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control
7 studies tagged control, dated 2010-12-16 to 2020-01-01. Bibliography: BibTeX (.bib) · RIS (.ris)
Newest in this topic
- Plasma Amyloid Concentration in Alzheimer's Disease: Performance of a High-Throughput Amyloid Assay in Distinguishing Alzheimer's Disease Cases from Controls 2020-01-01
- CSF or serum neurofilament light added to α-Synuclein panel discriminates Parkinson's from controls 2019-11-18
- Correlations between serum and CSF pNfH levels in ALS, FTD and controls: a comparison of three analytical approaches 2019-09-01
Studies
7 studies carry this tag.
Date range
2010 to 2020
Top journals
- Journal of Alzheimer's disease : JAD 2
- CNS & neurological disorders drug targets 1
- Clinical chemistry and laboratory medicine 1
- Journal of neural transmission (Vienna, Austria : 1996) 1
- Movement disorders : official journal of the Movement Disorder Society 1
6 journals in all.
Newest first, then by address. Each study has a usefulness score out of 100 (rubric version 4.0); tap a score to see what makes it up. Tags were assigned automatically from each study's title and summary. The same studies as a filter in the library.
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In this sample, the high-throughput blood Aβ assay was not able to distinguish well between AD patients and controls.
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41/100
CSF neurofilament contributes to a panel of CSF α-synuclein species in differentiating PD patients from healthy controls.
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Serum levels also correlated strongly with CSF levels for Simoa-based measurements (both ϱ = 0.62).
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There is no molecular explanation for the many presenilin 1 (PSEN1) mutations causing Alzheimer's disease, but both gain of function relating to amyloid production and loss of isolated PSEN1 function have been implied.
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Pre-treatment with co-ultraPEALut significantly reduced inducible nitric oxide synthase and glial fibrillary acidic protein expression, restored neuronal nitric oxide synthase and brainderived neurotrophic factor and reduced the apoptosis.
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Alzheimer's disease (AD) is characterized by a progressive cognitive impairment tightly correlated with the accumulation of amyloid-β (Aβ) peptides (mainly Aβ(1-42)).
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Cerebrospinal fluid (CSF) biomarkers, such as Aβ(42), total-tau and phosphorylated-tau(181) reflect neuropathological changes of Alzheimer's disease (AD).