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Newest in this topic

  1. Age, sex and APOE-ε4 modify the balance between soluble and fibrillar β-amyloid in non-demented individuals: topographical patterns across two independent cohorts 2022-03-02
  2. Rare mutations in SQSTM1 modify susceptibility to frontotemporal lobar degeneration 2014-06-05
  3. GAB2 alleles modify Alzheimer's risk in APOE epsilon4 carriers 2007-06-01

Studies

3 studies carry this tag.

Date range

2007 to 2022

Top journals

  • Acta neuropathologica 1
  • Molecular psychiatry 1
  • Neuron 1

Newest first, then by address. Each study has a usefulness score out of 100 (rubric version 4.0); tap a score to see what makes it up. Tags were assigned automatically from each study's title and summary. The same studies as a filter in the library.

  1. Amyloid (Aβ) pathology is the earliest detectable pathophysiological event along the Alzheimer's continuum, which can be measured both in the cerebrospinal fluid (CSF) and by Positron Emission Tomography (PET).

  2. Rare mutations in SQSTM1 modify susceptibility to frontotemporal lobar degeneration

    ✓ Peer-reviewed alzhub usefulness score 86 out of 100. Acta neuropathologica 2014-06-05 Genetics

    Mutations in the gene coding for Sequestosome 1 (SQSTM1) have been genetically associated with amyotrophic lateral sclerosis (ALS) and Paget disease of bone.

  3. GAB2 alleles modify Alzheimer's risk in APOE epsilon4 carriers

    ✓ Peer-reviewed alzhub usefulness score 36 out of 100. Neuron 2007-06-01 Genetics

    The apolipoprotein E (APOE) epsilon4 allele is the best established genetic risk factor for late-onset Alzheimer's disease (LOAD).

All topic tags with their study counts