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Home › Research › Topics › deletion

deletion

4 studies tagged deletion, dated 2008-02-01 to 2021-09-29. Bibliography: BibTeX (.bib) · RIS (.ris)

Newest in this topic

  1. Potentiation of amyloid beta phagocytosis and amelioration of synaptic dysfunction upon FAAH deletion in a mouse model of Alzheimer's disease 2021-09-29
  2. Deletion of Alzheimer's disease-associated CD33 results in an inflammatory human microglia phenotype 2021-02-04
  3. Conditional genetic deletion of CSF1 receptor in microglia ameliorates the physiopathology of Alzheimer's disease 2021-01-05

Studies

4 studies carry this tag.

Date range

2008 to 2021

Top journals

  • Alzheimer's research & therapy 1
  • Glia 1
  • Journal of neuroinflammation 1
  • The Journal of neuroscience : the official journal of the Society for Neuroscience 1

Newest first, then by address. Each study has a usefulness score out of 100 (rubric version 4.0); tap a score to see what makes it up. Tags were assigned automatically from each study's title and summary. The same studies as a filter in the library.

  1. Potentiation of amyloid beta phagocytosis and amelioration of synaptic dysfunction upon FAAH deletion in a mouse model of Alzheimer's disease

    ✓ Peer-reviewed alzhub usefulness score 43 out of 100. Journal of neuroinflammation 2021-09-29 TREM2 and immune genetics

    Our findings support the therapeutic potential of modulating, rather than suppressing, neuroinflammation in Alzheimer's disease.

  2. Deletion of Alzheimer's disease-associated CD33 results in an inflammatory human microglia phenotype

    ✓ Peer-reviewed alzhub usefulness score 40 out of 100. Glia 2021-02-04 Genetics

    Genome-wide association studies demonstrated that polymorphisms in the CD33/sialic acid-binding immunoglobulin-like lectin 3 gene are associated with late-onset Alzheimer's disease (AD).

  3. Conditional genetic deletion of CSF1 receptor in microglia ameliorates the physiopathology of Alzheimer's disease

    ✓ Peer-reviewed alzhub usefulness score 43 out of 100. Alzheimer's research & therapy 2021-01-05 TREM2 and immune genetics

    Our results provide new insights on the role of CSF1R in microglia and how it interacts with the TREM2/β-Catenin and IL-34 system to clear Aβ and ameliorates the physiopathology of AD.

  4. Axonal transport rates in vivo are unaffected by tau deletion or overexpression in mice

    ✓ Peer-reviewed alzhub usefulness score 36 out of 100. The Journal of neuroscience : the official journal of the Society for Neuroscience 2008-02-01 Biomarkers

    Elevated tau expression has been proposed as a possible basis for impaired axonal transport in Alzheimer's disease.

All topic tags with their study counts