness score 40 out of 100. Frontiers in aging neuroscience 2016-03-11 Biomarkers

    Alzheimer disease (AD) is characterized by progressive memory loss, reduction in cognitive functions, and damage to the brain.

  6. β-Amyloid pathogenesis: Chemical properties versus cellular levels

    ✓ Peer-reviewed alzhub usefulness score 60 out of 100. Alzheimer's & dementia : the journal of the Alzheimer's Association 2015-07-28 Biomarkers

    Although genetic Aβ variants cause early-onset Alzheimer's disease, literature reports on Aβ properties are heterogeneous, obscuring molecular mechanisms, as illustrated by recent failures of Aβ-level targeting trials.

  7. Modulation of γ-secretase activity by multiple enzyme-substrate interactions: implications in pathogenesis of Alzheimer's disease

    ✓ Peer-reviewed alzhub usefulness score 40 out of 100. PloS one 2012-03-30 Biomarkers

    Gradual saturation of γ-secretase with its substrate can be the pathogenic process in different alleged causes of AD.

All topic tags with their study counts