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Home › Research › Topics › mitochondrial-dysfunction

mitochondrial-dysfunction

3 studies tagged mitochondrial-dysfunction, dated 2005-12-12 to 2023-03-13. Bibliography: BibTeX (.bib) · RIS (.ris)

Newest in this topic

  1. Gastrodin and Gastrodigenin Improve Energy Metabolism Disorders and Mitochondrial Dysfunction to Antagonize Vascular Dementia 2023-03-13
  2. Mitochondrial dysfunction in Alzheimer's disease: Role in pathogenesis and novel therapeutic opportunities 2019-03-06
  3. Lipid- and receptor-binding regions of apolipoprotein E4 fragments act in concert to cause mitochondrial dysfunction and neurotoxicity 2005-12-12

Studies

3 studies carry this tag.

Date range

2005 to 2023

Top journals

  • British journal of pharmacology 1
  • Molecules (Basel, Switzerland) 1
  • Proceedings of the National Academy of Sciences of the United States of America 1

Newest first, then by address. Each study has a usefulness score out of 100 (rubric version 4.0); tap a score to see what makes it up. Tags were assigned automatically from each study's title and summary. The same studies as a filter in the library.

  1. Gastrodin and Gastrodigenin Improve Energy Metabolism Disorders and Mitochondrial Dysfunction to Antagonize Vascular Dementia

    ✓ Peer-reviewed alzhub usefulness score 43 out of 100. Molecules (Basel, Switzerland) 2023-03-13 Biomarkers

    Vascular dementia (VD) is the second most common dementia syndrome worldwide, and effective treatments are lacking.

  2. Mitochondrial dysfunction in Alzheimer's disease: Role in pathogenesis and novel therapeutic opportunities

    ✓ Peer-reviewed alzhub usefulness score 38 out of 100. British journal of pharmacology 2019-03-06 Genetics

    Dysfunction of cell bioenergetics is a common feature of neurodegenerative diseases, the most common of which is Alzheimer's disease (AD).

  3. Lipid- and receptor-binding regions of apolipoprotein E4 fragments act in concert to cause mitochondrial dysfunction and neurotoxicity

    ✓ Peer-reviewed alzhub usefulness score 36 out of 100. Proceedings of the National Academy of Sciences of the United States of America 2005-12-12 Genetics

    Apolipoprotein (apo) E4, a 299-aa protein and a major risk factor for Alzheimer's disease, can be cleaved to generate C-terminal-truncated fragments that cause neurotoxicity in vitro and neurodegeneration and behavioral deficits in transgenic mice.

All topic tags with their study counts