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lewy-bodies
6 studies tagged lewy-bodies, dated 2004-12-24 to 2024-07-03. Bibliography: BibTeX (.bib) · RIS (.ris)
Newest in this topic
- Plasma biomarkers of amyloid, tau, axonal, and neuroinflammation pathologies in dementia with Lewy bodies 2024-07-03
- Plasma GFAP associates with secondary Alzheimer's pathology in Lewy body disease 2023-03-31
- Cerebrospinal Fluid EV Concentration and Size Are Altered in Alzheimer's Disease and Dementia with Lewy Bodies 2022-01-28
Studies
6 studies carry this tag.
Date range
2004 to 2024
Top journals
- Acta neuropathologica 1
- Alzheimer's research & therapy 1
- Annals of clinical and translational neurology 1
- Annals of neurology 1
- Cells 1
6 journals in all.
Newest first, then by address. Each study has a usefulness score out of 100 (rubric version 4.0); tap a score to see what makes it up. Tags were assigned automatically from each study's title and summary. The same studies as a filter in the library.
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Amyloid, tau, neurodegeneration and neuroinflammation plasma biomarkers are modified in DLB, albeit with moderate diagnosis performance.
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Linear models showed that plasma GFAP was significantly higher in αSyn+AD compared to αSyn (β = 0.31, 95% CI = 0.065-0.56, and P = 0.015), after covarying for age at plasma, plasma-to-death interval, and sex; plasma p-tau 181 was not (P = 0.37).
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Alzheimer's disease (AD), dementia with Lewy bodies (DLB) and frontotemporal dementia (FTD) represent the three major neurodegenerative dementias characterized by abnormal brain protein accumulation.
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NFL and αSyn oligomers in CSF faithfully differentiate early MSA not only from CON but also from Lewy body synucleinopathies.
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A major difference in the revised diagnostic criteria for Alzheimer's disease (AD) is the incorporation of biomarkers to support a clinical diagnosis and allow the identification of preclinical AD due to AD neuropathological processes.
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Dementia is a frequent complication of Parkinson's disease (PD) and usually occurs late in the protracted course of the illness.