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neuropathology
7 studies tagged neuropathology, dated 2019-05-08 to 2024-03-27. Bibliography: BibTeX (.bib) · RIS (.ris)
Newest in this topic
- Neuropathology, Neuroimaging, and Fluid Biomarkers in Alzheimer's Disease 2024-03-27
- Plasma p-tau212 antemortem diagnostic performance and prediction of autopsy verification of Alzheimer's disease neuropathology 2024-03-23
- Optimal blood tau species for the detection of Alzheimer's disease neuropathology: an immunoprecipitation mass spectrometry and autopsy study 2023-12-30
Studies
7 studies carry this tag.
Date range
2019 to 2024
Top journals
- Acta neuropathologica 1
- Acta neuropathologica communications 1
- Alzheimer's & dementia : the journal of the Alzheimer's Association 1
- Brain : a journal of neurology 1
- Diagnostics (Basel, Switzerland) 1
7 journals in all.
Newest first, then by address. Each study has a usefulness score out of 100 (rubric version 4.0); tap a score to see what makes it up. Tags were assigned automatically from each study's title and summary. The same studies as a filter in the library.
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An improved understanding of the pathobiology of Alzheimer's disease (AD) should lead ultimately to an earlier and more accurate diagnosis of AD, providing the opportunity to intervene earlier in the disease process and to improve outcomes.
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Blood phosphorylated tau (p-tau) biomarkers, including p-tau217, show high associations with Alzheimer's disease (AD) neuropathologic change and clinical stage.
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Plasma-to-autopsy studies are essential for validation of blood biomarkers and understanding their relation to Alzheimer's disease (AD) pathology.
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This study supports that PET- and CSF-related A/T/N profiles can be used to predict accurately the stages of AD neuropathology.
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Blood-based biomarkers such as tau phosphorylated at threonine 181 (phosphorylated-tau181) represent an accessible, cost-effective and scalable approach for the in vivo detection of Alzheimer's disease pathophysiology.
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Alzheimer disease (AD) is a chronic neurodegenerative disease with a multitude of contributing genetic factors, many of which are related to inflammation.
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Redistribution of the water channel aquaporin-4 (AQP4) away from astrocyte endfeet and into parenchymal processes is a striking histological feature in mouse models of Alzheimer's disease (AD) and other neurological conditions with prominent astrogliosis.