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Newest in this topic

  1. Oligomers of β-amyloid protein (Aβ1-42) induce the activation of cyclooxygenase-2 in astrocytes via an interaction with interleukin-1β, tumour necrosis factor-α, and a nuclear factor κ-B mechanism in the rat brain 2012-05-14
  2. Neuropsychological tests and functional nuclear neuroimaging provide evidence of subclinical impairment in Nasu-Hakola disease heterozygotes 2005-04-01

Studies

2 studies carry this tag.

Date range

2005 to 2012

Top journals

  • Experimental neurology 1
  • Functional neurology 1

Newest first, then by address. Each study has a usefulness score out of 100 (rubric version 4.0); tap a score to see what makes it up. Tags were assigned automatically from each study's title and summary. The same studies as a filter in the library.

  1. Despite growing evidence indicating the effects of cytokines, including interleukin-1beta (IL-1β) and tumour necrosis factor-α (TNFα), and the enzyme cyclooxygenase-2 (COX-2) in Alzheimer's diseases, little is known about the signalling mechanisms that mediate its activation in response to beta-amyloid protein (Aβ).

  2. Neuropsychological tests and functional nuclear neuroimaging provide evidence of subclinical impairment in Nasu-Hakola disease heterozygotes

    ✓ Peer-reviewed alzhub usefulness score 36 out of 100. Functional neurology 2005-04-01 TREM2 and immune genetics

    Nasu-Hakola disease is a rare, recessively inherited disease characterized by presenile dementia and bone cysts.

All topic tags with their study counts