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Home › Research › Topics › interleukin-1

interleukin-1

3 studies tagged interleukin-1, dated 1995-03-01 to 2012-05-14. Bibliography: BibTeX (.bib) · RIS (.ris)

Newest in this topic

  1. Oligomers of β-amyloid protein (Aβ1-42) induce the activation of cyclooxygenase-2 in astrocytes via an interaction with interleukin-1β, tumour necrosis factor-α, and a nuclear factor κ-B mechanism in the rat brain 2012-05-14
  2. Association of interleukin-1 gene polymorphisms with Alzheimer's disease 2000-03-01
  3. Interleukin-1 expression in different plaque types in Alzheimer's disease: significance in plaque evolution 1995-03-01

Studies

3 studies carry this tag.

Date range

1995 to 2012

Top journals

  • Annals of neurology 1
  • Experimental neurology 1
  • Journal of neuropathology and experimental neurology 1

Newest first, then by address. Each study has a usefulness score out of 100 (rubric version 4.0); tap a score to see what makes it up. Tags were assigned automatically from each study's title and summary. The same studies as a filter in the library.

  1. Oligomers of β-amyloid protein (Aβ1-42) induce the activation of cyclooxygenase-2 in astrocytes via an interaction with interleukin-1β, tumour necrosis factor-α, and a nuclear factor κ-B mechanism in the rat brain

    ✓ Peer-reviewed alzhub usefulness score 36 out of 100. Experimental neurology 2012-05-14 Biomarkers

    Despite growing evidence indicating the effects of cytokines, including interleukin-1beta (IL-1β) and tumour necrosis factor-α (TNFα), and the enzyme cyclooxygenase-2 (COX-2) in Alzheimer's diseases, little is known about the signalling mechanisms that mediate its activation in response to beta-amyloid protein (Aβ).

  2. Association of interleukin-1 gene polymorphisms with Alzheimer's disease

    ✓ Peer-reviewed alzhub usefulness score 43 out of 100. Annals of neurology 2000-03-01 Genetics

    Interleukin-1 (IL-1) is markedly overexpressed in Alzheimer's disease.

  3. Interleukin-1 expression in different plaque types in Alzheimer's disease: significance in plaque evolution

    ✓ Peer-reviewed alzhub usefulness score 36 out of 100. Journal of neuropathology and experimental neurology 1995-03-01 Risk genes beyond APOE

    The histologically apparent polymorphism of plaques containing beta-amyloid in Alzheimer's disease is thought to represent different stages in plaque evolution. beta-amyloid-immunopositive plaques were classified according to the pattern of beta-amyloid distribution (diffuse vs dense-core) and the presence or absence of dystrophic beta-amyloid precursor protein-immunopositive (beta-APP+) neurites (neuritic vs non-neuritic).

All topic tags with their study counts