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Home › Research › Topics › contributes

contributes

7 studies tagged contributes, dated 2011-11-17 to 2022-04-26. Bibliography: BibTeX (.bib) · RIS (.ris)

Newest in this topic

  1. APOE4 exacerbates α-synuclein seeding activity and contributes to neurotoxicity in Alzheimer's disease with Lewy body pathology 2022-04-26
  2. Reactive Astrocytes Contribute to Alzheimer's Disease-Related Neurotoxicity and Synaptotoxicity in a Neuron-Astrocyte Co-culture Assay 2022-01-21
  3. The human-specific CASP4 gene product contributes to Alzheimer-related synaptic and behavioural deficits 2016-08-11

Studies

7 studies carry this tag.

Date range

2011 to 2022

Top journals

  • Journal of neurochemistry 2
  • Acta neuropathologica 1
  • Frontiers in cellular neuroscience 1
  • Human molecular genetics 1
  • Neurobiology of aging 1

6 journals in all.

Newest first, then by address. Each study has a usefulness score out of 100 (rubric version 4.0); tap a score to see what makes it up. Tags were assigned automatically from each study's title and summary. The same studies as a filter in the library.

  1. APOE4 exacerbates α-synuclein seeding activity and contributes to neurotoxicity in Alzheimer's disease with Lewy body pathology

    ✓ Peer-reviewed alzhub usefulness score 59 out of 100. Acta neuropathologica 2022-04-26 Genetics

    Approximately half of Alzheimer's disease (AD) brains have concomitant Lewy pathology at autopsy, suggesting that α-synuclein (α-SYN) aggregation is a regulated event in the pathogenesis of AD.

  2. Reactive Astrocytes Contribute to Alzheimer's Disease-Related Neurotoxicity and Synaptotoxicity in a Neuron-Astrocyte Co-culture Assay

    ✓ Peer-reviewed alzhub usefulness score 43 out of 100. Frontiers in cellular neuroscience 2022-01-21 Biomarkers

    Pathogenesis of presymptomatic disease stages remains elusive, although studies suggest that the early structural and functional alterations likely occur at neuronal dendritic spines.

  3. The human-specific CASP4 gene product contributes to Alzheimer-related synaptic and behavioural deficits

    ✓ Peer-reviewed alzhub usefulness score 36 out of 100. Human molecular genetics 2016-08-11 TREM2 and immune genetics

    Recent studies have indicated that innate immune signalling molecules are involved in late-onset Alzheimer's disease (LOAD) risk.

  4. Cardiovascular disease contributes to Alzheimer's disease: evidence from large-scale genome-wide association studies

    ✓ Peer-reviewed alzhub usefulness score 76 out of 100. Neurobiology of aging 2013-10-19 Genetics

    Alzheimer's disease (AD) is the most common and complex neurodegenerative disease in the elderly individuals.

  5. PICALM gene rs3851179 polymorphism contributes to Alzheimer's disease in an Asian population

    ✓ Peer-reviewed alzhub usefulness score 64 out of 100. Neuromolecular medicine 2013-04-10 Genetics

    PICALM gene rs3851179 polymorphism was reported to an Alzheimer's disease (AD) susceptibility locus in a Caucasian population.

  6. Impaired mitochondrial biogenesis contributes to mitochondrial dysfunction in Alzheimer's disease

    ✓ Peer-reviewed alzhub usefulness score 36 out of 100. Journal of neurochemistry 2011-12-08 Risk genes beyond APOE

    Mitochondrial dysfunction is a prominent feature of Alzheimer's disease (AD) brain.

  7. Cell adhesion molecules contribute to Alzheimer's disease: multiple pathway analyses of two genome-wide association studies

    ✓ Peer-reviewed alzhub usefulness score 47 out of 100. Journal of neurochemistry 2011-11-17 Genetics

    Alzheimer's disease (AD) is a kind of complex neurological disorder.

All topic tags with their study counts