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Newest in this topic

  1. Fusobacterium nucleatum infection-induced neurodegeneration and abnormal gut microbiota composition in Alzheimer's disease-like rats 2022-09-16
  2. New Insights into the Spontaneous Human Alzheimer's Disease-Like Model Octodon degus: Unraveling Amyloid-β Peptide Aggregation and Age-Related Amyloid Pathology 2018-01-01
  3. Alzheimer's disease-like APP processing in wild-type mice identifies synaptic defects as initial steps of disease progression 2016-01-12

Studies

3 studies carry this tag.

Date range

2016 to 2022

Top journals

  • Frontiers in neuroscience 1
  • Journal of Alzheimer's disease : JAD 1
  • Molecular neurodegeneration 1

Newest first, then by address. Each study has a usefulness score out of 100 (rubric version 4.0); tap a score to see what makes it up. Tags were assigned automatically from each study's title and summary. The same studies as a filter in the library.

  1. Fusobacterium nucleatum infection-induced neurodegeneration and abnormal gut microbiota composition in Alzheimer's disease-like rats

    ✓ Peer-reviewed alzhub usefulness score 43 out of 100. Frontiers in neuroscience 2022-09-16 Biomarkers

    Oral infection with F. nucleatum could contribute to abnormalities in cognitive ability and pathological change in the brain of AD-like rats, which may be related to abnormal gut microbiota composition.

  2. New Insights into the Spontaneous Human Alzheimer's Disease-Like Model Octodon degus: Unraveling Amyloid-β Peptide Aggregation and Age-Related Amyloid Pathology

    ✓ Peer-reviewed alzhub usefulness score 38 out of 100. Journal of Alzheimer's disease : JAD 2018-01-01 Biomarkers

    Alzheimer's disease (AD) is the most common cause of dementia worldwide.

  3. Alzheimer's disease-like APP processing in wild-type mice identifies synaptic defects as initial steps of disease progression

    ✓ Peer-reviewed alzhub usefulness score 40 out of 100. Molecular neurodegeneration 2016-01-12 Biomarkers

    This demonstrates that an AD-like APP processing, yielding to levels of APP, βCTF and Aβ42/Aβ40 ratio similar to those observed in AD patients, are sufficient to rapidly trigger early steps of the amyloidogenic and Tau pathways in vivo.

All topic tags with their study counts